Jiro Nakamura

Summary

Affiliation: University of Tsukuba
Country: Japan

Publications

  1. ncbi request reprint Augmentation of lipolysis in adipocytes from fed rats, but not from starved rats, by inhibition of rolipram-sensitive phosphodiesterase 4
    Jiro Nakamura
    Institute of Clinical Medicine, University of Tsukuba, Tsukuba shi, Ibaraki ken 305 8577, Japan
    Arch Biochem Biophys 425:106-14. 2004
  2. doi request reprint Protein kinase CbetaI interacts with the beta1-adrenergic signaling pathway to attenuate lipolysis in rat adipocytes
    Jiro Nakamura
    Graduate School of Comprehensive Human Sciences, University of Tsukuba, Tsukuba shi, Ibaraki Ken, Japan
    Biochim Biophys Acta 1781:277-81. 2008
  3. doi request reprint Phorbol 12-myristate 13-acetate inhibits the antilipolytic action of insulin, probably via the activity of protein kinase Cε
    Jiro Nakamura
    Graduate School of Comprehensive Human Sciences, University of Tsukuba, Tsukuba shi, Ibaraki Ken, 305 8575, Japan
    Eur J Pharmacol 648:188-94. 2010
  4. ncbi request reprint Protein kinase C-dependent antilipolysis by insulin in rat adipocytes
    Jiro Nakamura
    Graduate School of Comprehensive Human Sciences, University of Tsukuba, Tsukuba shi, Ibaraki Ken, 305 8575, Japan
    Biochim Biophys Acta 1771:1195-201. 2007
  5. ncbi request reprint Protein kinase C attenuates beta-adrenergic receptor-mediated lipolysis, probably through inhibition of the beta1-adrenergic receptor system
    Jiro Nakamura
    Graduate School of Comprehensive Human Sciences, University of Tsukuba, Tsukuba shi, Ibaraki ken 305 8575, Japan
    Arch Biochem Biophys 447:1-10. 2006
  6. ncbi request reprint Polyol pathway and protein kinase C activity of rat Schwannoma cells
    Hideki Kamiya
    The Third Department of Internal Medicine, Nagoya University School of Medicine, Nagoya, Japan
    Diabetes Metab Res Rev 19:131-9. 2003
  7. ncbi request reprint [Development of therapeutic agents for diabetic neuropathies]
    Jiro Nakamura
    Division of Metabolic Diseases, Department of Internal Medicine, Nagoya University Graduate School of Medicine
    Nihon Rinsho 63:614-21. 2005
  8. ncbi request reprint Clinical potential of aldose reductase inhibitors in diabetic neuropathy
    Yoji Hamada
    Division of Metabolic Diseases, Nagoya University Graduate School of Medicine, Nagoya, Japan
    Treat Endocrinol 3:245-55. 2004
  9. ncbi request reprint [Pathogenesis and progression of neuropathies in diabetic patients with endothelial dysfunction]
    Keiko Naruse
    Department of Internal Medicine, Aichi Gakuin University School of Dentistry
    Nihon Rinsho 63:516-20. 2005
  10. ncbi request reprint [Role of polyol pathway in pathogenesis of diabetic microangiopathy]
    Jiro Nakamura
    Division of Metabolic Diseases, Department of Internal Medicine, Nagoya University Graduate School of Medicine
    Nihon Rinsho 63:52-61. 2005

Collaborators

  • Seikoh Horiuchi
  • Keiko Naruse
  • Yoji Hamada
  • Hideki Kamiya
  • Eitaro Nakashima
  • Nigishi Hotta
  • Weiguo Li
  • Koichi Kato
  • Kazuma Miwa
  • Yutaka Yasuda
  • Hiroko Hirooka
  • Yutaka Oiso
  • Naoki Takahashi
  • Noboru Akiyama
  • Yasuhide Kasuya

