Lorella M T Canzoniero

Summary

Affiliation: Via Port'Arsa
Country: Italy

Publications

  1. ncbi request reprint Calcium in Alzheimer's disease pathogenesis: too much, too little or in the wrong place?
    Lorella M T Canzoniero
    Department of Biology and Environmental Sciences, University of Sannio, Benevento, Italy
    J Alzheimers Dis 8:147-54; discussion 209-15. 2005
  2. doi request reprint Intracellular and plasma membrane-initiated pathways involved in the [Ca2+]i elevations induced by iodothyronines (T3 and T2) in pituitary GH3 cells
    Adelaide Del Viscovo
    Dipartimento di Scienze per la Biologia, la Geologia e l Ambiente, Universita del Sannio, Piazza Guerrazzi 1, Benevento, Italy
    Am J Physiol Endocrinol Metab 302:E1419-30. 2012
  3. ncbi request reprint Involvement of the nitric oxide/protein kinase G pathway in polychlorinated biphenyl-induced cell death in SH-SY 5Y neuroblastoma cells
    Lorella M T Canzoniero
    Department of Biological and Environmental Sciences, University of Sannio, Benevento, Italy
    J Neurosci Res 84:692-7. 2006
  4. ncbi request reprint Raising intracellular calcium attenuates neuronal apoptosis triggered by staurosporine or oxygen-glucose deprivation in the presence of glutamate receptor blockade
    Lorella M T Canzoniero
    Center for the Study of Nervous System Injury and Department of Neurology, Washington University School of Medicine, St Louis, MO 63110, USA
    Neurobiol Dis 15:520-8. 2004
  5. ncbi request reprint NMDA antagonists exacerbate neuronal death caused by proteasome inhibition in cultured cortical and striatal neurons
    B Joy Snider
    Center for the Study of Nervous System Injury and Department of Neurology, Washington University School of Medicine, St Louis, MO, USA
    Eur J Neurosci 15:419-28. 2002
  6. ncbi request reprint Depolarization-induced 65zinc influx into cultured cortical neurons
    Christian T Sheline
    Center for the Study of Nervous System Injury, Department of Neurology, Washington University School of Medicine, 660 South Euclid Avenue, St Louis, Missouri 63110, USA
    Neurobiol Dis 10:41-53. 2002
  7. ncbi request reprint Membrane-permeant chelators can attenuate Zn2+-induced cortical neuronal death
    Lorella M T Canzoniero
    Washington University School of Medicine, 660 S Euclid Avenue, St Louis, MO 63110, USA
    Neuropharmacology 45:420-8. 2003
  8. pmc Mild acidosis enhances AMPA receptor-mediated intracellular zinc mobilization in cortical neurons
    Valerio Frazzini
    Department of Basic and Applied Medical Science, Molecular Neurology Unit, CeSI Center for Excellence on Aging, University G D Annunzio, Chieti, Italy
    Mol Med 13:356-61. 2007
  9. doi request reprint Decreased presence of perforated synapses in a triple-transgenic mouse model of Alzheimer's disease
    Carlo Bertoni-Freddari
    Neurobiology of Aging Laboratory, INRCA Research Department, Ancona, Italy
    Rejuvenation Res 11:309-13. 2008

Collaborators

Detail Information

Publications9

  1. ncbi request reprint Calcium in Alzheimer's disease pathogenesis: too much, too little or in the wrong place?
    Lorella M T Canzoniero
    Department of Biology and Environmental Sciences, University of Sannio, Benevento, Italy
    J Alzheimers Dis 8:147-54; discussion 209-15. 2005
    ....
  2. doi request reprint Intracellular and plasma membrane-initiated pathways involved in the [Ca2+]i elevations induced by iodothyronines (T3 and T2) in pituitary GH3 cells
    Adelaide Del Viscovo
    Dipartimento di Scienze per la Biologia, la Geologia e l Ambiente, Universita del Sannio, Piazza Guerrazzi 1, Benevento, Italy
    Am J Physiol Endocrinol Metab 302:E1419-30. 2012
    ..Collectively, the present results suggest that T2 and T3 exert short-term nongenomic effects on intracellular calcium and NO by modulating plasma membrane and mitochondrial pathways that differ between these iodothyronines...
  3. ncbi request reprint Involvement of the nitric oxide/protein kinase G pathway in polychlorinated biphenyl-induced cell death in SH-SY 5Y neuroblastoma cells
    Lorella M T Canzoniero
    Department of Biological and Environmental Sciences, University of Sannio, Benevento, Italy
    J Neurosci Res 84:692-7. 2006
    ..These results suggest that PCB-induced cell death in neuroblastoma cells is mediated by an activation of the cGMP/PKG pathway triggered by NO production...
  4. ncbi request reprint Raising intracellular calcium attenuates neuronal apoptosis triggered by staurosporine or oxygen-glucose deprivation in the presence of glutamate receptor blockade
    Lorella M T Canzoniero
    Center for the Study of Nervous System Injury and Department of Neurology, Washington University School of Medicine, St Louis, MO 63110, USA
    Neurobiol Dis 15:520-8. 2004
    ..Raising [Ca(2+)](i) either by activating voltage-sensitive Ca(2+) channels with (-) Bay K8644 or by application of low concentrations of kainate attenuated both staurosporine and oxygen-glucose deprivation-induced apoptosis...
  5. ncbi request reprint NMDA antagonists exacerbate neuronal death caused by proteasome inhibition in cultured cortical and striatal neurons
    B Joy Snider
    Center for the Study of Nervous System Injury and Department of Neurology, Washington University School of Medicine, St Louis, MO, USA
    Eur J Neurosci 15:419-28. 2002
    ..In particular, concern is raised that glutamate receptor antagonists might exacerbate, rather than attenuate, proteasome inhibition-induced neuronal death...
  6. ncbi request reprint Depolarization-induced 65zinc influx into cultured cortical neurons
    Christian T Sheline
    Center for the Study of Nervous System Injury, Department of Neurology, Washington University School of Medicine, 660 South Euclid Avenue, St Louis, Missouri 63110, USA
    Neurobiol Dis 10:41-53. 2002
    ..K(+)-induced, Gd(3+)-sensitive (45)Ca(2+) accumulation but not (65)Zn(2+) accumulation was sharply attenuated by lowering extracellular pH to 6.6...
  7. ncbi request reprint Membrane-permeant chelators can attenuate Zn2+-induced cortical neuronal death
    Lorella M T Canzoniero
    Washington University School of Medicine, 660 S Euclid Avenue, St Louis, MO 63110, USA
    Neuropharmacology 45:420-8. 2003
    ..These data point to a potential therapeutic role for membrane-permeant Zn(2+) chelators, perhaps especially possessing low Zn(2+) affinity, in attenuating neuronal death after certain acute insults...
  8. pmc Mild acidosis enhances AMPA receptor-mediated intracellular zinc mobilization in cortical neurons
    Valerio Frazzini
    Department of Basic and Applied Medical Science, Molecular Neurology Unit, CeSI Center for Excellence on Aging, University G D Annunzio, Chieti, Italy
    Mol Med 13:356-61. 2007
    ..Additionally, our results show that selective Ca2+ imaging mandates the use of intracellular heavy metal chelators to avoid confounding effects of endogenous metals such as Zn2+...
  9. doi request reprint Decreased presence of perforated synapses in a triple-transgenic mouse model of Alzheimer's disease
    Carlo Bertoni-Freddari
    Neurobiology of Aging Laboratory, INRCA Research Department, Ancona, Italy
    Rejuvenation Res 11:309-13. 2008
    ....