Y Bando

Summary

Affiliation: Asahikawa Medical College
Country: Japan

Publications

  1. ncbi request reprint 150-kDa oxygen-regulated protein attenuates myocardial ischemia-reperfusion injury in rat heart
    Alexey N Aleshin
    Division of Cardiovascular Surgery, Department of Surgery, Osaka University Graduate School of Medicine, 2 2 Yamadaoka, Suita, Osaka 565 0871, Japan
    J Mol Cell Cardiol 38:517-25. 2005
  2. ncbi request reprint Phosphorylated PKR contributes the induction of GRP94 under ER stress
    Mototsugu Ito
    Graduate School of Pharmaceutical Science, The University of Tokyo, 7 3 1 Hongo, Bunkyo ku, Tokyo 13 0033, Japan
    Biochem Biophys Res Commun 360:615-20. 2007
  3. doi request reprint The functional role of stress proteins in ER stress mediated cell death
    Yoshio Bando
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical University, Asahikawa, 078 8510, Japan
    Anat Sci Int 87:14-23. 2012
  4. ncbi request reprint RA410/Sly1 suppresses MPP+ and 6-hydroxydopamine-induced cell death in SH-SY5Y cells
    Yoshio Bando
    Department of Anatomy and Neuroscience, Osaka University Graduate School of Medicine, Osaka 565 0871, Japan
    Neurobiol Dis 18:143-51. 2005
  5. ncbi request reprint Double-strand RNA dependent protein kinase (PKR) is involved in the extrastriatal degeneration in Parkinson's disease and Huntington's disease
    Yoshio Bando
    Department of Anatomy, Asahikawa Medical College, Midorigaoka Higashi 2 1 1 1, Asahikawa 078 8510, Japan
    Neurochem Int 46:11-8. 2005
  6. ncbi request reprint Implications of protease M/neurosin in myelination during experimental demyelination and remyelination
    Yoshio Bando
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical College, Asahikawa, Hokkaido 078 8510, Japan
    Neurosci Lett 405:175-80. 2006
  7. ncbi request reprint ORP150/HSP12A protects renal tubular epithelium from ischemia-induced cell death
    Yoshio Bando
    Department of Anatomy I, Asahikawa Medical College, Asahikawa, Hokkaido, Japan
    FASEB J 18:1401-3. 2004
  8. ncbi request reprint GRP94 reduces cell death in SH-SY5Y cells perturbated calcium homeostasis
    Y Bando
    Department of Anatomy and Neuroscience, Osaka University Graduate School of Medicine, Osaka 565 0871, Japan
    Apoptosis 9:501-8. 2004
  9. doi request reprint Differential changes in axonal conduction following CNS demyelination in two mouse models
    Yoshio Bando
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical College, Asahikawa, Hokkaido 078 8510, Japan
    Eur J Neurosci 28:1731-42. 2008
  10. ncbi request reprint GRP94 (94 kDa glucose-regulated protein) suppresses ischemic neuronal cell death against ischemia/reperfusion injury
    Yoshio Bando
    Department of Anatomy and Neuroscience, Osaka University Graduate School of Medicine, Osaka, Japan
    Eur J Neurosci 18:829-40. 2003

