Joerg Wenzel

Summary

Affiliation: University of Bonn
Country: Germany

Publications

  1. ncbi Treatment of recalcitrant dermatomyositis with efalizumab
    Andrea Huber
    Acta Derm Venereol 86:254-5. 2006
  2. doi Pathogenesis of cutaneous lupus erythematosus: common and different features in distinct subsets
    J Wenzel
    Department of Dermatology, University of Bonn, Germany
    Lupus 19:1020-8. 2010
  3. ncbi Role of the chemokine receptor CCR4 and its ligand thymus- and activation-regulated chemokine/CCL17 for lymphocyte recruitment in cutaneous lupus erythematosus
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Bonn, Germany
    J Invest Dermatol 124:1241-8. 2005
  4. ncbi Efficacy and safety of methotrexate in recalcitrant cutaneous lupus erythematosus: results of a retrospective study in 43 patients
    J Wenzel
    Department of Dermatology, University of Bonn, Sigmund Freud Strasse 25, 53105 Bonn, Germany
    Br J Dermatol 153:157-62. 2005
  5. ncbi The role of cytotoxic skin-homing CD8+ lymphocytes in cutaneous cytotoxic T-cell lymphoma and pityriasis lichenoides
    Joerg Wenzel
    Department of Dermatology, Institute of Pathology, University of Bonn, Bonn, Germany
    J Am Acad Dermatol 53:422-7. 2005
  6. ncbi Scarring skin lesions of discoid lupus erythematosus are characterized by high numbers of skin-homing cytotoxic lymphocytes associated with strong expression of the type I interferon-induced protein MxA
    J Wenzel
    Department of Dermatology, University of Bonn, Sigmund Freud Strasse 25, 53105 Bonn, Germany
    Br J Dermatol 153:1011-5. 2005
  7. ncbi [Systemic treatment of cutaneous lupus erythematosus]
    Joerg Wenzel
    Klinik und Poliklinik fur Dermatologie, Universitatsklinikum Bonn
    J Dtsch Dermatol Ges 1:694-704. 2003
  8. ncbi [Topical treatment of bullous pemphigoid with tacrolimus. Case report with brief literature review]
    Joerg Wenzel
    Klinik und Poliklinik fur Dermatologie, Universitatsklinikum Bonn
    J Dtsch Dermatol Ges 3:207-10. 2005
  9. ncbi Type I interferon-associated skin recruitment of CXCR3+ lymphocytes in dermatomyositis
    J Wenzel
    Department of Dermatology, University of Bonn, Germany
    Clin Exp Dermatol 31:576-82. 2006
  10. ncbi Type I interferon-associated cytotoxic inflammation in lichen planus
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Bonn, Germany
    J Cutan Pathol 33:672-8. 2006

Collaborators

Detail Information

Publications60

  1. ncbi Treatment of recalcitrant dermatomyositis with efalizumab
    Andrea Huber
    Acta Derm Venereol 86:254-5. 2006
  2. doi Pathogenesis of cutaneous lupus erythematosus: common and different features in distinct subsets
    J Wenzel
    Department of Dermatology, University of Bonn, Germany
    Lupus 19:1020-8. 2010
    ..Here we review the main pathological features described in CLE patients against the background of the clinical diversity of different CLE subtypes...
  3. ncbi Role of the chemokine receptor CCR4 and its ligand thymus- and activation-regulated chemokine/CCL17 for lymphocyte recruitment in cutaneous lupus erythematosus
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Bonn, Germany
    J Invest Dermatol 124:1241-8. 2005
    ..Our investigations suggest that CCR4 and TARC/CCL17 play a role in the pathophysiology of CLE. In particular, cytotoxic CD8+ T cells expressing CCR4 appear to be involved in scarring subtypes of CLE...
  4. ncbi Efficacy and safety of methotrexate in recalcitrant cutaneous lupus erythematosus: results of a retrospective study in 43 patients
    J Wenzel
    Department of Dermatology, University of Bonn, Sigmund Freud Strasse 25, 53105 Bonn, Germany
    Br J Dermatol 153:157-62. 2005
    ..In recent years several other immunosuppressive agents have been successfully applied in CLE. However, there are no large studies or explicit guidelines on the use of these drugs in CLE...
