A Ben-Ze'ev

Summary

Affiliation: Weizmann Institute of Science
Country: Israel

Publications

  1. ncbi request reprint The use of two-dimensional gel electrophoresis in studies on the role of cytoskeletal plaque proteins as tumor suppressors
    A Ben-Ze'ev
    Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot, Israel
    Electrophoresis 17:1752-63. 1996
  2. ncbi request reprint Cytoskeletal and adhesion proteins as tumor suppressors
    A Ben-Ze'ev
    Department of Molecular Cell Biology, The Weizmann Institute of Science, Rehovot 76100, Israel
    Curr Opin Cell Biol 9:99-108. 1997
  3. ncbi request reprint Differential molecular interactions of beta-catenin and plakoglobin in adhesion, signaling and cancer
    A Ben-Ze'ev
    Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot, Israel
    Curr Opin Cell Biol 10:629-39. 1998
  4. ncbi request reprint The dual role of cytoskeletal anchor proteins in cell adhesion and signal transduction
    A Ben-Ze'ev
    Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot, Israel
    Ann N Y Acad Sci 886:37-47. 1999
  5. pmc The cyclin D1 gene is a target of the beta-catenin/LEF-1 pathway
    M Shtutman
    Department of Molecular Cell Biology, The Weizmann Institute of Science, Rehovot 76100, Israel
    Proc Natl Acad Sci U S A 96:5522-7. 1999
  6. ncbi request reprint Plakoglobin and beta-catenin: protein interactions, regulation and biological roles
    J Zhurinsky
    Department of Molecular Cell Biology, The Weizmann Institute of Science, Rehovot, Israel
    J Cell Sci 113:3127-39. 2000
  7. ncbi request reprint Differential interaction of plakoglobin and beta-catenin with the ubiquitin-proteasome system
    E Sadot
    Department of Molecular Cell Biology, The Weizmann Institute of Science, Rehovot, Israel
    Oncogene 19:1992-2001. 2000
  8. ncbi request reprint Modulation of alpha-actinin levels affects cell motility and confers tumorigenicity on 3T3 cells
    U Gluck
    Department of Molecular Genetics and Virology, Weizmann Institute of Science, Rehovot, Israel
    J Cell Sci 107:1773-82. 1994
  9. pmc Transient induction of vinculin gene expression in 3T3 fibroblasts stimulated by serum-growth factors
    A Ben-Ze'ev
    Department of Molecular Genetics and Virology, Weizmann Institute of Science, Rehovot, Israel
    Cell Regul 1:621-36. 1990
  10. doi request reprint Global analysis of L1-transcriptomes identified IGFBP-2 as a target of ezrin and NF-κB signaling that promotes colon cancer progression
    A Ben-Shmuel
    Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot, Israel
    Oncogene 32:3220-30. 2013

Collaborators

Detail Information

Publications11

  1. ncbi request reprint The use of two-dimensional gel electrophoresis in studies on the role of cytoskeletal plaque proteins as tumor suppressors
    A Ben-Ze'ev
    Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot, Israel
    Electrophoresis 17:1752-63. 1996
    ..Here we demonstrate the usefulness of the application of two dimensional (2-D) gel electrophoresis in these studies...
  2. ncbi request reprint Cytoskeletal and adhesion proteins as tumor suppressors
    A Ben-Ze'ev
    Department of Molecular Cell Biology, The Weizmann Institute of Science, Rehovot 76100, Israel
    Curr Opin Cell Biol 9:99-108. 1997
    ..In addition, such junctional plaque proteins have been shown to act as effective suppressors of tumorigenesis...
  3. ncbi request reprint Differential molecular interactions of beta-catenin and plakoglobin in adhesion, signaling and cancer
    A Ben-Ze'ev
    Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot, Israel
    Curr Opin Cell Biol 10:629-39. 1998
    ....
  4. ncbi request reprint The dual role of cytoskeletal anchor proteins in cell adhesion and signal transduction
    A Ben-Ze'ev
    Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot, Israel
    Ann N Y Acad Sci 886:37-47. 1999
    ....
  5. pmc The cyclin D1 gene is a target of the beta-catenin/LEF-1 pathway
    M Shtutman
    Department of Molecular Cell Biology, The Weizmann Institute of Science, Rehovot 76100, Israel
    Proc Natl Acad Sci U S A 96:5522-7. 1999
    ..Increased beta-catenin levels may thus promote neoplastic conversion by triggering cyclin D1 gene expression and, consequently, uncontrolled progression into the cell cycle...
  6. ncbi request reprint Plakoglobin and beta-catenin: protein interactions, regulation and biological roles
    J Zhurinsky
    Department of Molecular Cell Biology, The Weizmann Institute of Science, Rehovot, Israel
    J Cell Sci 113:3127-39. 2000
    ..The functional difference between catenins is reflected in their differential involvement in embryonic development and cancer progression...
  7. ncbi request reprint Differential interaction of plakoglobin and beta-catenin with the ubiquitin-proteasome system
    E Sadot
    Department of Molecular Cell Biology, The Weizmann Institute of Science, Rehovot, Israel
    Oncogene 19:1992-2001. 2000
    ....
  8. ncbi request reprint Modulation of alpha-actinin levels affects cell motility and confers tumorigenicity on 3T3 cells
    U Gluck
    Department of Molecular Genetics and Virology, Weizmann Institute of Science, Rehovot, Israel
    J Cell Sci 107:1773-82. 1994
    ....
  9. pmc Transient induction of vinculin gene expression in 3T3 fibroblasts stimulated by serum-growth factors
    A Ben-Ze'ev
    Department of Molecular Genetics and Virology, Weizmann Institute of Science, Rehovot, Israel
    Cell Regul 1:621-36. 1990
    ....
  10. doi request reprint Global analysis of L1-transcriptomes identified IGFBP-2 as a target of ezrin and NF-κB signaling that promotes colon cancer progression
    A Ben-Shmuel
    Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot, Israel
    Oncogene 32:3220-30. 2013
    ..Finally, we found that IGFBP-2 and L1 can form a molecular complex suggesting that L1-mediated signaling by the L1-ezrin-NF-κB pathway, that induces IGFBP-2 expression, has an important role in CRC progression. ..
  11. pmc Differential mechanisms of LEF/TCF family-dependent transcriptional activation by beta-catenin and plakoglobin
    J Zhurinsky
    Department of Molecular Cell Biology, The Weizmann Institute of Science, Rehovot, 76100, Israel
    Mol Cell Biol 20:4238-52. 2000
    ..These results suggest that overexpression of plakoglobin does not directly activate transcription and that formation of catenin-LEF-DNA complexes is negatively regulated by the catenin N- and C-terminal domains...