Katsuya Endo

Summary

Affiliation: Tohoku University
Country: Japan

Publications

  1. ncbi Involvement of NF-kappa B pathway in TL1A gene expression induced by lipopolysaccharide
    Katsuya Endo
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, Sendai, Japan
    Cytokine 49:215-20. 2010
  2. ncbi Increased expression of NKX2.3 mRNA transcribed from the risk haplotype for ulcerative colitis in the involved colonic mucosa
    Takashi Arai
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, Sendai 980 8574, Japan
    Hum Immunol 72:587-91. 2011
  3. ncbi Changes of faecal microbiota in patients with Crohn's disease treated with an elemental diet and total parenteral nutrition
    Hisashi Shiga
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, Sendai 980 8574, Japan
    Dig Liver Dis 44:736-42. 2012
  4. ncbi TNFSF15 transcripts from risk haplotype for Crohn's disease are overexpressed in stimulated T cells
    Yoichi Kakuta
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, Aoba, Sendai 980 8574, Japan
    Hum Mol Genet 18:1089-98. 2009
  5. ncbi FCGR3A-158 polymorphism influences the biological response to infliximab in Crohn's disease through affecting the ADCC activity
    Rintaro Moroi
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, Aoba ku, Sendai, Japan
    Immunogenetics 65:265-71. 2013
  6. ncbi Two cases of diffuse duodenitis associated with ulcerative colitis
    Katsuya Endo
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, 1 1 Seiryo, Aoba ku, Sendai 980 8574, Japan
    Case Rep Gastrointest Med 2012:396521. 2012
  7. ncbi Short and long-term outcomes of endoscopic balloon dilatation for Crohn's disease strictures
    Katsuya Endo
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, Sendai 980 8574, Japan
    World J Gastroenterol 19:86-91. 2013
  8. ncbi IL-10 deficiency leads to somatic mutations in a model of IBD
    Yuichirou Sato
    Department of Gastroenterology, Tohoku University Graduate School of Medicine, Sendai, Japan
    Carcinogenesis 27:1068-73. 2006
  9. ncbi [Inflammatory bowel disease: IBD]
    Katsuya Endo
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, Sendai 980 8574, Japan
    Rinsho Byori 57:527-32. 2009

Collaborators

Detail Information

Publications9

  1. ncbi Involvement of NF-kappa B pathway in TL1A gene expression induced by lipopolysaccharide
    Katsuya Endo
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, Sendai, Japan
    Cytokine 49:215-20. 2010
    ..To determine the immunological contribution of TL1A to CD, it is essential to investigate the transcriptional regulation of TL1A...
  2. ncbi Increased expression of NKX2.3 mRNA transcribed from the risk haplotype for ulcerative colitis in the involved colonic mucosa
    Takashi Arai
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, Sendai 980 8574, Japan
    Hum Immunol 72:587-91. 2011
    ..The demonstrated allelic expression imbalance supports the idea that the risk haplotype of NKX2.3 confers susceptibility to UC through increasing expression of NKX2.3 mRNA in the colonic mucosa...
  3. ncbi Changes of faecal microbiota in patients with Crohn's disease treated with an elemental diet and total parenteral nutrition
    Hisashi Shiga
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, Sendai 980 8574, Japan
    Dig Liver Dis 44:736-42. 2012
    ..Elemental diet and total parenteral nutrition are effective therapies for Crohn's disease; however, changes of microbiota as a result of both treatments have not been fully elucidated...
  4. ncbi TNFSF15 transcripts from risk haplotype for Crohn's disease are overexpressed in stimulated T cells
    Yoichi Kakuta
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, Aoba, Sendai 980 8574, Japan
    Hum Mol Genet 18:1089-98. 2009
    ..The promoter activity of -358C (risk allele) was higher than that of -358T (non-risk allele) in stimulated T cells. This effect of -358T/C on the transcriptional activity in stimulated T cells may confer susceptibility to CD...
  5. ncbi FCGR3A-158 polymorphism influences the biological response to infliximab in Crohn's disease through affecting the ADCC activity
    Rintaro Moroi
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, Aoba ku, Sendai, Japan
    Immunogenetics 65:265-71. 2013
    ....
  6. ncbi Two cases of diffuse duodenitis associated with ulcerative colitis
    Katsuya Endo
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, 1 1 Seiryo, Aoba ku, Sendai 980 8574, Japan
    Case Rep Gastrointest Med 2012:396521. 2012
    ..Further studies involving a large number of patients are needed to clarify whether the upper gastrointestinal tract is a target organ in UC...
  7. ncbi Short and long-term outcomes of endoscopic balloon dilatation for Crohn's disease strictures
    Katsuya Endo
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, Sendai 980 8574, Japan
    World J Gastroenterol 19:86-91. 2013
    ..To investigate the short and long-term outcomes of endoscopic balloon dilatation (EBD) for Crohn's disease (CD) strictures...
  8. ncbi IL-10 deficiency leads to somatic mutations in a model of IBD
    Yuichirou Sato
    Department of Gastroenterology, Tohoku University Graduate School of Medicine, Sendai, Japan
    Carcinogenesis 27:1068-73. 2006
    ..Our data demonstrate that the chronic inflammatory environment in the colon promotes the generation of mutations...
  9. ncbi [Inflammatory bowel disease: IBD]
    Katsuya Endo
    Division of Gastroenterology, Tohoku University Graduate School of Medicine, Sendai 980 8574, Japan
    Rinsho Byori 57:527-32. 2009
    ..Recently, molecular approaches that facilitate evaluation of alterations in non-cultivatable intestinal bacteria in IBD patients have supported the latter hypothesis...