Tetsuya Tagami

Summary

Affiliation: National Hospital Organization
Country: Japan

Publications

  1. ncbi request reprint Short-term effects of β-adrenergic antagonists and methimazole in new-onset thyrotoxicosis caused by Graves' disease
    Tetsuya Tagami
    Clinical Research Institute, Division of Endocrinology and Metabolism, National Hospital Organization Kyoto Medical Center, Japan
    Intern Med 51:2285-90. 2012
  2. doi request reprint A selective peroxisome proliferator-activated receptor-gamma modulator, telmisartan, binds to the receptor in a different fashion from thiazolidinediones
    Tetsuya Tagami
    Clinical Research Institute, Kyoto Medical Center, National Hospital Organization, Fukakusa, Kyoto, Japan
    Endocrinology 150:862-70. 2009
  3. doi request reprint The retinoid X receptor binding to the thyroid hormone receptor: relationship with cofactor binding and transcriptional activity
    Tetsuya Tagami
    Department of Endocrinology and Metabolism, Clinical Research Institute, Kyoto Medical Center, National Hospital Organization, Mukaihata cho, Fukakusa, Fushimi ku, Japan
    J Mol Endocrinol 42:415-28. 2009
  4. ncbi request reprint The incidence of gestational hyperthyroidism and postpartum thyroiditis in treated patients with Graves' disease
    Tetsuya Tagami
    Clinical Research Institute, Division of Endocrinology and Metabolism, Kyoto Medical Center, National Hospital Organization, Kyoto 612 8555, Japan
    Thyroid 17:767-72. 2007
  5. ncbi request reprint Lipid profiles in the untreated patients with Hashimoto thyroiditis and the effects of thyroxine treatment on subclinical hypothyroidism with Hashimoto thyroiditis
    Tetsuya Tagami
    Division of Endocrinology and Metabolism, Clinical Research Institute, National Hospital Organization Kyoto Medical Center, Kyoto, Japan
    Endocr J 57:253-8. 2010
  6. doi request reprint Identification of a novel human thyroid hormone receptor beta isoform as a transcriptional modulator
    Tetsuya Tagami
    Division of Endocrinology, Metabolism and Hypertension, Clinical Research Institute, Kyoto Medical Center, National Hospital Organization, Kyoto 612 8555, Japan
    Biochem Biophys Res Commun 396:983-8. 2010
  7. doi request reprint Aberrant expression of thyroid hormone receptor beta isoform may cause inappropriate secretion of TSH in a TSH-secreting pituitary adenoma
    Tetsuya Tagami
    Division of Endocrinology and Metabolism, Clinical Research Institute, National Hospital Organization Kyoto Medical Center, Mukaihata cho 1 1, Fukakusa, Fushimi ku, Kyoto 612 8555, Japan
    J Clin Endocrinol Metab 96:E948-52. 2011
  8. ncbi request reprint Multi-center study on the prevalence of hypothyroidism in patients with hypercholesterolemia
    Tetsuya Tagami
    Clinical Research Institute, Division of Endocrinology and Metabolism, National Hospital Organization Kyoto Medical Center, Kyoto, Japan
    Endocr J 58:449-57. 2011
  9. ncbi request reprint Adiponectin in anorexia nervosa and bulimia nervosa
    Tetsuya Tagami
    Clinical Research Institute, Center for Endocrine and Metabolic Diseases, Kyoto National Hospital, Kyoto 612 8555, Japan
    J Clin Endocrinol Metab 89:1833-7. 2004
  10. ncbi request reprint Leptin-to-adiponectin ratio as a potential atherogenic index in obese type 2 diabetic patients
    Noriko Satoh
    Clinical Research Institute for Endocrine Metabolic Disease, Kyoto Medical Center, Japan
    Diabetes Care 27:2488-90. 2004

