Tatsuya Mishima

Summary

Affiliation: Kyorin University
Country: Japan

Publications

  1. ncbi Reduction of neurotransmitter release by the exogenous H3 domain peptide of HPC-1/syntaxin 1A in cultured rat hippocampal neurons
    Tatsuya Mishima
    Department of Physiology, Kyorin University School of Medicine, Shinkawa 6 20 2, Mitaka, Tokyo 181 8611, Japan
    Neurosci Lett 329:273-6. 2002
  2. ncbi Impairment of catecholamine systems during induction of long-term potentiation at hippocampal CA1 synapses in HPC-1/syntaxin 1A knock-out mice
    Tatsuya Mishima
    Department of Cell Physiology, Kyorin University School of Medicine, Mitaka, Tokyo 181 8611, Japan
    J Neurosci 32:381-9. 2012
  3. ncbi Analysis of knock-out mice to determine the role of HPC-1/syntaxin 1A in expressing synaptic plasticity
    Tomonori Fujiwara
    Department of Cell Physiology, Kyorin University School of Medicine, Mitaka, Tokyo 181 8611, Japan
    J Neurosci 26:5767-76. 2006
  4. ncbi Mitochondrial membrane potential and intracellular ATP content after transient experimental ischemia in the cultured hippocampal neuron
    Takehiko Iijima
    Department of Anesthesiology, School of Medicine, Kyorin University, 6 20 2 Shinkawa, Mitaka City, Tokyo 181, Japan
    Neurochem Int 43:263-9. 2003
  5. ncbi Neuroprotective effect of propofol on necrosis and apoptosis following oxygen-glucose deprivation--relationship between mitochondrial membrane potential and mode of death
    Takehiko Iijima
    Department of Anesthesiology, Kyorin University, School of Medicine, Tokyo, Japan
    Brain Res 1099:25-32. 2006
  6. ncbi Calcium loading capacity and morphological changes in mitochondria in an ischemic preconditioned model
    Takehiko Iijima
    Department of Anesthesiology, Kyorin University, School of Medicine, Japan
    Neurosci Lett 448:268-72. 2008
  7. ncbi Mitochondrial hyperpolarization after transient oxygen-glucose deprivation and subsequent apoptosis in cultured rat hippocampal neurons
    Takehiko Iijima
    Department of Anesthesiology, Kyorin University School of Medicine, 6 20 2 Shinkawa, Mitaka City 181 8611, Tokyo, Japan
    Brain Res 993:140-5. 2003

Detail Information

Publications7

  1. ncbi Reduction of neurotransmitter release by the exogenous H3 domain peptide of HPC-1/syntaxin 1A in cultured rat hippocampal neurons
    Tatsuya Mishima
    Department of Physiology, Kyorin University School of Medicine, Shinkawa 6 20 2, Mitaka, Tokyo 181 8611, Japan
    Neurosci Lett 329:273-6. 2002
    ..These results suggest that the H3 domain peptide reduces neurotransmitter release by retarding the refilling of readily releasable vesicles...
  2. ncbi Impairment of catecholamine systems during induction of long-term potentiation at hippocampal CA1 synapses in HPC-1/syntaxin 1A knock-out mice
    Tatsuya Mishima
    Department of Cell Physiology, Kyorin University School of Medicine, Mitaka, Tokyo 181 8611, Japan
    J Neurosci 32:381-9. 2012
    ..Theses results suggest that HPC-1/syntaxin 1A regulates catecholaminergic systems via exocytosis of dense-core synaptic vesicles, and that deletion of HPC-1/syntaxin 1A causes impairment of LTP induction...
  3. ncbi Analysis of knock-out mice to determine the role of HPC-1/syntaxin 1A in expressing synaptic plasticity
    Tomonori Fujiwara
    Department of Cell Physiology, Kyorin University School of Medicine, Mitaka, Tokyo 181 8611, Japan
    J Neurosci 26:5767-76. 2006
    ..Interestingly, the KO mice had impaired conditioned fear memory extinction. These observations suggest that HPC-1/syntaxin 1A may be closely related to synaptic plasticity...
  4. ncbi Mitochondrial membrane potential and intracellular ATP content after transient experimental ischemia in the cultured hippocampal neuron
    Takehiko Iijima
    Department of Anesthesiology, School of Medicine, Kyorin University, 6 20 2 Shinkawa, Mitaka City, Tokyo 181, Japan
    Neurochem Int 43:263-9. 2003
    ..This pathological finding of an energy-producing system after OGD may provide a clue to explain post-ischemic energy failure...
  5. ncbi Neuroprotective effect of propofol on necrosis and apoptosis following oxygen-glucose deprivation--relationship between mitochondrial membrane potential and mode of death
    Takehiko Iijima
    Department of Anesthesiology, Kyorin University, School of Medicine, Tokyo, Japan
    Brain Res 1099:25-32. 2006
    ..Propofol induces a moratorium on neuronal death, during which pharmacological intervention might be able to prevent cell death...
  6. ncbi Calcium loading capacity and morphological changes in mitochondria in an ischemic preconditioned model
    Takehiko Iijima
    Department of Anesthesiology, Kyorin University, School of Medicine, Japan
    Neurosci Lett 448:268-72. 2008
    ..Our observations suggest that subpopulations of mitochondria with specific morphologies are linked to the CLC and mPT...
  7. ncbi Mitochondrial hyperpolarization after transient oxygen-glucose deprivation and subsequent apoptosis in cultured rat hippocampal neurons
    Takehiko Iijima
    Department of Anesthesiology, Kyorin University School of Medicine, 6 20 2 Shinkawa, Mitaka City 181 8611, Tokyo, Japan
    Brain Res 993:140-5. 2003
    ..Neurons were still viable even during hyperpolarization of mitochondria, but this hyperpolarization appears to be linked to subsequent apoptotic change...