Peixin Dong

Summary

Affiliation: Graduate School
Country: Japan

Publications

  1. pmc MicroRNA-194 inhibits epithelial to mesenchymal transition of endometrial cancer cells by targeting oncogene BMI-1
    Peixin Dong
    Department of Gynecology, Hokkaido University Graduate School of Medicine and School of Medicine, Hokkaido University, Sapporo, Japan
    Mol Cancer 10:99. 2011
  2. pmc Mutant p53 gain-of-function induces epithelial-mesenchymal transition through modulation of the miR-130b-ZEB1 axis
    P Dong
    Department of Gynecology, Hokkaido University School of Medicine, Hokkaido University, Sapporo, Japan
    Oncogene 32:3286-95. 2013
  3. pmc Emerging therapeutic biomarkers in endometrial cancer
    Peixin Dong
    Department of Women s Health Educational System, Hokkaido University School of Medicine, Hokkaido University, N15, W7, Sapporo 060 8638, Japan
    Biomed Res Int 2013:130362. 2013
  4. pmc Elevated expression of p53 gain-of-function mutation R175H in endometrial cancer cells can increase the invasive phenotypes by activation of the EGFR/PI3K/AKT pathway
    Peixin Dong
    Hospital and Institute of Obstetrics and Gynecology, Fudan University Shanghai Medical College, Shanghai, PR China
    Mol Cancer 8:103. 2009
  5. ncbi request reprint MicroRNA-106b modulates epithelial-mesenchymal transition by targeting TWIST1 in invasive endometrial cancer cell lines
    Peixin Dong
    Department of Women s Health Educational System, Hokkaido University School of Medicine, Hokkaido University, Sapporo, Japan
    Mol Carcinog 53:349-59. 2014
  6. ncbi request reprint Identification of KLF17 as a novel epithelial to mesenchymal transition inducer via direct activation of TWIST1 in endometrioid endometrial cancer
    Peixin Dong
    Department of Women s Health Educational System, Hokkaido University School of Medicine, Hokkaido University, Sapporo 0608638, Japan
    Carcinogenesis 35:760-8. 2014
  7. ncbi request reprint Distribution of lymph node metastasis sites in endometrial cancer undergoing systematic pelvic and para-aortic lymphadenectomy: a proposal of optimal lymphadenectomy for future clinical trials
    Tetsuji Odagiri
    Department of Obstetrics and Gynecology, Hokkaido University Graduate School of Medicine, Sapporo, Japan
    Ann Surg Oncol 21:2755-61. 2014
  8. ncbi request reprint p53 dominant-negative mutant R273H promotes invasion and migration of human endometrial cancer HHUA cells
    Peixin Dong
    Department of Gynecology, Hokkaido University Graduate School of Medicine and School of Medicine, Hokkaido University, Sapporo 060 8638, Japan
    Clin Exp Metastasis 24:471-83. 2007
  9. doi request reprint New revised FIGO 2008 staging system for endometrial cancer produces better discrimination in survival compared with the 1988 staging system
    Tatsuya Kato
    Department of Obstetrics and Gynecology, Hokkaido University Graduate School of Medicine, Sapporo, Japan
    J Surg Oncol 106:938-41. 2012
  10. doi request reprint Allopurinol attenuates oxidative stress and cardiac fibrosis in angiotensin II-induced cardiac diastolic dysfunction
    Nan Jia
    Ruijin Hospital, Shanghai Institute of Hypertension, Shanghai Jiao Tong University Medical School, Shanghai, PR China
    Cardiovasc Ther 30:117-23. 2012