  5. ncbi The role of cytotoxic skin-homing CD8+ lymphocytes in cutaneous cytotoxic T-cell lymphoma and pityriasis lichenoides
    Joerg Wenzel
    Department of Dermatology, Institute of Pathology, University of Bonn, Bonn, Germany
    J Am Acad Dermatol 53:422-7. 2005
    ..This case prompted us to reinvestigate the role of cytotoxic T lymphocytes in PL and its relationship to antiviral immunity...
  6. ncbi Scarring skin lesions of discoid lupus erythematosus are characterized by high numbers of skin-homing cytotoxic lymphocytes associated with strong expression of the type I interferon-induced protein MxA
    J Wenzel
    Department of Dermatology, University of Bonn, Sigmund Freud Strasse 25, 53105 Bonn, Germany
    Br J Dermatol 153:1011-5. 2005
    ..Because recent investigations have highlighted the relevance of cytotoxic lymphocytes in autoimmune tissue destruction, we hypothesized that the scarring CDLE lesions might be caused by cytotoxic T lymphocytes...
  7. ncbi [Systemic treatment of cutaneous lupus erythematosus]
    Joerg Wenzel
    Klinik und Poliklinik fur Dermatologie, Universitatsklinikum Bonn
    J Dtsch Dermatol Ges 1:694-704. 2003
    ....
  8. ncbi [Topical treatment of bullous pemphigoid with tacrolimus. Case report with brief literature review]
    Joerg Wenzel
    Klinik und Poliklinik fur Dermatologie, Universitatsklinikum Bonn
    J Dtsch Dermatol Ges 3:207-10. 2005
    ..He was treated with 0.1% tacrolimus ointment, which was able to control the disease. We briefly review the literature and discuss the potential role of tacrolimus as a novel option for the topical treatment of BP...
  9. ncbi Type I interferon-associated skin recruitment of CXCR3+ lymphocytes in dermatomyositis
    J Wenzel
    Department of Dermatology, University of Bonn, Germany
    Clin Exp Dermatol 31:576-82. 2006
    ..We hypothesized that a similar mechanism might also be involved in the pathogenesis of DM skin lesions...
  10. ncbi Type I interferon-associated cytotoxic inflammation in lichen planus
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Bonn, Germany
    J Cutan Pathol 33:672-8. 2006
    ..Here, we investigated whether type I IFNs are also involved in the pathogenesis of LP...
  11. ncbi IP10/CXCL10 - CXCR3 interaction: a potential self-recruiting mechanism for cytotoxic lymphocytes in lichen sclerosus et atrophicus
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Germany
    Acta Derm Venereol 87:112-7. 2007
    ..Infiltrating lymphocytes, containing IP10 in their granules, could provide an important self-perpetuating mechanism...
  12. ncbi CXCR3-mediated recruitment of cytotoxic lymphocytes in lupus erythematosus profundus
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Germany
    J Am Acad Dermatol 56:648-50. 2007
    ..Recently, we observed a case of LEP involving the lower right eyelid. Our immunohistological analyses of lesional skin biopsies revealed a type I IFN signature in the context of cytotoxic lobular panniculitis...
  13. ncbi Identification of type I interferon-associated inflammation in the pathogenesis of cutaneous lupus erythematosus opens up options for novel therapeutic approaches
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Sigmund Freud Strasse 25, 53105 Bonn, Germany
    Exp Dermatol 16:454-63. 2007
    ..Here, we review the role of type I IFNs, adhesion molecules and chemokine receptors in CLE and discuss options for novel therapeutic approaches...
  14. ncbi Gene expression profiling of lichen planus reflects CXCL9+-mediated inflammation and distinguishes this disease from atopic dermatitis and psoriasis
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Bonn, Germany
    J Invest Dermatol 128:67-78. 2008
    ..Gene expression profiling might complement the diagnostic spectrum in dermatology and may provide new pathogenetic insights...
  15. ncbi The expression pattern of interferon-inducible proteins reflects the characteristic histological distribution of infiltrating immune cells in different cutaneous lupus erythematosus subsets
    J Wenzel
    Department of Dermatology, University of Bonn, Sigmund Freud Strasse 25, 53105 Bonn, Germany
    Br J Dermatol 157:752-7. 2007
    ..We hypothesized that the expression pattern of IFN-inducible proteins reflects the characteristic distribution of the inflammatory infiltrate in different subsets of cutaneous LE...
