Maki Tateyama

Summary

Affiliation: Tohoku University
Country: Japan

Publications

  1. doi Dendritic cells in muscle lesions of sarcoidosis
    Maki Tateyama
    Department of Neurology, Tohoku University School of Medicine, Sendai 980 8574, Japan
    Hum Pathol 42:340-6. 2011
  2. doi CCR7+ myeloid dendritic cells together with CCR7+ T cells and CCR7+ macrophages invade CCL19+ nonnecrotic muscle fibers in inclusion body myositis
    Maki Tateyama
    Department of Neurology, Tohoku University School of Medicine, Sendai, Japan
    J Neurol Sci 279:47-52. 2009
  3. ncbi Expression of CCR7 and its ligands CCL19/CCL21 in muscles of polymyositis
    Maki Tateyama
    Department of Neurology, Tohoku University School of Medicine, 1 1 Seiryo machi, Sendai 980 8574, Japan
    J Neurol Sci 249:158-65. 2006
  4. ncbi Expression profiling with progression of dystrophic change in dysferlin-deficient mice (SJL)
    Naoki Suzuki
    Department of Neurology, Tohoku University School of Medicine, 1 1 Seiryo machi, Sendai 980 8574, Japan
    Neurosci Res 52:47-60. 2005
  5. doi Continuous administration of poloxamer 188 reduces overload-induced muscular atrophy in dysferlin-deficient SJL mice
    Naoki Suzuki
    Department of Neurology, Tohoku University School of Medicine, 1 1 Seiryo machi, Aoba ku, Sendai, Japan
    Neurosci Res 72:181-6. 2012
  6. ncbi Late-onset distal myopathy with rimmed vacuoles without mutation in the GNE or dysferlin genes
    Naoki Suzuki
    Department of Neurology, Tohoku University School of Medicine, Seiryo machi, Sendai 980 8574, Japan
    Muscle Nerve 32:812-4. 2005
  7. ncbi Familial inclusion body myositis: a report on two Japanese sisters
    Maki Tateyama
    Department of Neurology, Tohoku University School of Medicine, Sendai
    Intern Med 42:1035-8. 2003
  8. doi Exome sequencing identifies a novel TTN mutation in a family with hereditary myopathy with early respiratory failure
    Rumiko Izumi
    Department of Medical Genetics, Tohoku University School of Medicine, Sendai, Japan
    J Hum Genet 58:259-66. 2013
  9. pmc Clinical features and a mutation with late onset of limb girdle muscular dystrophy 2B
    Toshiaki Takahashi
    Department of Neurology, Tohoku University School of Medicine, 1 1 Seiryo machi, Sendai 980 8574, Japan
    J Neurol Neurosurg Psychiatry 84:433-40. 2013
  10. pmc Two cases of elderly-onset hereditary neuropathy with liability to pressure palsy manifesting bilateral peroneal nerve palsies
    Norihiko Kawaguchi
    Department of Neurology, School of Medicine, Tohoku University, Sendai, Japan
    Case Rep Neurol 4:149-55. 2012