Collaborators

Detail Information

Publications28

  1. ncbi request reprint 150-kDa oxygen-regulated protein attenuates myocardial ischemia-reperfusion injury in rat heart
    Alexey N Aleshin
    Division of Cardiovascular Surgery, Department of Surgery, Osaka University Graduate School of Medicine, 2 2 Yamadaoka, Suita, Osaka 565 0871, Japan
    J Mol Cell Cardiol 38:517-25. 2005
    ..Our results indicated cytoprotective roles for ORP150 in rat heart and suggested a therapeutic role for the protein both in preventing cardiomyocyte death and in preserving contractile function after ischemic damage...
  2. ncbi request reprint Phosphorylated PKR contributes the induction of GRP94 under ER stress
    Mototsugu Ito
    Graduate School of Pharmaceutical Science, The University of Tokyo, 7 3 1 Hongo, Bunkyo ku, Tokyo 13 0033, Japan
    Biochem Biophys Res Commun 360:615-20. 2007
    ..These results suggest that a novel mechanism other than ERSE-dependent mRNA transcription is required for the induction of GRP94 and phosphorylation of PKR contributes to the induction of GRP94 under ER stress...
  3. doi request reprint The functional role of stress proteins in ER stress mediated cell death
    Yoshio Bando
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical University, Asahikawa, 078 8510, Japan
    Anat Sci Int 87:14-23. 2012
    ..These findings suggest that hypoxia and oxidative stress target the ER and secretory pathway, resulting in ER stress, and that these proteins exert cytoprotective effects in various diseases associated with ER stress...
  4. ncbi request reprint RA410/Sly1 suppresses MPP+ and 6-hydroxydopamine-induced cell death in SH-SY5Y cells
    Yoshio Bando
    Department of Anatomy and Neuroscience, Osaka University Graduate School of Medicine, Osaka 565 0871, Japan
    Neurobiol Dis 18:143-51. 2005
    ..These results suggest that RA410/Sly1 plays an important cytoprotective role in MPP+ and 6-OHDA-induced cellular perturbation...
  5. ncbi request reprint Double-strand RNA dependent protein kinase (PKR) is involved in the extrastriatal degeneration in Parkinson's disease and Huntington's disease
    Yoshio Bando
    Department of Anatomy, Asahikawa Medical College, Midorigaoka Higashi 2 1 1 1, Asahikawa 078 8510, Japan
    Neurochem Int 46:11-8. 2005
    ..These results suggest that a close association exists between PKR and extrastriatal degeneration in PD and HD pathology...
  6. ncbi request reprint Implications of protease M/neurosin in myelination during experimental demyelination and remyelination
    Yoshio Bando
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical College, Asahikawa, Hokkaido 078 8510, Japan
    Neurosci Lett 405:175-80. 2006
    ..Our data emphasize that the maintenance of myelination is an important function of protease M/neurosin in OLGs, suggesting its relation to the oligodendroglial response to myelin disorders...
  7. ncbi request reprint ORP150/HSP12A protects renal tubular epithelium from ischemia-induced cell death
    Yoshio Bando
    Department of Anatomy I, Asahikawa Medical College, Asahikawa, Hokkaido, Japan
    FASEB J 18:1401-3. 2004
    ..These data are consistent with the possibility that ORP150 exerts cytoprotective effects in renal tubular epithelia subjected to I/R injury and suggest a key role for ER stress in the renal tubular response to acute renal failure...
  8. ncbi request reprint GRP94 reduces cell death in SH-SY5Y cells perturbated calcium homeostasis
    Y Bando
    Department of Anatomy and Neuroscience, Osaka University Graduate School of Medicine, Osaka 565 0871, Japan
    Apoptosis 9:501-8. 2004
    ..These results indicated that suppression of GRP94 is associated with accelerated cell death. Moreover, expression of GRP94 suppressed A23187-induced cell death and stabilized calcium homeostasis...
  9. doi request reprint Differential changes in axonal conduction following CNS demyelination in two mouse models
    Yoshio Bando
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical College, Asahikawa, Hokkaido 078 8510, Japan
    Eur J Neurosci 28:1731-42. 2008
    ....
  10. ncbi request reprint GRP94 (94 kDa glucose-regulated protein) suppresses ischemic neuronal cell death against ischemia/reperfusion injury
    Yoshio Bando