  16. doi CXCR3 <-> ligand-mediated skin inflammation in cutaneous lichenoid graft-versus-host disease
    Joerg Wenzel
    Department of Dermatology at the Universitie of Bonn, Bonn, Germany
    J Am Acad Dermatol 58:437-42. 2008
    ....
  17. doi An IFN-associated cytotoxic cellular immune response against viral, self-, or tumor antigens is a common pathogenetic feature in "interface dermatitis"
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Bonn, Germany
    J Invest Dermatol 128:2392-402. 2008
    ....
  18. doi Transcriptional profiling identifies an interferon-associated host immune response in invasive squamous cell carcinoma of the skin
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Bonn, Germany
    Int J Cancer 123:2605-15. 2008
    ..Collectively, our findings support the concept that IFN-associated host responses play an important role in tumor immunosurveillance in the skin...
  19. doi Type I interferon-associated cytotoxic inflammation in cutaneous lupus erythematosus
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Sigmund Freud Strasse 25, 53105 Bonn, Germany
    Arch Dermatol Res 301:83-6. 2009
    ..These and other studies suggest that a complex network of IFN-associated cytokines, chemokines and adhesion molecules orchestrates and promotes tissue injury observed in LE skin...
  20. ncbi The expression of human leukocyte antigen-DR and CD25 on circulating T cells in cutaneous lupus erythematosus and correlation with disease activity
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Bonn, Germany
    Exp Dermatol 14:454-9. 2005
    ..In patients with SLE, the expression of activation markers on circulating T cells reflects disease activity. Here, we investigated whether this also holds true for patients with CLE...
  21. ncbi The value of lymphocytopenia as a marker of systemic involvement in cutaneous lupus erythematosus
    J Wenzel
    Department of Dermatology, University of Bonn, Sigmund Freud Strasse 25, 53105 Bonn, Germany
    Br J Dermatol 146:869-71. 2002
    ..Recent studies revealed some markers indicating systemic manifestations of CLE patients. However, the significance of diminished peripheral lymphocyte numbers as a marker of systemic involvement in CLE has not been investigated before...
  22. ncbi Infraorbital eyelid edema as the presenting sign of bronchogenic carcinoma
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Sigmund Freud Strasse 25, 53105 Bonn, Germany
    Int J Dermatol 41:386-7. 2002
  23. ncbi Flame figures in urticarial lesions accompanying systemic lupus erythematosus
    J Wenzel
    Department of Dermatology, University of Bonn, Germany
    Am J Dermatopathol 23:533-5. 2001
    ..Hematoxylin-eosin-stained sections revealed flame figures. We review urticarial lesions in lupus erythematosus and discuss the possible pathomechanisms...
  24. ncbi Topical treatment of pyoderma gangraenosum
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Germany
    Dermatology 205:221-3. 2002
    ..We give a review about these topically used drugs and the mechanisms probably involved...
  25. ncbi Bullous lupus erythematosus in a patient with pre-existing dermatomyositis
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Sigmund Freud Strasse 25, 53105, Bonn, Germany
    Rheumatol Int 24:114-6. 2004
    ..Observations of transitions between DM and LE are extremely rare. Nevertheless, this case demonstrates the close relationship between these two disorders in the context of autoimmune connective tissue disease...
  26. ncbi [Cutaneous lesions in lupus erythematosus]
    J Wenzel
    Klinik und Poliklinik fur Dermatologie, Universitatsklinikum Bonn
    Dtsch Med Wochenschr 128:1655-8. 2003
  27. ncbi Association of inclusion body myositis with subacute cutaneous lupus erythematosus
    J Wenzel
    Department of Dermatology, University of Bonn, Germany
    Rheumatol Int 21:75-7. 2001
    ..The presented case is discussed in the context of earlier reports of inclusion body myositis and lupus erythematosus...
  28. ncbi Successful treatment of Dowling-Degos disease with Er:YAG laser
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Bonn, Germany
    Dermatol Surg 28:748-50. 2002
    ..The typical clinical picture is a reticulate pigmentation of the flexures, genital folds, and pectoral skin areas. Many different treatment options have been tried in recent years without a convincing therapeutic benefit...
