C Banfi

Summary

Affiliation: Centro Cardiologico Monzino
Country: Italy

Publications

  1. ncbi Statins prevent tissue factor induction by protease-activated receptors 1 and 2 in human umbilical vein endothelial cells in vitro
    C Banfi
    Centro Cardiologico Monzino IRCCS, Milan, Italy
    J Thromb Haemost 9:1608-19. 2011
  2. ncbi Mitochondrial reactive oxygen species: a common pathway for PAR1- and PAR2-mediated tissue factor induction in human endothelial cells
    C Banfi
    Monzino Cardiologic Center IRCCS, Milan, Italy
    J Thromb Haemost 7:206-16. 2009
  3. ncbi Tissue factor induction by protease-activated receptor 1 requires intact caveolin-enriched membrane microdomains in human endothelial cells
    C Banfi
    Monzino Cardiologic Centre IRCCS, and Department of Pharmacological Sciences, University of Milan, Via Parea 4, Milan, Italy
    J Thromb Haemost 5:2437-44. 2007
  4. ncbi Oxidized LDLs influence thrombotic response and cyclooxygenase 2
    C Banfi
    Department of Pharmacological Sciences, University of Milan, Milan, Italy
    Prostaglandins Leukot Essent Fatty Acids 67:169-73. 2002
  5. ncbi Proteomic profile of differentially expressed plasma proteins from dystrophic mice and following suberoylanilide hydroxamic acid treatment
    C Colussi
    Centro Cardiologico Monzino IRCCS, Milano, Italy
    Proteomics Clin Appl 4:71-83. 2010
  6. ncbi [PAI-1, the primary plasmatic inhibitor of fibrinolysis. Physiopathologic role and molecular mechanisms]
    C Banfi
    Dipartimento di Scienze Farmacologiche, , Milan, Italy
    Minerva Endocrinol 27:181-91. 2002
  7. ncbi Surfactant protein B and RAGE increases in the plasma during cardiopulmonary bypass: a pilot study
    P Agostoni
    Centro Cardiologico Monzino, IRCCS, Dept of Cardiovascular Sciences, University of Milan, Milan, Italy
    Eur Respir J 37:841-7. 2011

Collaborators

  • E Tremoli
  • P Agostoni
  • C Colussi
  • S Straino
  • M C Capogrossi
  • Dante Rotili
  • M Brioschi
  • Antonello Mai
  • F Spallotta
  • Carlo Gaetano

Detail Information

Publications7

  1. ncbi Statins prevent tissue factor induction by protease-activated receptors 1 and 2 in human umbilical vein endothelial cells in vitro
    C Banfi
    Centro Cardiologico Monzino IRCCS, Milan, Italy
    J Thromb Haemost 9:1608-19. 2011
    ....
  2. ncbi Mitochondrial reactive oxygen species: a common pathway for PAR1- and PAR2-mediated tissue factor induction in human endothelial cells
    C Banfi
    Monzino Cardiologic Center IRCCS, Milan, Italy
    J Thromb Haemost 7:206-16. 2009
    ..PARs regulate a broad range of cellular functions and are involved in the pathogenesis of inflammatory disorders. Moreover, PAR1 and PAR2 activation in the endothelium shifts it toward a prothrombotic condition...
  3. ncbi Tissue factor induction by protease-activated receptor 1 requires intact caveolin-enriched membrane microdomains in human endothelial cells
    C Banfi
    Monzino Cardiologic Centre IRCCS, and Department of Pharmacological Sciences, University of Milan, Via Parea 4, Milan, Italy
    J Thromb Haemost 5:2437-44. 2007
    ..Signaling through PAR1 and PAR2 shifts the endothelium towards a prothrombotic phenotype, thereby exacerbating the initial pathophysiologic condition...
  4. ncbi Oxidized LDLs influence thrombotic response and cyclooxygenase 2
    C Banfi
    Department of Pharmacological Sciences, University of Milan, Milan, Italy
    Prostaglandins Leukot Essent Fatty Acids 67:169-73. 2002
    ..Among lipid constituents of oxLDLs, Ox-PAPC, a mixture of oxidized arachidonic acid-containing phospholipids, prevents Cox-2 expression, suggesting that it could be considered responsible for the biological activity of oxLDLs...
  5. ncbi Proteomic profile of differentially expressed plasma proteins from dystrophic mice and following suberoylanilide hydroxamic acid treatment
    C Colussi
    Centro Cardiologico Monzino IRCCS, Milano, Italy
    Proteomics Clin Appl 4:71-83. 2010
    ..The further development of these compounds as drugs for DMD treatment is currently hampered by the lack of knowledge about DIs effect in large dystrophic animal models and that of suitable biomarkers to monitor their efficacy...
  6. ncbi [PAI-1, the primary plasmatic inhibitor of fibrinolysis. Physiopathologic role and molecular mechanisms]
    C Banfi
    Dipartimento di Scienze Farmacologiche, , Milan, Italy
    Minerva Endocrinol 27:181-91. 2002
    ..It now seems reasonable to view PAI-1 as one of the factors contributing to the complex gene-environment interactions involved in the formation and dissolution of thrombi...
  7. ncbi Surfactant protein B and RAGE increases in the plasma during cardiopulmonary bypass: a pilot study
    P Agostoni
    Centro Cardiologico Monzino, IRCCS, Dept of Cardiovascular Sciences, University of Milan, Milan, Italy
    Eur Respir J 37:841-7. 2011
    ..This behaviour was confirmed when RAGE and SPB were normalised for protein content. Parallel changes were observed for immature SPB. Plasma RAGE and SPBs are sensitive and rapid markers of lung distress...