Research Topics
| M K J SiddiquiSummaryCountry: India Publications
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Publications
Biomonitoring of organochlorines in women with benign and malignant breast diseaseM K J Siddiqui
Analytical Toxicology, Industrial Toxicology Research Centre, P O Box No 80, M G Marg, Lucknow 226 001, India
Environ Res 98:250-7. 2005....
Comparison of some trace elements concentration in blood, tumor free breast and tumor tissues of women with benign and malignant breast lesions: an Indian studyM K J Siddiqui
Analytical Toxicology, Industrial Toxicology Research Centre P O Box 80, M G Marg, Lucknow 226001, India
Environ Int 32:630-7. 2006..Further, modulation of trace elements level in both benign and malignant breast diseases patients may be of potential to be used as diagnostic marker of the disease process and its possible relationship etiologically...
Persistent chlorinated pesticides and intra-uterine foetal growth retardation: a possible associationM K J Siddiqui
Industrial Toxicology Research Centre, Post Box No 80, M G Marg, Lucknow 226 001, India
Int Arch Occup Environ Health 76:75-80. 2003..To examine the association between DDT (dichlorodiphenyl trichloroethane) and HCH (hexachlorocyclohexane) exposure and intra-uterine growth retardation (IUGR, <10th percentile of birth weight for gestational age)...
Blood levels of polycyclic aromatic hydrocarbons in children and their association with oxidative stress indices: an Indian perspectiveVipul K Singh
Analytical Toxicology Section, Industrial Toxicology Research Centre P O Box 80, M G Marg, Lucknow 226001, India
Clin Biochem 41:152-61. 2008....
Delta-aminolevulinic acid dehydratase inhibition and oxidative stress in relation to blood lead among urban adolescentsM Ahamed
Analytical Toxicology, Industrial Toxicology Research Centre, PO Box-80, MG Marg, Lucknow 226 001, India
Hum Exp Toxicol 25:547-53. 2006..The potential of oxidative stress parameters to be used as biomarkers of lead toxicity warranted further investigation...
Oxidative stress and neurological disorders in relation to blood lead levels in childrenM Ahamed
Industrial Toxicology Research Centre, M G Marg, Lucknow, India
Redox Rep 13:117-22. 2008..Lead-induced oxidative stress as an underlying mechanism for neurological diseases in children warranted further investigation...
Biomonitoring of organochlorines, glutathione, lipid peroxidation and cholinesterase activity among pesticide sprayers in mango orchardsVipul K Singh
Analytical Toxicology Section, Industrial Toxicology Research Centre P O Box 80, M G Marg, Lucknow 226001, India
Clin Chim Acta 377:268-72. 2007..We determined the pesticide exposure levels along with their biochemical and clinical effects in 31 sprayers, compared with 18 controls...
Childhood aplastic anaemia in Lucknow, India: incidence, organochlorines in the blood and review of case reports following exposure to pesticidesM Ahamed
Analytical Toxicology, Industrial Toxicology Research Centre, P.O. Box-80, M.G. Marg, Lucknow-226 001, India
Clin Biochem 39:762-6. 2006....
Possible mechanism of pesticide toxicity-related oxidative stress leading to airway narrowingC Kesavachandran
Epidemiology Section, Industrial Toxicology Research Centre, Lucknow, India
Redox Rep 11:159-62. 2006..001) in sprayers compared to the controls suggest inhibition of cholinesterase activity. The present study shows that pesticide toxicity might lead to oxidative stress and airway narrowing resulting in decreased peak expiratory flow rate...
Blood levels of polycyclic aromatic hydrocarbons in children of Lucknow, IndiaVipul K Singh
Analytical Toxicology Section, Industrial Toxicology Research Centre, MG Marg, Lucknow, India
Arch Environ Contam Toxicol 54:348-54. 2008..Further study with a larger sample size and accompanying environmental data is desired to validate the findings of this pilot study and strengthen the database of PAHs exposure in India...