Collaborators

Detail Information

Publications31

  1. ncbi request reprint Short-term effects of β-adrenergic antagonists and methimazole in new-onset thyrotoxicosis caused by Graves' disease
    Tetsuya Tagami
    Clinical Research Institute, Division of Endocrinology and Metabolism, National Hospital Organization Kyoto Medical Center, Japan
    Intern Med 51:2285-90. 2012
    ..The objective is to elucidate the effects of β-blockers on anti-thyroid drug therapy in GD...
  2. doi request reprint A selective peroxisome proliferator-activated receptor-gamma modulator, telmisartan, binds to the receptor in a different fashion from thiazolidinediones
    Tetsuya Tagami
    Clinical Research Institute, Kyoto Medical Center, National Hospital Organization, Fukakusa, Kyoto, Japan
    Endocrinology 150:862-70. 2009
    ....
  3. doi request reprint The retinoid X receptor binding to the thyroid hormone receptor: relationship with cofactor binding and transcriptional activity
    Tetsuya Tagami
    Department of Endocrinology and Metabolism, Clinical Research Institute, Kyoto Medical Center, National Hospital Organization, Mukaihata cho, Fukakusa, Fushimi ku, Japan
    J Mol Endocrinol 42:415-28. 2009
    ....
  4. ncbi request reprint The incidence of gestational hyperthyroidism and postpartum thyroiditis in treated patients with Graves' disease
    Tetsuya Tagami
    Clinical Research Institute, Division of Endocrinology and Metabolism, Kyoto Medical Center, National Hospital Organization, Kyoto 612 8555, Japan
    Thyroid 17:767-72. 2007
    ..In both situations, changes of TRAb were also valuable in differentiating the recurrence of GD from GTT or PTT...
  5. ncbi request reprint Lipid profiles in the untreated patients with Hashimoto thyroiditis and the effects of thyroxine treatment on subclinical hypothyroidism with Hashimoto thyroiditis
    Tetsuya Tagami
    Division of Endocrinology and Metabolism, Clinical Research Institute, National Hospital Organization Kyoto Medical Center, Kyoto, Japan
    Endocr J 57:253-8. 2010
    ..In conclusion, the prevalence of dyslipidemia increases along with hypofunction of the thyroid and T4 replacement therapy may improve lipid profile in the cases of SH with Hashimoto thyroiditis...
  6. doi request reprint Identification of a novel human thyroid hormone receptor beta isoform as a transcriptional modulator
    Tetsuya Tagami
    Division of Endocrinology, Metabolism and Hypertension, Clinical Research Institute, Kyoto Medical Center, National Hospital Organization, Kyoto 612 8555, Japan
    Biochem Biophys Res Commun 396:983-8. 2010
    ..TR beta 4 weakly but significantly inhibited transcription mediated by functional TR. Thus, this novel isoform may modulate hormone action as an endogenous antagonist in the tissue or cellular context...
  7. doi request reprint Aberrant expression of thyroid hormone receptor beta isoform may cause inappropriate secretion of TSH in a TSH-secreting pituitary adenoma
    Tetsuya Tagami
    Division of Endocrinology and Metabolism, Clinical Research Institute, National Hospital Organization Kyoto Medical Center, Mukaihata cho 1 1, Fukakusa, Fushimi ku, Kyoto 612 8555, Japan
    J Clin Endocrinol Metab 96:E948-52. 2011
    ..The mechanism of a defect in negative regulation of TSH in a TSHoma is still unclear...
  8. ncbi request reprint Multi-center study on the prevalence of hypothyroidism in patients with hypercholesterolemia
    Tetsuya Tagami
    Clinical Research Institute, Division of Endocrinology and Metabolism, National Hospital Organization Kyoto Medical Center, Kyoto, Japan
    Endocr J 58:449-57. 2011
    ....
  9. ncbi request reprint Adiponectin in anorexia nervosa and bulimia nervosa
    Tetsuya Tagami
    Clinical Research Institute, Center for Endocrine and Metabolic Diseases, Kyoto National Hospital, Kyoto 612 8555, Japan
    J Clin Endocrinol Metab 89:1833-7. 2004
    ..These findings suggest that abnormal feeding behavior in the patients with eating disorders may reduce circulating adiponectin level, and weight recovery can restore it...
  10. ncbi request reprint Leptin-to-adiponectin ratio as a potential atherogenic index in obese type 2 diabetic patients
    Noriko Satoh
    Clinical Research Institute for Endocrine Metabolic Disease, Kyoto Medical Center, Japan
    Diabetes Care 27:2488-90. 2004
  11. doi request reprint Histopathological diagnosis of primary aldosteronism using CYP11B2 immunohistochemistry
