Y Kanda

Summary

Affiliation: National Defense Medical College
Country: Japan

Publications

  1. pmc Thrombin-induced p38 mitogen-activated protein kinase activation is mediated by epidermal growth factor receptor transactivation pathway
    Y Kanda
    Department of Pharmacology, National Defense Medical College, 3 2, Namiki, Tokorozawa, Saitama, 359 8513, Japan
    Br J Pharmacol 132:1657-64. 2001
  2. ncbi request reprint Thrombin activates p38 mitogen-activated protein kinase in vascular smooth muscle cells
    Y Kanda
    Department of Pharmacology, National Defense Medical College, Tokorozawa, Saitama, Japan
    Life Sci 68:1989-2000. 2001
  3. pmc Adrenaline increases glucose transport via a Rap1-p38MAPK pathway in rat vascular smooth muscle cells
    Y Kanda
    Department of Pharmacology, National Defense Medical College, Tokorozawa, Saitama, Japan
    Br J Pharmacol 151:476-82. 2007
  4. ncbi request reprint Arachidonic acid metabolites in acute myocardial infarction
    B Takase
    1st Department of Internal Medicine, National Defense Medical College, Saitama, Japan
    Angiology 47:649-61. 1996
  5. pmc Binding of Delta1, Jagged1, and Jagged2 to Notch2 rapidly induces cleavage, nuclear translocation, and hyperphosphorylation of Notch2
    K Shimizu
    Departments of Hematology and Oncology, Graduate School of Medicine, University of Tokyo, Japan
    Mol Cell Biol 20:6913-22. 2000

Collaborators

  • A Kurita
  • H Nakamura
  • K Shimizu
  • B Takase
  • S Chiba
  • H Hirai
  • T Saito
  • M Kurokawa
  • N Hosoya
  • K Kumano
  • Y Hamada
  • A Uehata
  • K Katsura
  • T Nishioka
  • K Mizuno
  • T Maruyama

Detail Information

Publications5

  1. pmc Thrombin-induced p38 mitogen-activated protein kinase activation is mediated by epidermal growth factor receptor transactivation pathway
    Y Kanda
    Department of Pharmacology, National Defense Medical College, 3 2, Namiki, Tokorozawa, Saitama, 359 8513, Japan
    Br J Pharmacol 132:1657-64. 2001
    ..In contrast, PDGF-induced DNA synthesis was not affected by AG1478. In conclusion, these data suggest that the EGF receptor transactivation and subsequent p38 MAPK activation is required for thrombin-induced proliferation of VSMC...
  2. ncbi request reprint Thrombin activates p38 mitogen-activated protein kinase in vascular smooth muscle cells
    Y Kanda
    Department of Pharmacology, National Defense Medical College, Tokorozawa, Saitama, Japan
    Life Sci 68:1989-2000. 2001
    ..These results suggest that thrombin activates p38 MAPK in a manner dependent on Gbetagamma, protein kinase C, a tyrosine kinase, and Ras, that p38 MAPK has a role in thrombin-induced mitogenic response in the cells...
  3. pmc Adrenaline increases glucose transport via a Rap1-p38MAPK pathway in rat vascular smooth muscle cells
    Y Kanda
    Department of Pharmacology, National Defense Medical College, Tokorozawa, Saitama, Japan
    Br J Pharmacol 151:476-82. 2007
    ..Adrenaline has been implicated in the pathogenesis of atherosclerosis. However, little is known regarding the role of adrenaline in glucose transport in VSMC...
  4. ncbi request reprint Arachidonic acid metabolites in acute myocardial infarction
    B Takase
    1st Department of Internal Medicine, National Defense Medical College, Saitama, Japan
    Angiology 47:649-61. 1996
    ..These values decreased to near-normal control levels by one month after the AMI attack. The findings in this study suggest that abnormalities of arachidonic acid metabolism accompany, and may play a role in the pathogenesis of, AMI...
  5. pmc Binding of Delta1, Jagged1, and Jagged2 to Notch2 rapidly induces cleavage, nuclear translocation, and hyperphosphorylation of Notch2
    K Shimizu
    Departments of Hematology and Oncology, Graduate School of Medicine, University of Tokyo, Japan
    Mol Cell Biol 20:6913-22. 2000
    ..Moreover, the findings of rapid cleavage, nuclear translocation, and phosphorylation of Notch2 after ligand binding facilitate the understanding of the Notch signaling...