Makoto Tsuneoka

Summary

Affiliation: Kurume University School of Medicine
Country: Japan

Publications

  1. ncbi request reprint Expression of Mina53, a product of a Myc target gene in mouse testis
    Makoto Tsuneoka
    Division of Human Genetics, Department of Forensic Medicine, Kurume University School of Medicine, Kurume, Japan
    Int J Androl 29:323-30. 2006
  2. ncbi request reprint A novel Myc-target gene, mimitin, that is involved in cell proliferation of esophageal squamous cell carcinoma
    Makoto Tsuneoka
    Division of Human Genetics, Department of Forensic Medicine, Kurume University School of Medicine, Japan
    J Biol Chem 280:19977-85. 2005
  3. ncbi request reprint Mina53 as a potential prognostic factor for esophageal squamous cell carcinoma
    Makoto Tsuneoka
    Division of Human Genetics, Department of Forensic Medicine, Kurume University School of Medicine, Kurume, Japan
    Clin Cancer Res 10:7347-56. 2004
  4. ncbi request reprint c-myc induces autophagy in rat 3Y1 fibroblast cells
    Makoto Tsuneoka
    Division of Human Genetics, Department of Forensic Medicine, Kurume University School of Medicine, Kurume, Japan
    Cell Struct Funct 28:195-204. 2003
  5. ncbi request reprint A novel myc target gene, mina53, that is involved in cell proliferation
    Makoto Tsuneoka
    Division of Human Genetics, Department of Forensic Medicine, Kurume University School of Medicine, Kurume 830 0011, Japan
    J Biol Chem 277:35450-9. 2002
  6. pmc Increased expression of a Myc target gene Mina53 in human colon cancer
    Kwesi Teye
    Department of Forensic Medicine, Kurume University School of Medicine, Kurume, Japan
    Am J Pathol 164:205-16. 2004
  7. ncbi request reprint A novel I247T missense mutation in the haptoglobin 2 beta-chain decreases the expression of the protein and is associated with ahaptoglobinemia
    Kwesi Teye
    Division of Human Genetics, Department of Forensic Medicine, Kurume University School of Medicine, 830 0011 Kurume, Japan
    Hum Genet 114:499-502. 2004
  8. ncbi request reprint Immunohistochemical expressions of Cap43 and Mina53 proteins in neuroblastoma
    Suguru Fukahori
    Department of Pathology, Kurume University School of Medicine, Kurume, Fukuoka 830 0011, Japan
    J Pediatr Surg 42:1831-40. 2007
  9. ncbi request reprint Haptoglobin gene promoter polymorphism and haplotypes are unique in different populations
    Kwesi Teye
    Division of Human Genetics, Department of Forensic Medicine, Kurume University School of Medicine, Japan
    Hum Biol 78:121-6. 2006
  10. ncbi request reprint Expression of Myc target gene mina53 in subtypes of human lymphoma
    Kwesi Teye
    Research Center for Innovative Cancer Therapy, Kurume University School of Medicine, Kurume 830 0011, Japan
    Oncol Rep 18:841-8. 2007