    Kazutaka Nanba
    Department of Endocrinology, Metabolism, and Hypertension, National Hospital Organization Kyoto Medical Center, Fushimi ku, Kyoto, Japan
    J Clin Endocrinol Metab 98:1567-74. 2013
    ..Although primary aldosteronism (PA) is the most common cause of endocrine hypertension, histopathological methods to reveal the presence and sites of aldosterone overproduction remain to be established...
  12. ncbi request reprint Antithyroid drugs inhibit thyroid hormone receptor-mediated transcription
    Kenji Moriyama
    Division of Endocrinology and Metabolism, Clinical Research Institute, Kyoto Medical Center, National Hospital Organization, Kyoto 612 8555, Japan
    J Clin Endocrinol Metab 92:1066-72. 2007
    ..ATDs may also interfere with T(3) binding to nuclear thyroid hormone receptors (TRs). However, the effect of ATDs on the transcriptional activities of T(3) mediated by TRs has not been studied...
  13. doi request reprint Confirmatory testing in primary aldosteronism
    Kazutaka Nanba
    Department of Endocrinology, Metabolism, and Hypertension, National Hospital Organization Kyoto Medical Center, 1 1 Mukaihata cho, Fukakusa, Fushimi ku, Kyoto 612 8555, Japan
    J Clin Endocrinol Metab 97:1688-94. 2012
    ..Although confirmatory testing to verify aldosterone excess is a key step in the diagnosis of primary aldosteronism (PA), there is no consensus as to whether it is always needed and which of the tests need to be performed...
  14. doi request reprint Serum levels of IgG and IgG4 in Hashimoto thyroiditis
    Sachiko Tsukamoto Kawashima
    Division of Endocrinology and Metabolism, National Hospital Organization Kyoto Medical Center, Mukaihata cho 1 1, Fukakusa, Fushimi ku, Kyoto, 612 8555, Japan
    Endocrine 45:236-43. 2014
    ..In conclusion, at least a small portion of patients with HT with high titers of anti-thyroid antibodies may overlap the IgG4-related thyroiditis. ..
  15. ncbi request reprint Clinical and molecular features of a TSH-secreting pituitary microadenoma
    Takeshi Usui
    Department of Endocrinology Clinical Research Institute, National Hospital Organization Kyoto Medical Center, Japan
    Pituitary 8:127-34. 2005
    ..The gene expression pattern in the tumor might have a role in the clinical features of this case...
  16. ncbi request reprint Antiatherogenic effect of pioglitazone in type 2 diabetic patients irrespective of the responsiveness to its antidiabetic effect
    Noriko Satoh
    Diabetes Center and Clinical Research Institute of Kyoto National Hospital, Kyoto, Japan
    Diabetes Care 26:2493-9. 2003
    ..This study was designed to elucidate the relationship between the antiatherogenic and antidiabetic effects of pioglitazone, a TZD, in type 2 diabetic patients...
  17. doi request reprint Novel tandem germline RET proto-oncogene mutations in a patient with multiple endocrine neoplasia type 2B: report of a case and a literature review of tandem RET mutations with in silico analysis
    Kanako Tanase Nakao
    Department of Endocrinology and Metabolism, National Hospital Organization, Kyoto Medical Center, Kyoto, Japan
    Head Neck 35:E363-8. 2013
    ..Multiple endocrine neoplasia type 2B (MEN2B) is the rarest and most aggressive form of MEN2. MEN2B cases usually carry either an M918T or A883T mutation of the RET, but to date, there are 3 atypical MEN2B caused by tandem mutations...
  18. ncbi request reprint Two rare TSH receptor amino acid substitutions in toxic thyroid adenomas
    Kazutaka Nanba
    Department of Endocrinology and Metabolism, National Hospital Organization Kyoto Medical Center, Kyoto, Japan
    Endocr J 59:13-9. 2012
    ..Case 1 represents the second reported case of an I568F mutation and Case 2 represents the third reported case of an S281I mutation...
  19. ncbi request reprint Molecular mechanism of the inhibitory effect of aldosterone on endothelial NO synthase activity
    Daisuke Nagata
    Division of Endocrinology, National Hospital Organization Kyoto Medical Center, Research Institute, Kyoto, Japan
    Hypertension 48:165-71. 2006
    ..These results suggest that Aldo inhibits eNOS function through bimodal mechanisms of 5,6,7,8-tetrahydrobiopterin deficiency and protein phosphatase 2A activation...
  20. doi request reprint A novel GATA3 nonsense mutation in a newly diagnosed adult patient of hypoparathyroidism, deafness, and renal dysplasia (HDR) syndrome
