Affiliation: Showa University
- Inhibition of NAD(P)H oxidase reduces apoptosis and avascular retina in an animal model of retinopathy of prematurityYuta Saito
Department of Ophthalmology, University of North Carolina, Chapel Hill, NC 27599 7041, USA
Mol Vis 13:840-53. 2007....
- Triamcinolone reduces neovascularization, capillary density and IGF-1 receptor phosphorylation in a model of oxygen-induced retinopathyM Elizabeth Hartnett
Department of Ophthalmology, University of North Carolina, 6109A Neuroscience Research Building, Chapel Hill, NC 27599, USA
Invest Ophthalmol Vis Sci 47:4975-82. 2006..To study the effects of intravitreous triamcinolone acetonide (TA) on neovascularization (NV), capillary density, and retinal endothelial cell (REC) viability in a model of oxygen-induced retinopathy (OIR)...
- Effect of a dihydrobenzofuran derivative on lipid hydroperoxide-induced rabbit corneal neovascularizationHirotsugu Ogura
Department of Ophthalmology, School of Medicine, Showa University, Tokyo 142 8555, Japan
J Pharmacol Sci 103:234-40. 2007..These results indicate that LHP-induced corneal NV is inhibited by treatment with A-3922 and therefore may represent a potential pharmacological intervention for ocular neovascularization disorders...
- Activated NAD(P)H oxidase from supplemental oxygen induces neovascularization independent of VEGF in retinopathy of prematurity modelYuta Saito
Department of Ophthalmology, Showa University, Tokyo, Japan
Invest Ophthalmol Vis Sci 49:1591-8. 2008..To study NAD(P)H oxidase-dependent outcomes after oxygen stresses that are similar to those experienced by preterm infants today using a rat model of retinopathy of prematurity...