Ulrich O Wenzel

Summary

Affiliation: University Medical Center Hamburg-Eppendorf
Country: Germany

Publications

  1. ncbi request reprint Deficiency of the interleukin 17/23 axis accelerates renal injury in mice with deoxycorticosterone acetate+angiotensin ii-induced hypertension
    Christian F Krebs
    III Medizinische Klinik, University Hospital Hamburg Eppendorf, Martinistr 52, 20246 Hamburg, Germany
    Hypertension 63:565-71. 2014
  2. doi request reprint Treatment of arterial hypertension in obese patients
    Ulrich O Wenzel
    III Medizinische Klinik, Universitatsklinikum Hamburg Eppendorf, Hamburg, Germany
    Semin Nephrol 33:66-74. 2013
  3. ncbi request reprint Chronic anti-Thy-1 nephritis is aggravated in the nonclipped but not in the clipped kidney of Goldblatt hypertensive rats
    Ulrich O Wenzel
    Division of Nephrology, Department of Medicine, University Hospital Hamburg Eppendorf, Hamburg, Germany
    Kidney Int 61:2119-31. 2002
  4. doi request reprint Angiotensin II type 2 receptor deficiency aggravates renal injury and reduces survival in chronic kidney disease in mice
    Ralf A Benndorf
    Clinical Pharmacology Unit, Institute of Experimental and Clinical Toxicology and Pharmacology, University Hospital Hamburg Eppendorf, Hamburg, Germany
    Kidney Int 75:1039-49. 2009
  5. doi request reprint CCR5 deficiency does not reduce hypertensive end-organ damage in mice
    Christian Krebs
    Department of Medicine, Division of Nephrology, University Hospital Hamburg Eppendorf, Germany
    Am J Hypertens 25:479-86. 2012
  6. doi request reprint Rho kinase inhibition attenuates LPS-induced renal failure in mice in part by attenuation of NF-kappaB p65 signaling
    Catherine Meyer-Schwesinger
    Department of Internal Medicine, University Hospital Hamburg, Hamburg, Germany
    Am J Physiol Renal Physiol 296:F1088-99. 2009
  7. ncbi request reprint Management of arterial hypertension in obese patients
    Ulrich O Wenzel
    III Medizinische Klinik, Universitatsklinikum Hamburg Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany
    Curr Hypertens Rep 9:491-7. 2007
  8. ncbi request reprint Increased expression of (pro)renin receptor does not cause hypertension or cardiac and renal fibrosis in mice
    Alva Rosendahl
    Division of Nephrology, University Hospital Hamburg Eppendorf, Hamburg, Germany
    Lab Invest 94:863-72. 2014
  9. ncbi request reprint Treatment of arterial hypertension in obese patients
    Ulrich O Wenzel
    III Medizinische Klinik, Universitatsklinikum Hamburg Eppendorf, Hamburg, Germany
    Contrib Nephrol 151:230-42. 2006
  10. ncbi request reprint Angiotensin II infusion ameliorates the early phase of a mesangioproliferative glomerulonephritis
    Ulrich O Wenzel
    Division of Nephrology, Department of Medicine, University of Hamburg, Martinistrasse 52, 20246 Hamburg, Germany
    Kidney Int 61:1020-9. 2002

Collaborators

  • Rolf A K Stahl
  • Gunter Wolf
  • Ralf A Benndorf
  • Ulf Panzer
  • Genevieve Nguyen
  • Edzard Schwedhelm
  • Oliver M Steinmetz
  • Alva Rosendahl
  • Christian Krebs
  • Catherine Meyer-Schwesinger
  • Gianina Niemann
  • Christian F Krebs
  • Sascha Lange
  • Joachim Velden
  • Aurelie Contrepas
  • Heimo Ehmke
  • Harry van Goor
  • Michael Schwake
  • Hans Joachim Paust
  • Erfan Ahadzadeh
  • Michael Bader
  • Judith Peters
  • Thorsten Wiech
  • Alexander Lehners
  • Andrea Tannapfel
  • Michael N Quang
  • Christoph Fraune
  • Robin Schmidt-Haupt
  • Jan Eric Turner
  • Silke Dehde
  • Claudia von Ruffer
  • Friedrich Thaiss
  • Stefan Gatzemeier
  • Tobias N Meyer
  • Philipp Klug

