Shigetaka Yoshida

Summary

Affiliation: Asahikawa Medical College
Country: Japan

Publications

  1. ncbi request reprint Implications of protease M/neurosin in myelination during experimental demyelination and remyelination
    Yoshio Bando
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical College, Asahikawa, Hokkaido 078 8510, Japan
    Neurosci Lett 405:175-80. 2006
  2. doi request reprint Differential changes in axonal conduction following CNS demyelination in two mouse models
    Yoshio Bando
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical College, Asahikawa, Hokkaido 078 8510, Japan
    Eur J Neurosci 28:1731-42. 2008
  3. doi request reprint Klk8, a multifunctional protease in the brain and skin: analysis of knockout mice
    Shigetaka Yoshida
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical College, Hokkaido, Japan
    Biol Chem 391:375-80. 2010
  4. ncbi request reprint Involvement of neuropsin in the pathogenesis of experimental autoimmune encephalomyelitis
    Ryuji Terayama
    Department of Anatomy, Asahikawa Medical College, Asahikawa, Japan
    Glia 52:108-18. 2005
  5. ncbi request reprint Kallikrein 8 is involved in skin desquamation in cooperation with other kallikreins
    Mari Kishibe
    Departments of Structural Anatomy and Neuroscience and Dermatology, Asahikawa Medical College, Midorigaoka Higashi 2 1 1 1, Asahikawa 078 8510, Japan
    J Biol Chem 282:5834-41. 2007
  6. doi request reprint Minocycline reduces remyelination by suppressing ciliary neurotrophic factor expression after cuprizone-induced demyelination
    Tatsuhide Tanaka
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical University, Asahikawa, Hokkaido, Japan
    J Neurochem 127:259-70. 2013
  7. ncbi request reprint Regeneration of the abdominal postganglionic sympathetic system
    Masahiro Yamada
    Department of Anatomy 1, Asahikawa Medical College, Japan
    Neurosci Res 54:261-8. 2006
  8. ncbi request reprint Differential expression of protease M/neurosin in oligodendrocytes and their progenitors in an animal model of multiple sclerosis
    Ryuji Terayama
    Department of Anatomy, Asahikawa Medical College, 2 1 1 1 Midorigaoka Higashi, Asahikawa 078 8510, Japan
    Neurosci Lett 382:82-7. 2005
  9. doi request reprint Three-dimensional ultra-structures of myelin and the axons in the spinal cord: application of SEM with the osmium maceration method to the central nervous system in two mouse models
    Taichi Nomura
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical University, Asahikawa 078 8510, Japan
    Neurosci Res 75:190-7. 2013
  10. ncbi request reprint Differential expression of neuropsin and protease M/neurosin in oligodendrocytes after injury to the spinal cord
    Ryuji Terayama
    Department of Anatomy, Asahikawa Medical College, Asahikawa 078 8510, Japan
    Glia 48:91-101. 2004