    Department of Anatomy and Neuroscience, Osaka University Graduate School of Medicine, Osaka, Japan
    Eur J Neurosci 18:829-40. 2003
    ..These results confirmed that GRP94 could suppress ischemic injury to neurons, suggesting that gene transfer of GRP94 into the brain may have therapeutic potential in the treatment of cerebrovascular disease...
  11. ncbi request reprint Neuropsin promotes oligodendrocyte death, demyelination and axonal degeneration after spinal cord injury
    R Terayama
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical College, 2 1 1 1 Midorigaoka Higashi, Asahikawa 078 8510, Japan
    Neuroscience 148:175-87. 2007
    ..These observations suggest that neuropsin is involved in the secondary phase of the pathogenesis of SCI mediated by demyelination, oligodendrocyte death, and axonal degeneration...
  12. ncbi request reprint Hypothermic treatment restores glucose regulated protein 78 (GRP78) expression in ischemic brain
    M Aoki
    Department of Traumatology, Graduate School of Medicine, Osaka University, 2 2 Yamadaoka, Suita, 565 0871, Osaka, Japan
    Brain Res Mol Brain Res 95:117-28. 2001
    ..These results suggest that reduction in GRP78 expression contributes to cell damage in the ischemic brain and that hypothermia-mediated restoration of GRP78 expression is one mechanism that enhances neuronal survival...
  13. doi request reprint In vivo analysis of kallikrein-related peptidase 6 (KLK6) function in oligodendrocyte development and the expression of myelin proteins
    K Murakami
    Department of Functional Anatomy and Neuroscience, Asahikawa Medical University, Asahikawa, Japan
    Neuroscience 236:1-11. 2013
    ..These results indicate that KLK6 plays a functional role in oligodendrocyte development and the expression of myelin proteins...
  14. ncbi request reprint 150-kDa oxygen-regulated protein (ORP150) functions as a novel molecular chaperone in MDCK cells
    Y Bando
    Department of Anatomy and Neuroscience, Osaka University Graduate School of Medicine, Suita City, Japan
    Am J Physiol Cell Physiol 278:C1172-82. 2000
    ..These data indicate that ORP150 may function as a unique molecular chaperone in renal epithelial cells by facilitating protein transport/maturation in an environment where less ATP is accessible...
  15. pmc Involvement of caspase-4 in endoplasmic reticulum stress-induced apoptosis and Abeta-induced cell death
    Junichi Hitomi
    Department of Anatomy and Neuroscience, Graduate School of Medicine, Osaka University, Suita, Osaka 565 0871, Japan
    J Cell Biol 165:347-56. 2004
    ..Thus, caspase-4 can function as an ER stress-specific caspase in humans, and may be involved in pathogenesis of Alzheimer's disease...
  16. ncbi request reprint Involvement of neuropsin in the pathogenesis of experimental autoimmune encephalomyelitis
    Ryuji Terayama
    Department of Anatomy, Asahikawa Medical College, Asahikawa, Japan
    Glia 52:108-18. 2005
    ..These observations suggest that neuropsin is involved in the pathogenesis of EAE mediated by demyelination and oligodendroglial death...
  17. ncbi request reprint Differential expression of protease M/neurosin in oligodendrocytes and their progenitors in an animal model of multiple sclerosis
    Ryuji Terayama
    Department of Anatomy, Asahikawa Medical College, 2 1 1 1 Midorigaoka Higashi, Asahikawa 078 8510, Japan
    Neurosci Lett 382:82-7. 2005
    ..These observations suggest that the differential expression of protease M/neurosin in mature oligodendrocytes and their progenitors is involved in the pathogenesis of MS and EAE...
  18. ncbi request reprint Differential expression of neuropsin and protease M/neurosin in oligodendrocytes after injury to the spinal cord
    Ryuji Terayama
    Department of Anatomy, Asahikawa Medical College, Asahikawa 078 8510, Japan
    Glia 48:91-101. 2004
    ..These findings suggest that each subpopulation of oligodendrocytes based on the expression of neuropsin and protease M/neurosin has different roles in the response of the spinal cord to injury as well as in normal homeostasis...
  19. ncbi request reprint Novel function of PS2V: change in conformation of tau proteins
    Atsuko Nishikawa
    Department of Anatomy and Neuroscience, Graduate School of Medicine, Osaka University, 2 2 Yamadaoka, Suita, Osaka, Japan
