S Yoshino

Summary

Affiliation: Saga Medical School
Country: Japan

Publications

  1. ncbi request reprint Effect of a monoclonal antibody against interleukin-4 on the induction of oral tolerance in mice
    S Yoshino
    Department of Microbiology, Saga Medical School, Japan
    Eur J Pharmacol 336:203-9. 1997
  2. ncbi request reprint Augmented antibody-mediated footpad responses in mice treated with an anti-IL-4 monoclonal antibody
    S Yoshino
    Department of Microbiology, Saga Medical School, Japan
    Immunopharmacology 37:245-50. 1997
  3. ncbi request reprint Treatment with an anti-IL-4 monoclonal antibody blocks suppression of collagen-induced arthritis in mice by oral administration of type II collagen
    S Yoshino
    Department of Microbiology, Saga Medical School, Japan
    J Immunol 160:3067-71. 1998
  4. ncbi request reprint Enhancement of collagen-induced arthritis in mice by diesel exhaust particles
    S Yoshino
    Department of Microbiology, Saga Medical School, Saga 849 8501, Japan
    J Pharmacol Exp Ther 290:524-9. 1999
  5. ncbi request reprint Oral administration of lipopolysaccharide exacerbates collagen-induced arthritis in mice
    S Yoshino
    Department of Microbiology, Saga Medical School, Japan
    J Immunol 163:3417-22. 1999
  6. pmc The role of lipopolysaccharide injected systemically in the reactivation of collagen-induced arthritis in mice
    S Yoshino
    Department of Microbiology, Saga Medical School, 5 1 1 Nabeshima, Saga 849 8501, Japan
    Br J Pharmacol 129:1309-14. 2000

Detail Information

Publications6

  1. ncbi request reprint Effect of a monoclonal antibody against interleukin-4 on the induction of oral tolerance in mice
    S Yoshino
    Department of Microbiology, Saga Medical School, Japan
    Eur J Pharmacol 336:203-9. 1997
    ..Thus, the neutralization of interleukin-4 by anti-interleukin-4 antibodies appears to be effective in modulating the induction of oral tolerance...
  2. ncbi request reprint Augmented antibody-mediated footpad responses in mice treated with an anti-IL-4 monoclonal antibody
    S Yoshino
    Department of Microbiology, Saga Medical School, Japan
    Immunopharmacology 37:245-50. 1997
    ..Thus, treatment with anti-IL-4 antibodies appears to be effective in augmenting antibody-mediated in vivo responses in which antigen-specific IgG2a antibodies and IFN-gamma produced following the antibody treatment play a role...
  3. ncbi request reprint Treatment with an anti-IL-4 monoclonal antibody blocks suppression of collagen-induced arthritis in mice by oral administration of type II collagen
    S Yoshino
    Department of Microbiology, Saga Medical School, Japan
    J Immunol 160:3067-71. 1998
    ..Thus, the neutralization of IL-4 by an anti-IL-4 Ab appears to be effective in blocking suppression of CIA by oral administration of CII, suggesting that IL-4 may be critically involved in its suppression...
  4. ncbi request reprint Enhancement of collagen-induced arthritis in mice by diesel exhaust particles
    S Yoshino
    Department of Microbiology, Saga Medical School, Saga 849 8501, Japan
    J Pharmacol Exp Ther 290:524-9. 1999
    ..These results suggest that exposure to DEP may influence autoimmune disease...
  5. ncbi request reprint Oral administration of lipopolysaccharide exacerbates collagen-induced arthritis in mice
    S Yoshino
    Department of Microbiology, Saga Medical School, Japan
    J Immunol 163:3417-22. 1999
    ..These findings suggest that LPS from intestinal bacteria may play a role in the exacerbation of autoimmune joint inflammation...
  6. pmc The role of lipopolysaccharide injected systemically in the reactivation of collagen-induced arthritis in mice
    S Yoshino
    Department of Microbiology, Saga Medical School, 5 1 1 Nabeshima, Saga 849 8501, Japan
    Br J Pharmacol 129:1309-14. 2000
    ..4. These results suggest that LPS may play an important role in the reactivation of autoimmune joint inflammatory diseases such as rheumatoid arthritis in humans...