Research Topics
| Agnes KittelSummaryAffiliation: Institute of Experimental Medicine Country: Hungary Publications
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Detail Information
Publications
Localization of nucleoside triphosphate diphosphohydrolase-1 (NTPDase1) and NTPDase2 in pancreas and salivary glandAgnes Kittel
Department of Pharmacology, Institute of Experimental Medicine, Hungarian Academy of Sciences, PO Box 67, 1450 Budapest, Hungary
J Histochem Cytochem 52:861-71. 2004..Because NTPDase1 is expressed in non-vascular cell types, this finding suggests that NTPDase1 may have functions in the gastrointestinal tract that differ from those demonstrated in the vascular system...- Expression of NTPDase1 and caveolins in human cardiovascular diseaseAgnes Kittel
Department of Pharmacology, Hungarian Academy of Sciences, Institute of Experimental Medicine, P O BOX 67, 1450 Budapest, Hungary
Histochem Cell Biol 124:51-9. 2005..Changes in the expression of NTPDase1 and caveolins seem to be independent of human cardiovascular disease... - CD39 as a caveolar-associated ectonucleotidaseA Kittel
Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest, 1450, Hungary
Biochem Biophys Res Commun 262:596-9. 1999..g., palmitoylation) we hypothesize the same to be the case for CD39. Furthermore, its presence in the caveolae supports its participation in signalling events... - Localization of NTPDase1/CD39 in normal and transformed human pancreasAgnes Kittel
Department of Pathophysiology, Laboratory of Gastrointestinal Research, Institute of Experimental Medicine, Hungarian Academy of Sciences, PO Box 67, 1450 Budapest, Hungary
J Histochem Cytochem 50:549-56. 2002..We hypothesize that the significantly diminished activity of NTPDase1 in the tissues surrounding the ducts may be associated with the processes that lead to tumor formation in human pancreas... - Co-localization of P2Y1 receptor and NTPDase1/CD39 within caveolae in human placentaA Kittel
Department of Pharmacology, Institute of Experimental Medicine, Hungarian Academy of Sciences, P O BOX 67, Budapest, Hungary
Eur J Histochem 48:253-9. 2004..The differential localization of these P2 receptors might indicate their unique roles in the regulation of extracellular nucleotide concentrations in human placental tissues and consequent effects on vascular tone and blood fluidity... - Transient changes in the localization and activity of ecto-nucleotidases in rat hippocampus following lipopolysaccharide treatmentAgnes Kittel
Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest, Hungary
Int J Dev Neurosci 25:275-82. 2007.... - P2Y receptor mediated inhibitory modulation of noradrenaline release in response to electrical field stimulation and ischemic conditions in superfused rat hippocampus slicesCecilia Csölle
Laboratory of Molecular Pharmacology, Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest, Hungary
J Neurochem 106:347-60. 2008..Furthermore, endogenous activation of both facilitatory and inhibitory P2 receptors may participate in the modulation of pathological NA release under ischemic-like conditions... - ATP and ATPase secretion by exocrine pancreas in rat, guinea pig, and humanKrisztina Sz Kordás
Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest, Hungary
Pancreas 29:53-60. 2004..These differences in ATP levels and ATPase expression may indicate significant species differences in the purinergic regulation of pancreatic secretion... - Endotoxin can decrease isolated rat parotid acinar cell amylase secretion in a nitric oxide-independent mannerAdrienn Barta
Department of Pediatrics and Orthodontics, Faculty of Dentistry, Semmelweis University, Budapest, Hungary
Eur J Pharmacol 524:169-73. 2005..Thus, basal, acetylcholine-evoked or endotoxin-decreased cellular amylase secretion from rat isolated parotid acinar cells does not appear to be modulated by endogenous nitric oxide... - Modulation of neurotransmitter release by P2X and P2Y receptors in the rat spinal cordAttila Heinrich
Laboratory of Molecular Pharmacology, Institute of Experimental Medicine, Hungarian Academy of Sciences, H 1450 Budapest POB 67, Hungary
Neuropharmacology 54:375-86. 2008.... - Lack of neuroprotection in the absence of P2X7 receptors in toxin-induced animal models of Parkinson's diseaseZsuzsanna Hracsko
Laboratory of Molecular Pharmacology, Institute of Experimental Medicine, Hungarian Academy of Sciences, H 1083 Budapest, Szigony u, 43, Hungary
Mol Neurodegener 6:28. 2011..abstract:.. - Presynaptic α(2)-adrenoceptors control the inhibitory action of presynaptic CB(1) cannabinoid receptors on prefrontocortical norepinephrine release in the ratHardy Richter
Laboratory of Molecular Pharmacology, Institute of Experimental Medicine, Hungarian Academy of Sciences IEM HAS, H 1083 Budapest, Szigony u 43, Hungary Department of Pharmacology and Pharmacotherapy, Semmelweis University, Budapest, Hungary
Neuropharmacology 63:784-97. 2012..Furthermore, idazoxan triggered a decrease in CB(1)R density in the PFC, suggesting that high extracellular level of norepinephrine downregulates CB(1)Rs... - Increased sensitivity of striatal dopamine release to H2O2 upon chronic rotenone treatmentElisaveta Milusheva
Department of Pharmacology, Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest
Free Radic Biol Med 39:133-42. 2005.... - Modulation of dopaminergic neurotransmission in rat striatum upon in vitro and in vivo diclofenac treatmentElisaveta Milusheva
Institute of Neurobiology former Institute of Physiology, Bulgarian Academy of Sciences, Sofia, Bulgaria
J Neurochem 105:360-8. 2008..In conclusion, whereas in vitro DCF pre-treatment resembles the effect of the mitochondrial toxin rotenone, in vivo it rather counteracts than aggravates dopaminergic dysfunction...