Toshihiko Yanagita

Summary

Affiliation: University of Miyazaki
Country: Japan

Publications

  1. request reprint
    Yanagita T, Maruta T, Uezono Y, Satoh S, Yoshikawa N, Nemoto T, et al. Lithium inhibits function of voltage-dependent sodium channels and catecholamine secretion independent of glycogen synthase kinase-3 in adrenal chromaffin cells. Neuropharmacology. 2007;53:881-9 pubmed
    ..These results suggest that lithium selectively inhibits Na(+) influx thorough Na(+) channels and subsequent Ca(2+) influx and catecholamine secretion, independent of GSK-3 inhibition. ..
  2. Yanagita T, Maruta T, Nemoto T, Uezono Y, Matsuo K, Satoh S, et al. Chronic lithium treatment up-regulates cell surface Na(V)1.7 sodium channels via inhibition of glycogen synthase kinase-3 in adrenal chromaffin cells: enhancement of Na(+) influx, Ca(2+) influx and catecholamine secretion after lithium withdrawal. Neuropharmacology. 2009;57:311-21 pubmed publisher
    ..Thus, GSK-3 inhibition by LiCl up-regulated cell surface Na(V)1.7 via acceleration of alpha-subunit gene transcription, enhancing veratridine-induced Na(+) influx, Ca(2+) influx and catecholamine secretion. ..
  3. Yanagita T, Satoh S, Uezono Y, Matsuo K, Nemoto T, Maruta T, et al. Transcriptional up-regulation of cell surface Na V 1.7 sodium channels by insulin-like growth factor-1 via inhibition of glycogen synthase kinase-3? in adrenal chromaffin cells: enhancement of 22Na+ influx, 45Ca2+ influx and catecholamine secretion. Neuropharmacology. 2011;61:1265-74 pubmed publisher
    ..Thus, inhibition of GSK-3? caused by IGF-1 up-regulates cell surface expression of functional Na(+) channels via acceleration of ?-subunit gene transcription. ..