Mirko Voelkers

Summary

Affiliation: University of Heidelberg
Country: Germany

Publications

  1. ncbi High-sensitive Troponin T increase after exercise in patients with pulmonary arterial hypertension
    Mirko Völkers
    Department of Cardiology, Angiology and Respiratory Medicine, University Medical Center, Heidelberg, D 69120, Germany
    BMC Pulm Med 13:28. 2013
  2. ncbi Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes
    Mirko Voelkers
    Department of Internal Medicine III, Laboratory for Molecular and Translational Cardiology, Division of Cardiology, INF 350, University of Heidelberg, 69120 Heidelberg, Germany
    J Mol Cell Cardiol 48:1329-34. 2010
  3. ncbi S100A1: a regulator of striated muscle sarcoplasmic reticulum Ca2+ handling, sarcomeric, and mitochondrial function
    Mirko Völkers
    Division of Cardiology, Department of Internal Medicine III, Laboratory for Molecular and Translational Cardiology, University of Heidelberg, INF 350, 69120 Heidelberg, Germany
    J Biomed Biotechnol 2010:178614. 2010
  4. ncbi S100A1: a multifaceted therapeutic target in cardiovascular disease
    David Rohde
    Laboratory for Molecular and Translational Cardiology, Division of Cardiology, Department of Internal Medicine III, University of Heidelberg, INF 350, 69120 Heidelberg, Germany
    J Cardiovasc Transl Res 3:525-37. 2010
  5. ncbi Endothelin receptor antagonist and airway dysfunction in pulmonary arterial hypertension
    Annette S Droste
    Department of Cardiology, Angiology and Respiratory Medicine, Heidelberg, Germany
    Respir Res 10:129. 2009
  6. ncbi Decreased contractility due to energy deprivation in a transgenic rat model of hypertrophic cardiomyopathy
    Mark Luedde
    Department of Cardiology, University of Heidelberg, Germany
    J Mol Med (Berl) 87:411-22. 2009

Collaborators

Detail Information

Publications6

  1. ncbi High-sensitive Troponin T increase after exercise in patients with pulmonary arterial hypertension
    Mirko Völkers
    Department of Cardiology, Angiology and Respiratory Medicine, University Medical Center, Heidelberg, D 69120, Germany
    BMC Pulm Med 13:28. 2013
    ..The current study aimed to investigate the release of myocardial high-sensitive Troponin T (hsTnT) in patients with pulmonary arterial hypertension (PAH) in response to maximal physical exercise...
  2. ncbi Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes
    Mirko Voelkers
    Department of Internal Medicine III, Laboratory for Molecular and Translational Cardiology, Division of Cardiology, INF 350, University of Heidelberg, 69120 Heidelberg, Germany
    J Mol Cell Cardiol 48:1329-34. 2010
    ..This study shows for the first time that both Orai1 and Stim1 have a key role in cardiomyocyte SOCE regulating both normal and hypertrophic postnatal cardiac growth in vitro...
  3. ncbi S100A1: a regulator of striated muscle sarcoplasmic reticulum Ca2+ handling, sarcomeric, and mitochondrial function
    Mirko Völkers
    Division of Cardiology, Department of Internal Medicine III, Laboratory for Molecular and Translational Cardiology, University of Heidelberg, INF 350, 69120 Heidelberg, Germany
    J Biomed Biotechnol 2010:178614. 2010
    ....
  4. ncbi S100A1: a multifaceted therapeutic target in cardiovascular disease
    David Rohde
    Laboratory for Molecular and Translational Cardiology, Division of Cardiology, Department of Internal Medicine III, University of Heidelberg, INF 350, 69120 Heidelberg, Germany
    J Cardiovasc Transl Res 3:525-37. 2010
    ..In summary, elaborate basic and translational research established S100A1 as a multifaceted therapeutic target in cardiovascular disease, providing a promising novel therapeutic strategy to future cardiologists...
  5. ncbi Endothelin receptor antagonist and airway dysfunction in pulmonary arterial hypertension
    Annette S Droste
    Department of Cardiology, Angiology and Respiratory Medicine, Heidelberg, Germany
    Respir Res 10:129. 2009
    ..Bosentan (ET-1 receptor antagonist) improves pulmonary hemodynamics, exercise limitation, and disease severity in IPAH. We hypothesized that bosentan might affect airway obstruction...
  6. ncbi Decreased contractility due to energy deprivation in a transgenic rat model of hypertrophic cardiomyopathy
    Mark Luedde
    Department of Cardiology, University of Heidelberg, Germany
    J Mol Med (Berl) 87:411-22. 2009
    ..In summary, we demonstrate profound energetic alterations in DEL-TNT hearts, supporting the notion that inefficient cellular ATP utilization contributes to the pathogenesis of HCM...