A Oka

Summary

Affiliation: Tottori University
Country: Japan

Publications

  1. ncbi The up-regulation of metabotropic glutamate receptor 5 (mGluR5) in Down's syndrome brains
    A Oka
    Department of Mental Retardation and Birth Defect Research, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan
    Acta Neuropathol 97:275-8. 1999
  2. ncbi Distribution and development of CLN2 protein, the late-infantile neuronal ceroid lipofuscinosis gene product
    Y Kurachi
    National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan
    Acta Neuropathol 102:20-6. 2001
  3. ncbi Rapid immunologic diagnosis of classic late infantile neuronal ceroid lipofuscinosis
    Y Kurachi
    Department of Mental Retardation and Birth Defect Research, National Institute of Neuroscience, National Center of Neurology and Psychiatry NCNP, Tokyo, Japan
    Neurology 54:1676-80. 2000
  4. ncbi The expression of late infantile neuronal ceroid lipofuscinosis (CLN2) gene product in human brains
    A Oka
    Department of Mental Retardation and Birth Defect Research, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan
    Neurosci Lett 257:113-5. 1998
  5. ncbi Vertebral artery dissection in an infant: ultrastructural aberrations in connective tissue components
    K Ogura
    Division of Child Neurology, Institute of Neurological Sciences, Faculty of Medicine, Tottori University, Tottori, Japan
    Neuropediatrics 34:307-10. 2003

Collaborators

Detail Information

Publications5

  1. ncbi The up-regulation of metabotropic glutamate receptor 5 (mGluR5) in Down's syndrome brains
    A Oka
    Department of Mental Retardation and Birth Defect Research, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan
    Acta Neuropathol 97:275-8. 1999
    ..Since group I mGluR regulates the metabolism of amyloid precursor protein (APP) and accelerates its processing into non-amyloidogenic APP, the overexpression of mGluR5 may be related to the pathological state of APP metabolism in DS...
  2. ncbi Distribution and development of CLN2 protein, the late-infantile neuronal ceroid lipofuscinosis gene product
    Y Kurachi
    National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan
    Acta Neuropathol 102:20-6. 2001
    ..Evaluation of the immunoreactivity of the CLN2 protein may be useful for characterization of a variant form...
  3. ncbi Rapid immunologic diagnosis of classic late infantile neuronal ceroid lipofuscinosis
    Y Kurachi
    Department of Mental Retardation and Birth Defect Research, National Institute of Neuroscience, National Center of Neurology and Psychiatry NCNP, Tokyo, Japan
    Neurology 54:1676-80. 2000
    ..To establish a new method for rapid diagnosis of late infantile neuronal ceroid lipofuscinosis (LINCL, CLN2) using specific polyclonal antibodies against the CLN2 gene product...
  4. ncbi The expression of late infantile neuronal ceroid lipofuscinosis (CLN2) gene product in human brains
    A Oka
    Department of Mental Retardation and Birth Defect Research, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan
    Neurosci Lett 257:113-5. 1998
    ..Our results suggest the usefulness of these antibodies for the diagnosis of CLN2, which currently requires demonstration of characteristic ultrastructure by electron microscopy...
  5. ncbi Vertebral artery dissection in an infant: ultrastructural aberrations in connective tissue components
    K Ogura
    Division of Child Neurology, Institute of Neurological Sciences, Faculty of Medicine, Tottori University, Tottori, Japan
    Neuropediatrics 34:307-10. 2003
    ..Arterial dissection should be considered as a cause of stroke during infancy...