Collaborators

Detail Information

Publications12

  1. ncbi request reprint Implications of protease M/neurosin in myelination during experimental demyelination and remyelination
    Yoshio Bando
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical College, Asahikawa, Hokkaido 078 8510, Japan
    Neurosci Lett 405:175-80. 2006
    ..Our data emphasize that the maintenance of myelination is an important function of protease M/neurosin in OLGs, suggesting its relation to the oligodendroglial response to myelin disorders...
  2. doi request reprint Differential changes in axonal conduction following CNS demyelination in two mouse models
    Yoshio Bando
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical College, Asahikawa, Hokkaido 078 8510, Japan
    Eur J Neurosci 28:1731-42. 2008
    ....
  3. doi request reprint Klk8, a multifunctional protease in the brain and skin: analysis of knockout mice
    Shigetaka Yoshida
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical College, Hokkaido, Japan
    Biol Chem 391:375-80. 2010
    ..Klk8 might be involved in desquamation through the degradation of adhesive molecules that connect layers of the epidermis. Klk8 might thus be involved in tissue development and rearrangement...
  4. ncbi request reprint Involvement of neuropsin in the pathogenesis of experimental autoimmune encephalomyelitis
    Ryuji Terayama
    Department of Anatomy, Asahikawa Medical College, Asahikawa, Japan
    Glia 52:108-18. 2005
    ..These observations suggest that neuropsin is involved in the pathogenesis of EAE mediated by demyelination and oligodendroglial death...
  5. ncbi request reprint Kallikrein 8 is involved in skin desquamation in cooperation with other kallikreins
    Mari Kishibe
    Departments of Structural Anatomy and Neuroscience and Dermatology, Asahikawa Medical College, Midorigaoka Higashi 2 1 1 1, Asahikawa 078 8510, Japan
    J Biol Chem 282:5834-41. 2007
    ..We propose that in psoriatic lesion, KLK8 modulates hyperproliferation and prevents excessive hyperkeratosis by shedding the corneocytes...
  6. doi request reprint Minocycline reduces remyelination by suppressing ciliary neurotrophic factor expression after cuprizone-induced demyelination
    Tatsuhide Tanaka
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical University, Asahikawa, Hokkaido, Japan
    J Neurochem 127:259-70. 2013
    ..In vitro, CNTF directly affected the differentiation of oligodendrocytic cells. These findings suggest that minocycline reduces remyelination by suppressing CNTF expression by microglia after cuprizone-induced demyelination...
  7. ncbi request reprint Regeneration of the abdominal postganglionic sympathetic system
    Masahiro Yamada
    Department of Anatomy 1, Asahikawa Medical College, Japan
    Neurosci Res 54:261-8. 2006
    ..Therefore, postganglionic sympathetic nerves in the abdomen are able to regenerate and reinnervation occurs even after the removal of sympathetic ganglia...
  8. ncbi request reprint Differential expression of protease M/neurosin in oligodendrocytes and their progenitors in an animal model of multiple sclerosis
    Ryuji Terayama
    Department of Anatomy, Asahikawa Medical College, 2 1 1 1 Midorigaoka Higashi, Asahikawa 078 8510, Japan
    Neurosci Lett 382:82-7. 2005
    ..These observations suggest that the differential expression of protease M/neurosin in mature oligodendrocytes and their progenitors is involved in the pathogenesis of MS and EAE...
  9. doi request reprint Three-dimensional ultra-structures of myelin and the axons in the spinal cord: application of SEM with the osmium maceration method to the central nervous system in two mouse models
    Taichi Nomura
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical University, Asahikawa 078 8510, Japan
    Neurosci Res 75:190-7. 2013
    ..This technique will be a powerful tool for identifying the mechanisms underlying the pathogenesis in demyelination...
  10. ncbi request reprint Differential expression of neuropsin and protease M/neurosin in oligodendrocytes after injury to the spinal cord
    Ryuji Terayama
    Department of Anatomy, Asahikawa Medical College, Asahikawa 078 8510, Japan
    Glia 48:91-101. 2004
    ..These findings suggest that each subpopulation of oligodendrocytes based on the expression of neuropsin and protease M/neurosin has different roles in the response of the spinal cord to injury as well as in normal homeostasis...
  11. ncbi request reprint Epidermal lamellar granules transport different cargoes as distinct aggregates
    Akemi Ishida-Yamamoto
    Department of Dermatology, Asahikawa Medical College, Asahikawa, Japan
    J Invest Dermatol 122:1137-44. 2004
    ..This study provides strong evidence for the sequential synthesis and independent trafficking of various LG cargoes, including for the first time CatD and KLK8, from TGN...
  12. ncbi request reprint [Kallikrein-family serine protease in the central nervous system]
    Shigetaka Yoshida
    Department of Anatomy 1, Asahikawa Medical College, Midorigaoka Higashi 2 1 1 1, Asahikawa, Hokkaido 078 8510, Japan
    Kaibogaku Zasshi 78:77-82. 2003
    ..These proteases in balance with inhibitors are implicated in the modulation of the extracellular environment...