Interaction of lead with some essential trace metals in the blood of anemic children from Lucknow, IndiaM Ahamed
Analytical Toxicology Division, Industrial Toxicology Research Centre, P O Box 80, M G Marg, Lucknow 226 001, India
Clin Chim Acta 377:92-7. 2007..We performed a cross-sectional study to evaluate the association of blood lead levels>or=10 microg/dl with anemia, and its effect on hematological system and some essential trace metals in children...
Environmental exposure to lead and its correlation with biochemical indices in childrenM Ahamed
Analytical Toxicology, Industrial Toxicology Research Centre, M.G. Marg, Lucknow-226 001, India
Sci Total Environ 346:48-55. 2005....
Low level lead exposure and oxidative stress: current opinionsM Ahamed
Analytical Toxicology, Industrial Toxicology Research Centre, P O Box 80, M G Marg, Lucknow 226 001, India
Clin Chim Acta 383:57-64. 2007..The present review focuses on mechanisms for lead-induced oxidative stress and relevance of oxidative stress to lead-related human disease with low lead exposure...
Environmental exposure to lead as a risk for prostate cancerM K J Siddiqui
Industrial Toxicology Research Centre, Post Box No 80, M G Marg, Lucknow 226 001, India
Biomed Environ Sci 15:298-305. 2002..To evaluate the possible role of environmental exposure to lead as a risk factor for prostate pathology in patients suffering from prostate cancer (PCA) and benign prostate hyperplasia (BPH)...
Association of maternal blood pressure and hemoglobin level with organochlorines in human milkM K J Siddiqui
Analytical Toxicology, Industrial Toxicology Research Centre, Lucknow, India
Hum Exp Toxicol 21:1-6. 2002..05). These results highlight the need for periodic monitoring to determine organochlorine contamination in human milk and assess their impact on mother and on neonatal health...
Reversal of lead-induced oxidative stress by chelating agent, antioxidant, or their combination in the ratS K Tandon
Chemical Toxicology, Industrial Toxicology Research Center, 80, Lucknow 226001, India
Environ Res 90:61-6. 2002..The ideal treatment of lead poisoning seems to be a combination of a lead chelator and an antioxidant...
Some essential elements in maternal and cord blood in relation to birth weight and gestational age of the babyS Srivastava
University of Faizabad, India
Biol Trace Elem Res 86:97-105. 2002..Although, there are many confounders of low birth weight and preterm deliveries, a diminished placental transfer of these essential elements could be one of the several etiological factors for low birth weight of newborns...
Hematological and clinical chemistry changes induced by subchronic dosing of a novel phosphorothionate (RPR-V) in Wistar male and female ratsM F Rahman
Biochemical Toxicology, Biology Division, Indian Institute of Chemical Technology, Hyderabad, India
Drug Chem Toxicol 29:95-110. 2006..Alterations in these parameters show toxic stress in the treated animals especially on blood and blood-forming organs...
Biochemical effects of vepacide (from Azadirachta indica) on Wistar rats during subchronic exposureM F Rahman
Biochemical Toxicology, Biology Division, Indian Institute of Chemical Technology, Hyderabad 500 007, India
Ecotoxicol Environ Saf 59:332-9. 2004..The decrease in liver AcP may be due to the necrosis of cellular tissues. The changes observed in these enzyme activities could be useful as biomarkers of exposure to Vepacide...
Long-term effects of a novel phosphorothionate (RPR-II) on detoxifying enzymes in brain, lung, and kidney ratsM Mahboob
Toxicology Unit, Biology Division, Indian Institute of Chemical Technology, Tarnaka, Hyderabad, India
Ecotoxicol Environ Saf 53:355-60. 2002..The present subacute study suggests that RPR-II may bring about physiological upsets by altering GSH- and GST-dependent events in different tissues of exposed organisms...
LDH profiles of male and female rats treated with VepacideM F Rahman
Biochemical Toxicology, Biology Division, Indian Institute of Chemical Technology, Hyderabad 500 007, India
Phytother Res 16:122-6. 2002..Necrosis of the liver and kidney tissues was observed but in the lung tissue an increase in the LDH enzyme was seen. Therefore, it was concluded that the increase in LDH could be indicative of a stress adaptive response to the toxicant...