  29. ncbi Enhanced type I interferon signaling and recruitment of chemokine receptor CXCR3-expressing lymphocytes into the skin following treatment with the TLR7-agonist imiquimod
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Bonn, Germany
    J Cutan Pathol 32:257-62. 2005
    ..We hypothesize that TLR7-signaling strongly induces the production of interferon (IFN) alpha, which is able to enhance Th1-mediated cellular antiviral and antitumor immunity...
  30. ncbi Enhanced type I interferon signalling promotes Th1-biased inflammation in cutaneous lupus erythematosus
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Germany
    J Pathol 205:435-42. 2005
    ..It is proposed that type I IFNs induce a Th1-biased inflammatory immune response, with recruitment of CXCR3-expressing T-lymphocytes into the skin...
  31. ncbi Type I interferon-associated recruitment of cytotoxic lymphocytes: a common mechanism in regressive melanocytic lesions
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Bonn, Germany
    Am J Clin Pathol 124:37-48. 2005
    ..We believe this cytotoxic immune response represents an evolutionarily conserved pathway against intracellular pathogens...
  32. pmc Indoleamine 2,3-dioxygenase (IDO): the antagonist of type I interferon-driven skin inflammation?
    Marina Scheler
    Department of Dermatology, Friedrich Wilhelms University, Sigmund Freud Strasse 25, 53105 Bonn, Germany
    Am J Pathol 171:1936-43. 2007
    ..These findings reveal that both proinflammatory and counterregulatory mechanisms are operative in cutaneous lesions of LP. We propose that the balance of these mechanisms may be involved in the pathogenesis of this disorder...
  33. doi Real-time tissue elastography as promising diagnostic tool for diagnosis of lymph node metastases in patients with malignant melanoma: a prospective single-center experience
    Torsten Hinz
    Department of Dermatology and Allergology, Center of Integrated Oncology CIO Cologne Bonn, University of Bonn, Bonn, Germany
    Dermatology 226:81-90. 2013
    ....
  34. doi Role of high-resolution ultrasound and PET/CT imaging for preoperative characterization of sentinel lymph nodes in cutaneous melanoma
    Torsten Hinz
    Department of Dermatology and Allergy, University of Bonn, Bonn, Germany
    Ultrasound Med Biol 39:30-6. 2013
    ....
  35. doi Sentinel lymph node status as most important prognostic factor in patients with high-risk cutaneous melanomas (tumour thickness >4.00 mm): outcome analysis from a single institution
    Torsten Hinz
    Department of Dermatology and Allergology, Center of Integrated Oncology Cologne Bonn University of Bonn, Bonn, Germany
    Eur J Nucl Med Mol Imaging 39:1316-25. 2012
    ..The purpose of the current study was to determine the prognostic value of SLNB in patients with thick cutaneous melanomas (tumour thickness >4.0 mm) in terms of progression-free survival (PFS) and overall survival (OS)...
  36. ncbi Ultraviolet light protection by a sunscreen prevents interferon-driven skin inflammation in cutaneous lupus erythematosus
    Sabine Zahn
    Department of Dermatology, University of Bonn, Bonn, Germany
    Exp Dermatol 23:516-8. 2014
    ..The data of this study demonstrate that UV protection reduces lesional tissue damage and inhibits the typical IFN-driven inflammatory response in CLE. ..
  37. ncbi Indoleamine 2,3-dioxygenase-expressing antigen-presenting cells and peripheral T-cell tolerance: another piece to the atopic puzzle?
    Dagmar von Bubnoff
    Department of Dermatology, Friedrich Wilhelms University, Bonn, Germany
    J Allergy Clin Immunol 112:854-60. 2003
    ..We introduce the hypothesis that tryptophan degradation might add to the ability to control and downregulate allergen-specific T-cell responses in atopic individuals...
  38. ncbi Generalized lichen nitidus with involvement of the palms following interferon alpha treatment
    Marina Scheler
    Department of Dermatology, University of Bonn, Bonn, Germany
    Dermatology 215:236-9. 2007
    ..Furthermore in our patient we could show a strong lesional expression of MxA, a protein specifically induced by type I interferon. It is tempting to speculate that interferon alpha may be involved in the pathogenesis of lichen nitidus...
