Y Shirayoshi

Summary

Affiliation: National Institute of Genetics
Country: Japan

Publications

  1. ncbi Proto-oncogene of int-3, a mouse Notch homologue, is expressed in endothelial cells during early embryogenesis
    Y Shirayoshi
    Mammalian Development Laboratory, National Institute of Genetics, Mishima, Japan
    Genes Cells 2:213-24. 1997
  2. ncbi Recombination in the class III region of the mouse major histocompatibility complex
    M Yoshino
    Mammalian Genetics Laboratory, National Institute of Genetics, Mishima, Japan
    Immunogenetics 40:280-6. 1994
  3. ncbi Functional conservation of mouse Notch receptor family members
    H Kato
    Department of Medical Chemistry, Kyoto University, Faculty of Medicine, Sakyo ku, Japan
    FEBS Lett 395:221-4. 1996
  4. ncbi A human gene responsible for Zellweger syndrome that affects peroxisome assembly
    N Shimozawa
    Department of Pediatrics, Gifu University School of Medicine, Japan
    Science 255:1132-4. 1992
  5. ncbi Cyclic AMP-mediated augmentation of thrombomodulin gene expression: cell type-dependent usage of control regions
    Y Shirayoshi
    Division of Cell Biology, Meiji Institute of Health Science, Meiji Milk Products Co Ltd, Odawara, Japan
    Exp Cell Res 208:75-83. 1993
  6. ncbi Cloning of inv, a gene that controls left/right asymmetry and kidney development
    T Mochizuki
    Department of Medicine, Kidney Center, Tokyo Women s Medical University, School of Medicine, Japan
    Nature 395:177-81. 1998
  7. ncbi The ActR-I activin receptor protein is expressed in notochord, lens placode and pituitary primordium cells in the mouse embryo
    S I Yoshikawa
    Department of Molecular Biology, Biomolecular Engineering Research Institute, 6 2 3 Furuedai, Suita, Osaka, Japan
    Mech Dev 91:439-44. 2000
  8. ncbi Correction of a genetic defect in multipotent germline stem cells using a human artificial chromosome
    Y Kazuki
    Department of Biomedical Science, Institute of Regenerative Medicine and Biofunction, Graduate School of Medical Science, Tottori University, Yonago, Tottori, Japan
    Gene Ther 15:617-24. 2008

Collaborators

Detail Information

Publications8

  1. ncbi Proto-oncogene of int-3, a mouse Notch homologue, is expressed in endothelial cells during early embryogenesis
    Y Shirayoshi
    Mammalian Development Laboratory, National Institute of Genetics, Mishima, Japan
    Genes Cells 2:213-24. 1997
    ..However, the exact nature and the in vivo expression pattern of the int-3 proto-oncogene are unknown. The function of gene products in embryogenesis is also not clear...
  2. ncbi Recombination in the class III region of the mouse major histocompatibility complex
    M Yoshino
    Mammalian Genetics Laboratory, National Institute of Genetics, Mishima, Japan
    Immunogenetics 40:280-6. 1994
    ..The result demonstrated that an unequal distribution of recombination is a general feature of the mouse MHC, suggesting the presence of a recombinational hotspot within the Int3:Tnx interval...
  3. ncbi Functional conservation of mouse Notch receptor family members
    H Kato
    Department of Medical Chemistry, Kyoto University, Faculty of Medicine, Sakyo ku, Japan
    FEBS Lett 395:221-4. 1996
    ..These results indicate that all mouse Notch family members have biochemical functions similar to mNotch1, which transduces proliferative signal by direct interaction with the DNA binding protein RBP-Jkappa...
  4. ncbi A human gene responsible for Zellweger syndrome that affects peroxisome assembly
    N Shimozawa
    Department of Pediatrics, Gifu University School of Medicine, Japan
    Science 255:1132-4. 1992
    ..The homozygous patient apparently inherited the mutation from her parents, each of whom was heterozygous for that mutation...
  5. ncbi Cyclic AMP-mediated augmentation of thrombomodulin gene expression: cell type-dependent usage of control regions
    Y Shirayoshi
    Division of Cell Biology, Meiji Institute of Health Science, Meiji Milk Products Co Ltd, Odawara, Japan
    Exp Cell Res 208:75-83. 1993
    ..We propose that there are at least two separate genomic DNA regions that regulate cyclic AMP-dependent TM gene expression and that their functions are cell type dependent...
  6. ncbi Cloning of inv, a gene that controls left/right asymmetry and kidney development
    T Mochizuki
    Department of Medicine, Kidney Center, Tokyo Women s Medical University, School of Medicine, Japan
    Nature 395:177-81. 1998
    ..Transgenic introduction of a minigene encoding the candidate protein restores normal left/right asymmetry and kidney development in the inv mutant, confirming the identity of the candidate gene...
  7. ncbi The ActR-I activin receptor protein is expressed in notochord, lens placode and pituitary primordium cells in the mouse embryo
    S I Yoshikawa
    Department of Molecular Biology, Biomolecular Engineering Research Institute, 6 2 3 Furuedai, Suita, Osaka, Japan
    Mech Dev 91:439-44. 2000
    ..Interestingly, in the lens placodes and in early Rathke's pouch, ActR-I protein is transiently localized at the apical surface of the epithelial cells, indicating the presence of an apical-basal asymmetry in these cells...
  8. ncbi Correction of a genetic defect in multipotent germline stem cells using a human artificial chromosome
    Y Kazuki
    Department of Biomedical Science, Institute of Regenerative Medicine and Biofunction, Graduate School of Medical Science, Tottori University, Yonago, Tottori, Japan
    Gene Ther 15:617-24. 2008
    ..Therefore, the combination of using mGS cells with HACs provides a new tool for gene and cell therapies. The next step is to demonstrate functional restoration using animal models for future gene therapy...