    Biochem Biophys Res Commun 318:435-8. 2004
    ..In this manuscript, we make the first report of PS2V alterations in the conformation of the tau protein (unknown form of tau) in the human neuroblastoma cell line...
  20. ncbi request reprint Kallikrein 8 is involved in skin desquamation in cooperation with other kallikreins
    Mari Kishibe
    Departments of Structural Anatomy and Neuroscience and Dermatology, Asahikawa Medical College, Midorigaoka Higashi 2 1 1 1, Asahikawa 078 8510, Japan
    J Biol Chem 282:5834-41. 2007
    ..We propose that in psoriatic lesion, KLK8 modulates hyperproliferation and prevents excessive hyperkeratosis by shedding the corneocytes...
  21. ncbi request reprint Regeneration of the abdominal postganglionic sympathetic system
    Masahiro Yamada
    Department of Anatomy 1, Asahikawa Medical College, Japan
    Neurosci Res 54:261-8. 2006
    ..Therefore, postganglionic sympathetic nerves in the abdomen are able to regenerate and reinnervation occurs even after the removal of sympathetic ganglia...
  22. ncbi request reprint Novel interaction between HMGA1a and StIP1 in murine terminally differentiated retina
    Hiroaki Okuda
    Department of Anatomy and Neuroscience, Graduate School of Medicine, Osaka University, Suita, Osaka, Japan
    Mol Cell Neurosci 33:81-7. 2006
    ....
  23. ncbi request reprint Possible involvement of the expression and phosphorylation of N-Myc in the induction of HMGA1a by hypoxia in the human neuroblastoma cell line
    Takeshi Yanagita
    Department of Anatomy and Neuroscience, Graduate School of Medicine, Osaka University, 2 2 Yamadaoka, Suita, Osaka, Japan
    Neurosci Lett 374:47-52. 2005
    ..Such signal consolidation processes could play a role in neuronal survival after hypoxia in neurons...
  24. ncbi request reprint HMGA1a: sequence-specific RNA-binding factor causing sporadic Alzheimer's disease-linked exon skipping of presenilin-2 pre-mRNA
    Takayuki Manabe
    Department of Anatomy and Neuroscience, Graduate School of Medicine, Osaka University, Suita, Osaka 565 0871, Japan
    Genes Cells 12:1179-91. 2007
    ..These results provide insights into the molecular basis for PS2V-associated neurodegenerative diseases that are initiated by specific RNA binding of HMGA1a...
  25. pmc An RNA-dependent protein kinase is involved in tunicamycin-induced apoptosis and Alzheimer's disease
    Reiko Onuki
    Gene Function Research Center, National Institute of Advanced Industrial Science and Technology, Higashi, Tsukuba Science City, Japan
    EMBO J 23:959-68. 2004
    ..We also detected the accumulation of phosphorylated PKR in the nuclei of autopsied brain tissues in Alzheimer's disease. Thus, PKR might play a role in ER stress-induced apoptosis and in Alzheimer's disease...
  26. ncbi request reprint Immobilization of proteins on boron nitride nanotubes
    Chunyi Zhi
    Advanced Materials Laboratory, National Institute for Materials Science NIMS, Namiki 1 1, Tsukuba, Ibaraki 305 0044, Japan
    J Am Chem Soc 127:17144-5. 2005
    ..For the most effective immobilization, noncovalently functionalized BNNTs should be used. The effect of immobilization was studied using high-resolution transmission electron microscopy and energy dispersion spectroscopy...
  27. ncbi request reprint CD11b+Ly-6C(hi) suppressive monocytes in experimental autoimmune encephalomyelitis
    Bing Zhu
    Center for Neurologic Diseases, Brigham and Women s Hospital, Harvard Medical School, Boston, MA 02115, USA
    J Immunol 179:5228-37. 2007
    ..Further understanding of suppressive monocytes in autoimmune disease models may have important clinical implications for human autoimmune diseases...
  28. pmc Latency-associated peptide identifies a novel CD4+CD25+ regulatory T cell subset with TGFbeta-mediated function and enhanced suppression of experimental autoimmune encephalomyelitis
    Mei Ling Chen
    Center for Neurologic Diseases, Brigham and Women s Hospital, Harvard Medical School, Boston, MA 02115, USA
    J Immunol 180:7327-37. 2008
    ..Our work helps elucidate the ambiguity concerning the role of TGFbeta in CD4(+)CD25(+) Treg-mediated suppression and indicates that LAP is an authentic marker able to identify a TGFbeta-expressing CD4(+)CD25(+) Treg subset...