A Tamaoka

Summary

Affiliation: University of Tsukuba
Country: Japan

Publications

  1. ncbi Corticosteroid- responsive asymmetric neuropathy with a myelin protein zero gene mutation
    M Watanabe
    Department of Neurology, Institute of Clinical Medicine, University of Tsukuba, Ibaraki, Japan
    Neurology 59:767-9. 2002
  2. ncbi Association between catechol-O-methyltransferase gene polymorphisms and wearing-off and dyskinesia in Parkinson's disease
    M Watanabe
    Department of Neurology, Institute of Clinical Medicine, University of Tsukuba, Tsukuba, Japan
    Neuropsychobiology 48:190-3. 2003
  3. ncbi [Adult Sandhoff disease presented as a motor neuron disease phenotype with slow progression]
    Y Kohno
    Department of Neurology, Institute of Clinical Medicine, University of Tsukuba
    Rinsho Shinkeigaku 41:36-9. 2001
  4. doi Detection of elevated levels of α-synuclein oligomers in CSF from patients with Parkinson disease
    T Tokuda
    Department of Neurology, Kyoto Prefectural University of Medicine, Kyoto, Japan
    Neurology 75:1766-72. 2010
  5. doi Sequential imaging analysis using MIBG scintigraphy revealed progressive degeneration of cardiac sympathetic nerve in Parkinson's disease
    M Watanabe
    Department of Neurology, University of Tsukuba, Tsukuba, Ibaraki, Japan
    Eur J Neurol 18:1010-3. 2011
  6. ncbi Increased amyloid beta protein in the skin of patients with amyotrophic lateral sclerosis
    A Tamaoka
    Department of Neurology, Institute of Clinical Medicine, University of Tsukuba, Ibaraki, Japan
    J Neurol 247:633-5. 2000
  7. ncbi [A case of eosinophilic myositis proven by immunohistochemistry using antibodies against eosinophilic granule protein]
    H Harada
    Department of Neurology, Institute of Clinical Medicine, University of Tsukuba
    Rinsho Shinkeigaku 41:387-9. 2001
  8. ncbi Distinguishable effects of presenilin-1 and APP717 mutations on amyloid plaque deposition
    K Ishii
    Department of Molecular Biology, Tokyo Institute of Psychiatry, Tokyo, Japan
    Neurobiol Aging 22:367-76. 2001

Collaborators

Detail Information

Publications8

  1. ncbi Corticosteroid- responsive asymmetric neuropathy with a myelin protein zero gene mutation
    M Watanabe
    Department of Neurology, Institute of Clinical Medicine, University of Tsukuba, Ibaraki, Japan
    Neurology 59:767-9. 2002
    ..On nerve biopsy, there was demyelination and onion-bulb formation, and molecular analysis revealed that the patient was heterozygous for an MPZ mutation. The patient improved with corticosteroid treatment...
  2. ncbi Association between catechol-O-methyltransferase gene polymorphisms and wearing-off and dyskinesia in Parkinson's disease
    M Watanabe
    Department of Neurology, Institute of Clinical Medicine, University of Tsukuba, Tsukuba, Japan
    Neuropsychobiology 48:190-3. 2003
    ..030, odds ratio = 4.80) compared with controls, although these differences were not significant after Bonferroni's correction. Our results may help understand the mechanism that cause complications of levodopa therapy in PD patients...
  3. ncbi [Adult Sandhoff disease presented as a motor neuron disease phenotype with slow progression]
    Y Kohno
    Department of Neurology, Institute of Clinical Medicine, University of Tsukuba
    Rinsho Shinkeigaku 41:36-9. 2001
    ..These data appear to show that he is a compound heterozygote in the locus of the hexosaminidase B gene. This is the first Japanese case of adult Sanhoff disease presented as a motor neuron disease phenotype...
  4. doi Detection of elevated levels of α-synuclein oligomers in CSF from patients with Parkinson disease
    T Tokuda
    Department of Neurology, Kyoto Prefectural University of Medicine, Kyoto, Japan
    Neurology 75:1766-72. 2010
    ..The discovery of mutations in the SNCA gene encoding α-synuclein in familial parkinsonism and the accumulation of α-synuclein in the PD brain suggested a critical role for this protein in PD etiology...
  5. doi Sequential imaging analysis using MIBG scintigraphy revealed progressive degeneration of cardiac sympathetic nerve in Parkinson's disease
    M Watanabe
    Department of Neurology, University of Tsukuba, Tsukuba, Ibaraki, Japan
    Eur J Neurol 18:1010-3. 2011
    ..To estimate the utility of the imaging technique as a biomarker, we conducted sequential imaging analysis and power analysis...
  6. ncbi Increased amyloid beta protein in the skin of patients with amyotrophic lateral sclerosis
    A Tamaoka
    Department of Neurology, Institute of Clinical Medicine, University of Tsukuba, Ibaraki, Japan
    J Neurol 247:633-5. 2000
    ..Taking these previous findings together with those of the present study, our observations suggest that changes in extracellular matrix in skin of ALS patients may facilitate aggregation and deposition of Abeta...
  7. ncbi [A case of eosinophilic myositis proven by immunohistochemistry using antibodies against eosinophilic granule protein]
    H Harada
    Department of Neurology, Institute of Clinical Medicine, University of Tsukuba
    Rinsho Shinkeigaku 41:387-9. 2001
    ..Eosinophilic myositis was diagnosed by a biopsy of the left calf muscle showing mild infiltration of eosinophilis which was identified using antibodies against eosinophilic granule protein...
  8. ncbi Distinguishable effects of presenilin-1 and APP717 mutations on amyloid plaque deposition
    K Ishii
    Department of Molecular Biology, Tokyo Institute of Psychiatry, Tokyo, Japan
    Neurobiol Aging 22:367-76. 2001
    ..Our results suggest that PS-1 mutations affect cerebral accumulation of Abeta burden in a different fashion from APP717 mutations in their familial AD brains...