  39. doi High-resolution ultrasound combined with power Doppler sonography can reduce the number of sentinel lymph node biopsies in cutaneous melanoma
    Torsten Hinz
    Department of Dermatology and Allergology, Center of Integrated Oncology Cologne Bonn, University of Bonn, Bonn, Germany
    Dermatology 222:180-8. 2011
    ..Disadvantages of the SLNB procedure are the overall costs and potential side effects of the operation [J Dtsch Dermatol Ges 2009;7:318-327; J Am Dermatol 2010;62:737-748]...
  40. ncbi Tryptase detection in bone-marrow blood: a new diagnostic tool in systemic mastocytosis
    Julia Proelss
    Department of Dermatology, University of Bonn, Bonn, Germany
    J Am Acad Dermatol 56:453-7. 2007
    ..The diagnosis of systemic mastocytosis is substantially based on the histologic examination of bone-marrow biopsy specimens...
  41. ncbi Kaposi's sarcoma of the gastrointestinal tract: report of two cases and review of the literature
    Philip Kahl
    Department of Pathology, University of Bonn Medical School, Sigmund Freud Street 25, 53127 Bonn, Germany
    Pathol Res Pract 203:227-31. 2007
    ..Nevertheless, Kaposi's sarcoma must always be considered in lesions of the gastrointestinal tract or in gastrointestinal bleeding and should lead to further elucidation of the causes...
  42. ncbi Successful treatment of acrodermatitis continua suppurativa with topical tacrolimus 0.1% ointment
    D Wilsmann-Theis
    Department of Dermatology, University of Bonn, 53105, Germany
    Br J Dermatol 150:1194-7. 2004
    ..1% ointment. Our observations raise hopes that this new treatment strategy for ACS may constitute a novel effective therapeutic option for this recalcitrant condition...
  43. ncbi [Multiple skin cancers in a patient treated with hydroxyurea]
    A Wiechert
    Dermatologische Klinik, Universitatsklinikum Bonn, Sigmund Freud Strasse 25, 53105 Bonn
    Hautarzt 60:651-2, 654. 2009
    ..An association with multiple skin tumors has been reported. The presented case emphasizes this potential risk of hydroxyurea therapy. Continuous dermatologic monitoring of patients treated with hydroxyurea is recommended...
  44. doi New reasons for histopathological nail-clipping examination in the diagnosis of onychomycosis
    D Wilsmann-Theis
    Department of Dermatology and Allergy, University of Bonn, Bonn, Germany
    J Eur Acad Dermatol Venereol 25:235-7. 2011
    ..In addition, in cases of controlling the antifungal treatment course, the present gold standard is often not a reliable tool because of the inhibition of the fungal growth in the culture...
  45. ncbi Periodic genital pruritus caused by syringoma of the vulva
    Rainer Gerdsen
    Department of Dermatology, University of Bonn, Germany, Bonn, Germany
    Acta Obstet Gynecol Scand 81:369-70. 2002
  46. ncbi Scleroderma and malignancy. Mechanisms of interrelationship
    Joerg Wenzel
    Department of Dermatology, University of Bonn, Sigmund Freud Strasse 25, 53105 Bonn, Germany joerg wenzel ukb uni bonn de
    Eur J Dermatol 12:296-300. 2002
    ..This paper reviews the mechanisms described in earlier studies and discusses their possible pathogenetic meaning for the interrelationship and the pathogenesis of scleroderma and neoplasia...
  47. doi Efficacy of ablative laser treatment in Galli-Galli disease
    Harald Voth
    Department of Dermatology and Allergy, University of Bonn, Germany
    Arch Dermatol 147:317-20. 2011
    ..To date, no convincing treatment options are known for GGD. We sought to examine the therapeutic potential of ablative laser treatment in a patient with genetically confirmed GGD...
  48. doi Low-dose methotrexate - a therapeutical kick in TNF-alpha antagonist treatment for recalcitrant psoriasis vulgaris
    Dagmar Wilsmann-Theis
    Department of Dermatology and Allergy, University of Bonn, Bonn, Germany
    Dermatol Ther 27:55-9. 2014
    ....
  49. ncbi Circulating clonal CLA(+) and CD4(+) T cells in Sezary syndrome express the skin-homing chemokine receptors CCR4 and CCR10 as well as the lymph node-homing chemokine receptor CCR7
    M Sokolowska-Wojdylo
    Laboratory of Experimental Dermatology, Department of Dermatology, Rheinische Friedrich Wilhelm University, Sigmund Freud Str 25, 53105 Bonn, Germany
    Br J Dermatol 152:258-64. 2005
    ..It has recently been reported that the chemokine CCL27 expressed by keratinocytes attracts lymphocytes bearing the chemokine receptor CCR10...