Collaborators

Detail Information

Publications16

  1. ncbi request reprint Expression of Mina53, a product of a Myc target gene in mouse testis
    Makoto Tsuneoka
    Division of Human Genetics, Department of Forensic Medicine, Kurume University School of Medicine, Kurume, Japan
    Int J Androl 29:323-30. 2006
    ....
  2. ncbi request reprint A novel Myc-target gene, mimitin, that is involved in cell proliferation of esophageal squamous cell carcinoma
    Makoto Tsuneoka
    Division of Human Genetics, Department of Forensic Medicine, Kurume University School of Medicine, Japan
    J Biol Chem 280:19977-85. 2005
    ..Taken together, these results suggest that the novel gene mimitin is a direct transcriptional target of c-Myc, and is involved in Myc-dependent cell proliferation at least in ESCC cells...
  3. ncbi request reprint Mina53 as a potential prognostic factor for esophageal squamous cell carcinoma
    Makoto Tsuneoka
    Division of Human Genetics, Department of Forensic Medicine, Kurume University School of Medicine, Kurume, Japan
    Clin Cancer Res 10:7347-56. 2004
    ..We previously identified mina53, a novel Myc target gene. Here we investigated whether mina53 is related to esophageal squamous cell carcinoma (ESCC), a disease with poor prognosis...
  4. ncbi request reprint c-myc induces autophagy in rat 3Y1 fibroblast cells
    Makoto Tsuneoka
    Division of Human Genetics, Department of Forensic Medicine, Kurume University School of Medicine, Kurume, Japan
    Cell Struct Funct 28:195-204. 2003
    ..These results suggest that the autophagy caused by c-myc is not due to the apoptosis or tumorigenesis induced by c-myc. Taken together, our results suggest that the induction of autophagy is a novel function of c-myc...
  5. ncbi request reprint A novel myc target gene, mina53, that is involved in cell proliferation
    Makoto Tsuneoka
    Division of Human Genetics, Department of Forensic Medicine, Kurume University School of Medicine, Kurume 830 0011, Japan
    J Biol Chem 277:35450-9. 2002
    ..Taken together, these results indicate that mina53 is a direct target gene of Myc, suggesting that mina53 is involved in mammalian cell proliferation...
  6. pmc Increased expression of a Myc target gene Mina53 in human colon cancer
    Kwesi Teye
    Department of Forensic Medicine, Kurume University School of Medicine, Kurume, Japan
    Am J Pathol 164:205-16. 2004
    ..Together, our results indicate that the elevated expression of Mina53 is a characteristic feature in colon cancer, one that may have therapeutic applications...
  7. ncbi request reprint A novel I247T missense mutation in the haptoglobin 2 beta-chain decreases the expression of the protein and is associated with ahaptoglobinemia
    Kwesi Teye
    Division of Human Genetics, Department of Forensic Medicine, Kurume University School of Medicine, 830 0011 Kurume, Japan
    Hum Genet 114:499-502. 2004
    ..Since the -61C mutation also leads to low Hp expression, the genotype represents the first and most definitive ahaptoglobinemic case reported in Africa...
  8. ncbi request reprint Immunohistochemical expressions of Cap43 and Mina53 proteins in neuroblastoma
    Suguru Fukahori
    Department of Pathology, Kurume University School of Medicine, Kurume, Fukuoka 830 0011, Japan
    J Pediatr Surg 42:1831-40. 2007
    ..We studied the expressions of both Mina53, which is a myc target gene and is related to cell proliferation, and Cap43, which is related to metastasis suppression and downregulation of MYCN gene, in neuroblastoma...
  9. ncbi request reprint Haptoglobin gene promoter polymorphism and haplotypes are unique in different populations
    Kwesi Teye
    Division of Human Genetics, Department of Forensic Medicine, Kurume University School of Medicine, Japan
    Hum Biol 78:121-6. 2006
    ..Six base substitutions at the promoter region were population specific. Only 3 out of 18 haplotypes were shared among the populations. A probable application of HP in human population genetics appears legitimate...
  10. ncbi request reprint Expression of Myc target gene mina53 in subtypes of human lymphoma
    Kwesi Teye
    Research Center for Innovative Cancer Therapy, Kurume University School of Medicine, Kurume 830 0011, Japan
    Oncol Rep 18:841-8. 2007
    ..These results suggest that although Mina53 expression is not prominent in lymphoma in general, it may be related to tumor progression of B cell lymphoma...
  11. ncbi request reprint Overexpression of the myc target gene Mina53 in advanced renal cell carcinoma
    Hironori Ishizaki
    Department of Pathology, Kurume University School of Medicine, Kurume, Japan
    Pathol Int 57:672-80. 2007
    ..0001). In conclusion, Mina53 is overexpressed in RCC tissue from patients with poor prognostic factors, suggesting that Mina53 overexpression is one of the factors for poor prognosis in RCC...
  12. ncbi request reprint Immunohistochemical expression of Mina53 and Ki67 proteins in human primary gingival squamous cell carcinoma
    Keitaro Kuratomi
    Department of Pathology, Kurume University School of Medicine, Kurume, Japan
    Kurume Med J 53:71-8. 2006
    ....
  13. ncbi request reprint Heterozygosity for two novel null alleles of the KEL gene causes the Kell-null phenotype in a Japanese woman
    Yoshiro Koda
    Department of Forensic Medicine and Human Genetics, Kurume University School of Medicine, Yasumoto Hospital, Mizuma, Japan
    Br J Haematol 117:220-5. 2002
    ..Neither mutation was found in 114 randomly selected Japanese individuals. The results suggested that the Ko blood group phenotype might be owing to several distinct non-functional alleles without any prevalent allele...
  14. ncbi request reprint Accelerated expression of a Myc target gene Mina53 in aggressive hepatocellular carcinoma
    Sachiko Ogasawara
    Department of Pathology, Kurume University School of Medicine, Asahi machi, Kurume, Fukuoka
    Hepatol Res 40:330-6. 2010
    ..Conclusions: Our findings suggested that Mina53 expression is accelerated in HCC with a lower histological grade, with cell proliferation capability, or with a larger diameter, and Mina53 is related to biological malignancy of HCC...
  15. ncbi request reprint Investigation of the complex I assembly chaperones B17.2L and NDUFAF1 in a cohort of CI deficient patients
    Rutger O Vogel
    Nijmegen Centre for Mitochondrial Disorders, Department of Paediatrics, Radboud University Nijmegen Medical Centre, 6500 HB, Nijmegen, The Netherlands
    Mol Genet Metab 91:176-82. 2007
    ..These findings illustrate the difference in mechanism between B17.2L and NDUFAF1 and suggest that the involvement of NDUFAF1 in the assembly process could be indirect rather than direct via the binding to assembly intermediates...
  16. ncbi request reprint Glucose-dependent insulinotropic polypeptide (GIP) stimulation of pancreatic beta-cell survival is dependent upon phosphatidylinositol 3-kinase (PI3K)/protein kinase B (PKB) signaling, inactivation of the forkhead transcription factor Foxo1, and down-regu
    Su Jin Kim
    Department of Cellular and Physiological Sciences, University of British Columbia, 2146 Health Sciences Mall, Vancouver, British Columbia V6T 1Z3, Canada
    J Biol Chem 280:22297-307. 2005
    ..The results show that PI3K/PKB/Foxo1 signaling mediates GIP suppression of bax gene expression and that this module is a key pathway by which GIP regulates beta-cell apoptosis in vivo...