Collaborators

Detail Information

Publications10

  1. pmc MicroRNA-194 inhibits epithelial to mesenchymal transition of endometrial cancer cells by targeting oncogene BMI-1
    Peixin Dong
    Department of Gynecology, Hokkaido University Graduate School of Medicine and School of Medicine, Hokkaido University, Sapporo, Japan
    Mol Cancer 10:99. 2011
    ..The aims of this study was to determine the roles of BMI-1 in inducing EMT of endometrial cancer (EC) cells and the possible role of miRNA in controlling BMI-1 expression...
  2. pmc Mutant p53 gain-of-function induces epithelial-mesenchymal transition through modulation of the miR-130b-ZEB1 axis
    P Dong
    Department of Gynecology, Hokkaido University School of Medicine, Hokkaido University, Sapporo, Japan
    Oncogene 32:3286-95. 2013
    ..These data provide a novel understanding of the roles of p53 gain-of-function mutations in accelerating tumor progression and metastasis through modulation of the miR-130b-ZEB1 axis. ..
  3. pmc Emerging therapeutic biomarkers in endometrial cancer
    Peixin Dong
    Department of Women s Health Educational System, Hokkaido University School of Medicine, Hokkaido University, N15, W7, Sapporo 060 8638, Japan
    Biomed Res Int 2013:130362. 2013
    ....
  4. pmc Elevated expression of p53 gain-of-function mutation R175H in endometrial cancer cells can increase the invasive phenotypes by activation of the EGFR/PI3K/AKT pathway
    Peixin Dong
    Hospital and Institute of Obstetrics and Gynecology, Fudan University Shanghai Medical College, Shanghai, PR China
    Mol Cancer 8:103. 2009
    ..In human endometrial cancer, p53 mutation is more often associated with aggressive nonendometrioid cancer. However, it was unknown if p53 mutants contributed to endometrial cancer progression through the GOF properties...
  5. ncbi request reprint MicroRNA-106b modulates epithelial-mesenchymal transition by targeting TWIST1 in invasive endometrial cancer cell lines
    Peixin Dong
    Department of Women s Health Educational System, Hokkaido University School of Medicine, Hokkaido University, Sapporo, Japan
    Mol Carcinog 53:349-59. 2014
    ..Our data suggest that TWIST1 is a critical inducer of EMT in invasive EC cells and that miR-106b could suppress EC cell invasion by downregulating TWIST1 expression...
  6. ncbi request reprint Identification of KLF17 as a novel epithelial to mesenchymal transition inducer via direct activation of TWIST1 in endometrioid endometrial cancer
    Peixin Dong
    Department of Women s Health Educational System, Hokkaido University School of Medicine, Hokkaido University, Sapporo 0608638, Japan
    Carcinogenesis 35:760-8. 2014
    ..Thus, KLF17 may have an oncogenic role during EEC progression via initiating EMT through the regulation of TWIST1. ..
  7. ncbi request reprint Distribution of lymph node metastasis sites in endometrial cancer undergoing systematic pelvic and para-aortic lymphadenectomy: a proposal of optimal lymphadenectomy for future clinical trials
    Tetsuji Odagiri
    Department of Obstetrics and Gynecology, Hokkaido University Graduate School of Medicine, Sapporo, Japan
    Ann Surg Oncol 21:2755-61. 2014
    ..The aim of this study was to demonstrate the precise mapping of lymph node metastasis (LNM) sites in endometrial cancer...
  8. ncbi request reprint p53 dominant-negative mutant R273H promotes invasion and migration of human endometrial cancer HHUA cells
    Peixin Dong
    Department of Gynecology, Hokkaido University Graduate School of Medicine and School of Medicine, Hokkaido University, Sapporo 060 8638, Japan
    Clin Exp Metastasis 24:471-83. 2007
    ..Taken together, these results suggest that transdominance of R273H mutant over wt p53 rather than a gain-of-function promotes tumor metastasis by increasing invasion and migration in HHUA cells...
  9. doi request reprint New revised FIGO 2008 staging system for endometrial cancer produces better discrimination in survival compared with the 1988 staging system
    Tatsuya Kato
    Department of Obstetrics and Gynecology, Hokkaido University Graduate School of Medicine, Sapporo, Japan
    J Surg Oncol 106:938-41. 2012
    ..The aim of this study was to analyze the stage migration and survival of endometrial cancer by the revised FIGO 2008 staging system compared with the 1988 staging system...
  10. doi request reprint Allopurinol attenuates oxidative stress and cardiac fibrosis in angiotensin II-induced cardiac diastolic dysfunction
    Nan Jia
    Ruijin Hospital, Shanghai Institute of Hypertension, Shanghai Jiao Tong University Medical School, Shanghai, PR China
    Cardiovasc Ther 30:117-23. 2012
    ..We tested whether allopurinol could decrease myocardial oxidative stress and attenuate cardiac fibrosis and left ventricular diastolic dysfunction in angiotensin II (AngII)-induced hypertensive mice...