Detail Information

Publications10

  1. ncbi request reprint Deficiency of the interleukin 17/23 axis accelerates renal injury in mice with deoxycorticosterone acetate+angiotensin ii-induced hypertension
    Christian F Krebs
    III Medizinische Klinik, University Hospital Hamburg Eppendorf, Martinistr 52, 20246 Hamburg, Germany
    Hypertension 63:565-71. 2014
    ..IL-17/IL-23 deficiency accelerates DOCA+Ang II-induced albuminuria and hypertensive renal but not cardiac end-organ damage. ..
  2. doi request reprint Treatment of arterial hypertension in obese patients
    Ulrich O Wenzel
    III Medizinische Klinik, Universitatsklinikum Hamburg Eppendorf, Hamburg, Germany
    Semin Nephrol 33:66-74. 2013
    ..Therefore, treatment with a blocker of the renin-angiotensin-aldosterone-system and a thiazide diuretic should be considered as first-line antihypertensive drug therapy in obesity hypertension...
  3. ncbi request reprint Chronic anti-Thy-1 nephritis is aggravated in the nonclipped but not in the clipped kidney of Goldblatt hypertensive rats
    Ulrich O Wenzel
    Division of Nephrology, Department of Medicine, University Hospital Hamburg Eppendorf, Hamburg, Germany
    Kidney Int 61:2119-31. 2002
    ..To test whether a chronic model of the Thy-1 nephritis is more susceptible to high blood pressure, the repetitive hit model was evaluated in rats with 2-kidney, 1-clip Goldblatt hypertension...
  4. doi request reprint Angiotensin II type 2 receptor deficiency aggravates renal injury and reduces survival in chronic kidney disease in mice
    Ralf A Benndorf
    Clinical Pharmacology Unit, Institute of Experimental and Clinical Toxicology and Pharmacology, University Hospital Hamburg Eppendorf, Hamburg, Germany
    Kidney Int 75:1039-49. 2009
    ..Whether activation of the AT2 receptor will have therapeutic benefit in chronic kidney disease will require further study...
  5. doi request reprint CCR5 deficiency does not reduce hypertensive end-organ damage in mice
    Christian Krebs
    Department of Medicine, Division of Nephrology, University Hospital Hamburg Eppendorf, Germany
    Am J Hypertens 25:479-86. 2012
    ..CCR5 is expressed on infiltrating T cells in hypertensive mice and CCR5 antagonists reduce hypertension making CCR5 an interesting target in treatment of hypertension...
  6. doi request reprint Rho kinase inhibition attenuates LPS-induced renal failure in mice in part by attenuation of NF-kappaB p65 signaling
    Catherine Meyer-Schwesinger
    Department of Internal Medicine, University Hospital Hamburg, Hamburg, Germany
    Am J Physiol Renal Physiol 296:F1088-99. 2009
    ..Together, these data suggest that Rho kinase inhibition by preventive or therapeutic treatment effectively reduced endotoxic kidney injury in part by attenuation of NF-kappaB p65 activation...
  7. ncbi request reprint Management of arterial hypertension in obese patients
    Ulrich O Wenzel
    III Medizinische Klinik, Universitatsklinikum Hamburg Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany
    Curr Hypertens Rep 9:491-7. 2007
    ..Therefore, an ACE inhibitor or diuretic should be considered as first-line antihypertensive drug therapy in obesity-hypertension...
  8. ncbi request reprint Increased expression of (pro)renin receptor does not cause hypertension or cardiac and renal fibrosis in mice
    Alva Rosendahl
    Division of Nephrology, University Hospital Hamburg Eppendorf, Hamburg, Germany
    Lab Invest 94:863-72. 2014
    ..Renin expression increased eightfold in the kidney but no renal injury could be detected. In conclusion, our results suggest no major role for PRR in organ damage per se or related to its function as a receptor of renin. ..
  9. ncbi request reprint Treatment of arterial hypertension in obese patients
    Ulrich O Wenzel
    III Medizinische Klinik, Universitatsklinikum Hamburg Eppendorf, Hamburg, Germany
    Contrib Nephrol 151:230-42. 2006
    ..Sodium retention plays a central role in the development of obesity-related hypertension. Therefore, treatment with an ACE-inhibitors or a diuretic should be considered as first-line antihypertensive drug therapy in obesity-hypertension...
  10. ncbi request reprint Angiotensin II infusion ameliorates the early phase of a mesangioproliferative glomerulonephritis
    Ulrich O Wenzel
    Division of Nephrology, Department of Medicine, University of Hamburg, Martinistrasse 52, 20246 Hamburg, Germany
    Kidney Int 61:1020-9. 2002
    ..Inhibition of the renin-angiotensin system slows the progression of chronic renal disease...