Detail Information

Publications13

  1. ncbi request reprint Augmentation of lipolysis in adipocytes from fed rats, but not from starved rats, by inhibition of rolipram-sensitive phosphodiesterase 4
    Jiro Nakamura
    Institute of Clinical Medicine, University of Tsukuba, Tsukuba shi, Ibaraki ken 305 8577, Japan
    Arch Biochem Biophys 425:106-14. 2004
    ..LY294002 accelerated protein kinase A activation. These data suggest that the rolipram-sensitive PDE4 is an anti-lipolytic enzyme expressed according to food intake. LY294002 may potentiate lipolysis through inhibition of the PDE4...
  2. doi request reprint Protein kinase CbetaI interacts with the beta1-adrenergic signaling pathway to attenuate lipolysis in rat adipocytes
    Jiro Nakamura
    Graduate School of Comprehensive Human Sciences, University of Tsukuba, Tsukuba shi, Ibaraki Ken, Japan
    Biochim Biophys Acta 1781:277-81. 2008
    ..These data suggest that PKCbetaI interacts with beta1-AR signaling and attenuates lipolysis in rat adipocytes...
  3. doi request reprint Phorbol 12-myristate 13-acetate inhibits the antilipolytic action of insulin, probably via the activity of protein kinase Cε
    Jiro Nakamura
    Graduate School of Comprehensive Human Sciences, University of Tsukuba, Tsukuba shi, Ibaraki Ken, 305 8575, Japan
    Eur J Pharmacol 648:188-94. 2010
    ..These data suggest that PMA inhibits the antilipolytic signal mediated by the insulin receptor, PI3-K, Akt, and PDE3B. The most probable target of PMA is PKCε...
  4. ncbi request reprint Protein kinase C-dependent antilipolysis by insulin in rat adipocytes
    Jiro Nakamura
    Graduate School of Comprehensive Human Sciences, University of Tsukuba, Tsukuba shi, Ibaraki Ken, 305 8575, Japan
    Biochim Biophys Acta 1771:1195-201. 2007
    ..These results indicate that stimulation of insulin receptor induces a sustained activation of PKC-dependent antilipolysis in rat adipocytes...
  5. ncbi request reprint Protein kinase C attenuates beta-adrenergic receptor-mediated lipolysis, probably through inhibition of the beta1-adrenergic receptor system
    Jiro Nakamura
    Graduate School of Comprehensive Human Sciences, University of Tsukuba, Tsukuba shi, Ibaraki ken 305 8575, Japan
    Arch Biochem Biophys 447:1-10. 2006
    ..These results indicate that conventional and novel PKCs attenuate lipolysis mediated by beta-adrenergic receptors, probably through inhibition of the beta1-adrenergic receptor system...
  6. ncbi request reprint Polyol pathway and protein kinase C activity of rat Schwannoma cells
    Hideki Kamiya
    The Third Department of Internal Medicine, Nagoya University School of Medicine, Nagoya, Japan
    Diabetes Metab Res Rev 19:131-9. 2003
    ..This study investigated the effects of high glucose and increased polyol pathway activity on neural cell growth and PKC activities...
  7. ncbi request reprint [Development of therapeutic agents for diabetic neuropathies]
    Jiro Nakamura
    Division of Metabolic Diseases, Department of Internal Medicine, Nagoya University Graduate School of Medicine
    Nihon Rinsho 63:614-21. 2005
  8. ncbi request reprint Clinical potential of aldose reductase inhibitors in diabetic neuropathy
    Yoji Hamada
    Division of Metabolic Diseases, Nagoya University Graduate School of Medicine, Nagoya, Japan
    Treat Endocrinol 3:245-55. 2004
    ....
  9. ncbi request reprint [Pathogenesis and progression of neuropathies in diabetic patients with endothelial dysfunction]
    Keiko Naruse
    Department of Internal Medicine, Aichi Gakuin University School of Dentistry
    Nihon Rinsho 63:516-20. 2005
  10. ncbi request reprint [Role of polyol pathway in pathogenesis of diabetic microangiopathy]
    Jiro Nakamura
    Division of Metabolic Diseases, Department of Internal Medicine, Nagoya University Graduate School of Medicine
    Nihon Rinsho 63:52-61. 2005
  11. ncbi request reprint [Effects of voglibose, alpha-glucosidase inhibitor in treatment of impaired glucose tolerance]
    Jiro Nakamura
    Division of Metabolic Diseases, Department of Internal Medicine, Nagoya University Graduate School of Medicine
    Nihon Rinsho 63:457-61. 2005
  12. ncbi request reprint Suppression of 3-deoxyglucosone and heparin-binding epidermal growth factor-like growth factor mRNA expression by an aldose reductase inhibitor in rat vascular smooth muscle cells
    Weiguo Li
    Division of Metabolic Diseases, Nagoya University Graduate School of Medicine, Nagoya, Japan
    Biochem Biophys Res Commun 314:370-6. 2004
    ..The results suggest that the polyol pathway may play a substantial role in SMC hyperplasia under hyperglycemic condition in part by affecting HB-EGF mRNA expression via the production of carbonyl compounds and oxidative stress...
  13. ncbi request reprint The role of polyol pathway in glucose-induced apoptosis of cultured retinal pericytes
    Kazuma Miwa
    The Third Department of Internal Medicine, Nagoya University School of Medicine, 65 Tsuruma cho, Showa Ku, Japan
    Diabetes Res Clin Pract 60:1-9. 2003
    ....