  50. ncbi Lymphocytopenia in lupus erythematosus: close in vivo association to autoantibodies targeting nuclear antigens
    J Wenzel
    Department of Dermatology, University of Bonn, Bonn, Germany
    Br J Dermatol 150:994-8. 2004
    ..Antilymphocyte antibodies are regarded as the most likely rationale for the decreased cell numbers; however, their relevance has not been confirmed so far...
  51. doi Suppression of UV-induced damage by a liposomal sunscreen: a prospective, open-label study in patients with cutaneous lupus erythematosus and healthy controls
    Nikolaos Patsinakidis
    Department of Dermatology, University of Muenster, Muenster, Germany
    Exp Dermatol 21:958-61. 2012
    ..Histological analysis of skin biopsy specimens confirmed the clinical results. In conclusion, the use of a high-protection, broad-spectrum sunscreen can prevent UV-induced damage in patients with CLE and HCs...
  52. pmc Loss-of-function mutations in the keratin 5 gene lead to Dowling-Degos disease
    Regina C Betz
    Institute of Human Genetics, University of Bonn, D 53111 Bonn, Germany
    Am J Hum Genet 78:510-9. 2006
    ....
  53. ncbi Resolving lesions in human cutaneous leishmaniasis predominantly harbour chemokine receptor CXCR3-positive T helper 1/T cytotoxic type 1 cells
    B Geiger
    Department of Dermatology, Universitatsmedizin, Johannes Gutenberg University, Langenbeckstr 1, 55131 Mainz, Germany
    Br J Dermatol 162:870-4. 2010
    ..Cutaneous leishmaniasis (CL) is an epidemic disease affecting millions of individuals worldwide. Treatment options have several side-effects and a vaccine does not exist at present...
  54. ncbi Absence of CD26 expression on skin-homing CLA+ CD4+ T lymphocytes in peripheral blood is a highly sensitive marker for early diagnosis and therapeutic monitoring of patients with Sézary syndrome
    M Sokolowska-Wojdylo
    Laboratory of Experimental Dermatology, Department of Dermatology, University of Bonn, Germany
    Clin Exp Dermatol 30:702-6. 2005
    ..We conclude that the absence of CD26 expression on skin-homing CLA+ CD4+ T-helper cells is a very sensitive and highly specific parameter for early diagnosis and therapeutic monitoring of patients with SS...
  55. ncbi Systematic mutation screening of KRT5 supports the hypothesis that Galli-Galli disease is a variant of Dowling-Degos disease
    S Hanneken
    Department of Dermatology, University Hospital Dusseldorf, Moorenstrasse 5, 40225 Dusseldorf, Germany
    Br J Dermatol 163:197-200. 2010
    ..Mutations in the keratin 5 gene (KRT5) have been identified in the majority of patients with DDD and in a small number of patients with GGD...
  56. ncbi Enhanced expression levels of IL-31 correlate with IL-4 and IL-13 in atopic and allergic contact dermatitis
    Mark M Neis
    Department of Dermatology and Allergology, University Hospital RWTH Aachen, Germany
    J Allergy Clin Immunol 118:930-7. 2006
    ..IL-31 is produced by activated T lymphocytes, preferentially by TH2 cells. Transgenic mice overexpressing IL-31 have a phenotype resembling allergic dermatitis in human subjects...
  57. ncbi Successful rituximab treatment of severe pemphigus vulgaris resistant to multiple immunosuppressants
    Joerg Wenzel
    Acta Derm Venereol 85:185-6. 2005
  58. ncbi Successful treatment of chronic discoid lupus erythematosus of the scalp with imiquimod
    Rainer Gerdsen
    Dermatology 205:416-8. 2002
  59. ncbi Successful treatment of bullous congenital ichthyosiform erythroderma with erythromycin
    Katharina ten Freyhaus
    Dermatology 215:81-3. 2007
  60. ncbi Granuloma annulare induced by scabies
    Dagmar Wilsmann-Theis
    Acta Derm Venereol 83:318. 2003