Collaborators

Detail Information

Publications19

  1. doi Dendritic cells in muscle lesions of sarcoidosis
    Maki Tateyama
    Department of Neurology, Tohoku University School of Medicine, Sendai 980 8574, Japan
    Hum Pathol 42:340-6. 2011
    ....
  2. doi CCR7+ myeloid dendritic cells together with CCR7+ T cells and CCR7+ macrophages invade CCL19+ nonnecrotic muscle fibers in inclusion body myositis
    Maki Tateyama
    Department of Neurology, Tohoku University School of Medicine, Sendai, Japan
    J Neurol Sci 279:47-52. 2009
    ..The chemokine mediated attraction in dendritic and other immune cells and muscle cells may be crucial in sustained antigen presentation, T cell activation and immune attack to muscles in the pathogenesis of IBM...
  3. ncbi Expression of CCR7 and its ligands CCL19/CCL21 in muscles of polymyositis
    Maki Tateyama
    Department of Neurology, Tohoku University School of Medicine, 1 1 Seiryo machi, Sendai 980 8574, Japan
    J Neurol Sci 249:158-65. 2006
    ..The CCL19,CCL21/CCR7 chemokine system is expressed in inflamed muscles of polymyositis and may be involved in the pathomechanism of polymyositis...
  4. ncbi Expression profiling with progression of dystrophic change in dysferlin-deficient mice (SJL)
    Naoki Suzuki
    Department of Neurology, Tohoku University School of Medicine, 1 1 Seiryo machi, Sendai 980 8574, Japan
    Neurosci Res 52:47-60. 2005
    ..We must reveal some other molecules which may work with dysferlin in order to clarify the pathological network of dysferlinopathy. This process may lead to future improvements in the therapy for human dysferlinopathy...
  5. doi Continuous administration of poloxamer 188 reduces overload-induced muscular atrophy in dysferlin-deficient SJL mice
    Naoki Suzuki
    Department of Neurology, Tohoku University School of Medicine, 1 1 Seiryo machi, Aoba ku, Sendai, Japan
    Neurosci Res 72:181-6. 2012
    ..These results provide a basis for potential treatments for dysferlin-deficient skeletal muscle fibers...
  6. ncbi Late-onset distal myopathy with rimmed vacuoles without mutation in the GNE or dysferlin genes
    Naoki Suzuki
    Department of Neurology, Tohoku University School of Medicine, Seiryo machi, Sendai 980 8574, Japan
    Muscle Nerve 32:812-4. 2005
    ..We speculate that dysferlin is involved in the pathogenesis of the myopathy in these patients, which may represent a new disease entity presenting as a distal myopathy...
  7. ncbi Familial inclusion body myositis: a report on two Japanese sisters
    Maki Tateyama
    Department of Neurology, Tohoku University School of Medicine, Sendai
    Intern Med 42:1035-8. 2003
    ..One sister received corticosteroid and azathioprine and the therapy relieved dysphagia. Both of our patients had DR15(2)/4 (DRB1*1502/0405), suggesting a distinct genetic association with the disease in the Japanese population...
  8. doi Exome sequencing identifies a novel TTN mutation in a family with hereditary myopathy with early respiratory failure
    Rumiko Izumi
    Department of Medical Genetics, Tohoku University School of Medicine, Sendai, Japan
    J Hum Genet 58:259-66. 2013
    ..It is possible that focused analysis of TTN may detect more mutations in patients with MFMs, especially in those with early respiratory failure...
  9. pmc Clinical features and a mutation with late onset of limb girdle muscular dystrophy 2B
    Toshiaki Takahashi
    Department of Neurology, Tohoku University School of Medicine, 1 1 Seiryo machi, Sendai 980 8574, Japan
    J Neurol Neurosurg Psychiatry 84:433-40. 2013
    ..It is important that the late onset phenotype is found with prevalent mutations...
  10. pmc Two cases of elderly-onset hereditary neuropathy with liability to pressure palsy manifesting bilateral peroneal nerve palsies
    Norihiko Kawaguchi
    Department of Neurology, School of Medicine, Tohoku University, Sendai, Japan
    Case Rep Neurol 4:149-55. 2012
    ..Their clinical courses suggested that the Japanese sitting style termed 'seiza', a way of sitting on the floor with the lower legs crossed under the thighs, was a precipitating factor for the bilateral peroneal nerve palsies...
  11. ncbi A case of late onset riboflavin-responsive multiple acyl-CoA dehydrogenase deficiency manifesting as recurrent rhabdomyolysis and acute renal failure
    Rumiko Izumi
    Department of Neurology, Tohoku University School of Medicine, Japan
    Intern Med 50:2663-8. 2011
    ..Gene analysis is useful to confirm the diagnosis, and early diagnosis is important because riboflavin treatment has been effective in a significant number of patients with MADD...
  12. ncbi [Elevated serum aldolase activity in a patient of non-eosinophilic myofasciitis and synovitis with perifascicular atrophy]
    Hirohiko Ono
    Department of Neurology, Tohoku University School of Medicine
    Rinsho Shinkeigaku 49:119-22. 2009
    ..We diagnosed him as non-eosinophilic myofasciitis and synovitis with perifascicular atrophy. The serum aldolase activity is usuful for diagnosis and for monitoring the disease activity...
  13. ncbi Relapsing focal myositis: the localization detected by gallium citrate Ga 67 scintigraphy
    Tatsuro Misu
    Department of Neurology, Tohoku University Graduate School of Medicine, Sendai, Japan
    Arch Neurol 62:1930-1. 2005
  14. ncbi Novel dysferlin mutations and characteristic muscle atrophy in late-onset Miyoshi myopathy
    Naoki Suzuki
    Department of Neurology, Tohoku University School of Medicine, 1 1 Seiryo machi, Sendai 980 8574, Japan
    Muscle Nerve 29:721-3. 2004
    ..Even older patients with atrophy in the posterior compartment of the distal lower extremities and a relatively high serum creatine kinase level should be examined for the dysferlin gene...
  15. ncbi Expression of OX40 in muscles of polymyositis and granulomatous myopathy
    Maki Tateyama
    Department of Neurology, Tohoku University School of Medicine, 1 1, Seiryo machi, Aoba ku, 980 8574, Sendai, Japan
    J Neurol Sci 194:29-34. 2002
    ..OX40+ mononuclear cells with specific distributions in muscles may be involved in the pathomechanism of polymyositis and granulomatous myopathy, and can be a candidate molecule of selective immunotherapy in these diseases...
  16. ncbi [Pathomechanism and prevalence of sporadic inclusion body myositis (sIBM)]
    Naoki Suzuki
    Department of Neurology, Tohoku University School of Medicine
    Rinsho Shinkeigaku 51:964-6. 2011
    ..To elucidate the pathomechanism of sIBM is the most important way to make therapeutic approach. In this review we estimated the prevalence of sIBM in Japan and discuss the pathomechanism of sIBM...
  17. ncbi A case of McArdle disease: efficacy of vitamin B6 on fatigability and impaired glycogenolysis
    Rumiko Izumi
    Department of Neurology, Tohoku University School of Medicine, Sendai
    Intern Med 49:1623-5. 2010
    ..After administration of vitamin B6, fatigability was diminished and ischemic forearm exercise test showed improved glycogenolysis. Vitamin B6 may be beneficial for McArdle disease, especially for its easy fatigability...
  18. ncbi [Radial nerve palsy as a presenting feature of neuralgic amyotrophy]
    Hirofumi Tsuboi
    Department of Neurology, Tohoku University School of Medicine
    Rinsho Shinkeigaku 53:312-5. 2013
    ..Neuralgic amyotrophy should be kept in mind in diagnosing acute onset, painful radial palsy...
  19. ncbi [A case of recurrent encephalomyelitis associated with eosinophilia in CSF]
    Jun Suzuki
    Department of Neurology, Tohoku University School of Medicine
    Rinsho Shinkeigaku 52:571-5. 2012
    ..The remarkable responses to steroids without any additional therapy, compatible with idiopathic eosinophilic syndromes, confirmed that this was a case of idiopathic eosinophilic recurrent encephalomyelitis...