Shinji Inomata

Summary

Affiliation: Shiseido Research Center
Country: Japan

Publications

  1. ncbi request reprint Possible involvement of gelatinases in basement membrane damage and wrinkle formation in chronically ultraviolet B-exposed hairless mouse
    Shinji Inomata
    Skincare Ingredient Research Laboratories, Shiseido Material Development Research Center, 2 2 1 Hayabuchi, Tsuzuki ku, Yokohama 224 8558, Japan
    J Invest Dermatol 120:128-34. 2003
  2. ncbi request reprint Non-invasive study of gelatinases in sun-exposed and unexposed healthy human skin based on measurements in stratum corneum
    Keiko Takada
    Shiseido Research Center, 2 2 1 Hayabuchi, Tsuzuki ku, Yokohama, 224 8558, Japan
    Arch Dermatol Res 298:237-42. 2006
  3. ncbi request reprint gamma-Aminobutyric acid (A) receptor agonists accelerate cutaneous barrier recovery and prevent epidermal hyperplasia induced by barrier disruption
    Mitsuhiro Denda
    Shiseido Research Center, Fukuura, Kanazawa Ku, Yokohama, Japan
    J Invest Dermatol 119:1041-7. 2002
  4. doi request reprint Coenzyme Q10 protects against oxidative stress-induced cell death and enhances the synthesis of basement membrane components in dermal and epidermal cells
    Keiko Muta-Takada
    Shiseido Research Center, Yokohama, Japan
    Biofactors 35:435-41. 2009
  5. ncbi request reprint Unsaturated fatty acids induce calcium influx into keratinocytes and cause abnormal differentiation of epidermis
    Yuji Katsuta
    Shiseido Research Center, Yokohama, Japan
    J Invest Dermatol 124:1008-13. 2005
  6. ncbi request reprint trans-4-(Aminomethyl)cyclohexane carboxylic acid methylamide (t-AMCHA methylamide) inhibits the physical interaction between urokinase-type plasminogen activator and stratum corneum, and accelerates the recovery of barrier function
    Yuji Katsuta
    J Dermatol Sci 40:218-20. 2005

Detail Information

Publications6

  1. ncbi request reprint Possible involvement of gelatinases in basement membrane damage and wrinkle formation in chronically ultraviolet B-exposed hairless mouse
    Shinji Inomata
    Skincare Ingredient Research Laboratories, Shiseido Material Development Research Center, 2 2 1 Hayabuchi, Tsuzuki ku, Yokohama 224 8558, Japan
    J Invest Dermatol 120:128-34. 2003
    ....
  2. ncbi request reprint Non-invasive study of gelatinases in sun-exposed and unexposed healthy human skin based on measurements in stratum corneum
    Keiko Takada
    Shiseido Research Center, 2 2 1 Hayabuchi, Tsuzuki ku, Yokohama, 224 8558, Japan
    Arch Dermatol Res 298:237-42. 2006
    ..g., by promoting wrinkle formation...
  3. ncbi request reprint gamma-Aminobutyric acid (A) receptor agonists accelerate cutaneous barrier recovery and prevent epidermal hyperplasia induced by barrier disruption
    Mitsuhiro Denda
    Shiseido Research Center, Fukuura, Kanazawa Ku, Yokohama, Japan
    J Invest Dermatol 119:1041-7. 2002
    ..These results suggest that the gamma-aminobutyric acid (A)-like receptor is associated with skin barrier homeostasis and regulation of the receptor clinically effective for barrier dysfunctional or epidermal hyperproliferative diseases...
  4. doi request reprint Coenzyme Q10 protects against oxidative stress-induced cell death and enhances the synthesis of basement membrane components in dermal and epidermal cells
    Keiko Muta-Takada
    Shiseido Research Center, Yokohama, Japan
    Biofactors 35:435-41. 2009
    ..These results suggest that protection of epidermis against oxidative stress and enhancement of production of epidermal basement membrane components may be involved in the antiaging properties of CoQ10 in skin...
  5. ncbi request reprint Unsaturated fatty acids induce calcium influx into keratinocytes and cause abnormal differentiation of epidermis
    Yuji Katsuta
    Shiseido Research Center, Yokohama, Japan
    J Invest Dermatol 124:1008-13. 2005
    ..These results suggest that unsaturated fatty acids in sebum alter the calcium dynamics in epidermal keratinocytes and induce abnormal follicular keratinization...
  6. ncbi request reprint trans-4-(Aminomethyl)cyclohexane carboxylic acid methylamide (t-AMCHA methylamide) inhibits the physical interaction between urokinase-type plasminogen activator and stratum corneum, and accelerates the recovery of barrier function
    Yuji Katsuta
    J Dermatol Sci 40:218-20. 2005