    Kazutaka Nanba
    Department of Endocrinology and Metabolism, National Hospital Organization Kyoto Medical Center, Kyoto, Japan
    Endocr Pract 19:e17-20. 2013
    ..Here we report a novel mutation of GATA3 in a patient diagnosed with HDR syndrome at the age of 58 with extensive intracranial calcification...
  21. doi request reprint Lack of ACTH and androgen receptor expression in a giant adrenal myelolipoma associated with 21-hydroxylase deficiency
    Hanae Hagiwara
    Department of Endocrinology and Metabolism, National Hospital Organization Kyoto Medical Center, Kyoto, Japan
    Endocr Pathol 19:122-7. 2008
    ..Our finding that the tumor did not over-express ACTH or androgen receptor suggests a limited direct role for these hormones in the development of the myelolipoma...
  22. ncbi request reprint Toxic thyroid adenoma presenting as hypokalemic periodic paralysis
    Tetsuya Tagami
    Clinical Research Institute, Division of Endocrinology and Metabolism, Kyoto Medical Center, National Hospital Organization, Japan
    Endocr J 54:797-803. 2007
    ..Although thyrotoxic periodic paralysis is usually accompanied with Graves' disease, thyrotoxicosis of other conditions including Plummer's disease should be considered...
  23. doi request reprint Successful treatment of Cushing's disease caused by ectopic intracavernous microadenoma
    Mitsuteru Koizumi
    Department of Endocrinology and Metabolism, National Hospital Organization Kyoto Medical Center, Mukaihata cho Fukakusa Fushimi ku, Kyoto, 612 8555, Japan
    Pituitary 14:295-8. 2011
    ....
  24. ncbi request reprint Suppressed levels of growth hormone and insulin-like growth factor-1 during successful pregnancy in persistent acromegaly
    Akira Shimatsu
    Clinical Research Institute, National Hospital Organization Kyoto Medical Center, Kyoto, Japan
    Endocr J 57:551-3. 2010
    ..The suppression of GH secretion and IGF-1 may be due to increased estrogen or other factors circulating in mid- to late pregnancy...
  25. doi request reprint R27X nonsense mutation of the SDHB gene in a patient with sporadic malignant paraganglioma
    Masaki Naito
    Department of Endocrinology and Metabolism, National Hospital Organization, Kyoto Medical Center, Kyoto, Japan
    Endocrine 36:10-5. 2009
    ..Genetic analysis indicated a nonsense mutation at codon 27 of the SDHB gene. As recently described, SDHB mutations may cause extra-adrenal and malignant paragangliomas, such as in the present case...
  26. ncbi request reprint [Etiology and clinical guidelines for the diagnosis and treatment of pheochromocytoma in Japan]
    Mitsuhide Naruse
    Department of Endocrinology, Metabolism, and Hypertension, NHO Kyoto Medical Center, Kyoto, Japan
    Nihon Geka Gakkai Zasshi 113:378-83. 2012
    ..Careful long-term follow-up is essential even in patients with benign pheochromocytoma...
  27. ncbi request reprint [Thyrotoxic periodic paralysis]
    Tetsuya Tagami
    Division of Endocrinology and Metabolism, National Hospital Organization, Kyoto Medical Center
    Nihon Rinsho . 2006
  28. ncbi request reprint [Thyrotoxic myopathy]
    Tetsuya Tagami
    Division of Endocrinology and Metabolism, National Hospital Organization, Kyoto Medical Center
    Nihon Rinsho . 2006
  29. ncbi request reprint Genomic structure and characterization of the 5'-flanking region of the human ghrelin gene
    Naotetsu Kanamoto
    Department of Medicine and Clinical Science, Kyoto University Graduate School of Medicine, Kyoto, Japan
    Endocrinology 145:4144-53. 2004
    ..These results suggest a potential role for upstream stimulatory factor transcription factors in the regulation of human ghrelin expression...
  30. ncbi request reprint [Case of pseudo-hypoparathyroidism type II with Basedow's disease]
    Motomu Hashimoto
    Department of Nephrology, National Hospital Organization Kyoto Medical Center
    Clin Calcium 14:122-6. 2004
  31. ncbi request reprint Thyroid hormone action is disrupted by bisphenol A as an antagonist
    Kenji Moriyama
    Department of Medicine and Clinical Science, Graduate School of Medicine, Kyoto University, Kyoto 606 8507, Japan
    J Clin Endocrinol Metab 87:5185-90. 2002
    ..This is the first report that BPA can antagonize T(3) action at the transcriptional level. BPA may disrupt the function of various types of nuclear hormone receptors and their cofactors to disturb our internal hormonal environment...