Research Topics
| Ali SakSummaryAffiliation: University of Essen Country: Germany Publications
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Detail Information
Publications
Selective inactivation of DNA-dependent protein kinase with antisense oligodeoxynucleotides: consequences for the rejoining of radiation-induced DNA double-strand breaks and radiosensitivity of human cancer cell linesAli Sak
Department of Radiotherapy, University Essen, 45122 Essen, Germany
Cancer Res 62:6621-4. 2002..These data show that kinase activity of DNA-PKcs can be specifically inhibited with As-ODN as effective as Wortmannin and results in marked inhibition of DNA-Dsb rejoining and radiosensitization of NSCLC cell lines...
Increased radiation-induced apoptosis and altered cell cycle progression of human lung cancer cell lines by antisense oligodeoxynucleotides targeting p53 and p21(WAF1/CIP1)Ali Sak
Department of Radiotherapy, University Essen, 45122 Essen, Germany
Cancer Gene Ther 10:926-34. 2003....
Targeting of Rad51-dependent homologous recombination: implications for the radiation sensitivity of human lung cancer cell linesA Sak
Department of Radiotherapy, University Hospital Essen, 45122 Essen, Germany
Br J Cancer 92:1089-97. 2005..Although most of the Rad51 foci are co-localised with gamma-H2AX foci, no correlation of the relative fraction of persisting gamma-H2AX foci and SF2 is evident...
Inhibiting the aurora B kinase potently suppresses repopulation during fractionated irradiation of human lung cancer cell linesAli Sak
Department of Radiotherapy, University Hospital Essen, University Duisburg Essen, Essen, Germany
Int J Radiat Oncol Biol Phys 84:492-9. 2012..We assessed the combined effectiveness of inhibiting Aurora B kinase and irradiation on human NSCLC cell lines in vitro...
Long-term in vivo effects of cisplatin on gamma-H2AX foci signaling in peripheral lymphocytes of tumor patients after irradiationAli Sak
Department of Radiotherapy, University Hospital Essen, Essen, Germany
Clin Cancer Res 15:2927-34. 2009..This study determined the effects of cis-diamminedichloroplatinum(II) on radiation-induced foci formation of gamma-H2AX and Rad51 in lymphocytes...
Use of γH2AX and other biomarkers of double-strand breaks during radiotherapyAli Sak
Department of Radiotherapy, University Hospital Essen, Essen, Germany
Semin Radiat Oncol 20:223-31. 2010..Validation by prospective studies is needed...
Effect of separase depletion on ionizing radiation-induced cell cycle checkpoints and survival in human lung cancer cell linesA Sak
Department of Radiotherapy, University Hospital Essen, Essen, Germany
Cell Prolif 41:660-70. 2008..This study is to evaluate the effect of separase depletion on cell cycle progression of irradiated and non-irradiated cells through the G(2)/M phases and consecutive cell survival...
Radiation-induced apoptosis in human non-small-cell lung cancer cell lines is secondary to cell-cycle progression beyond the G2-phase checkpointM Stuschke
Department of Radiotherapy, Humboldt University, Charite, Berlin, Germany
Int J Radiat Biol 78:807-19. 2002..To characterize the relationship between cell-cycle progression and radiation-induced apoptosis in NSCLC cell lines with different p53 status...
gamma-H2AX foci formation in peripheral blood lymphocytes of tumor patients after local radiotherapy to different sites of the body: dependence on the dose-distribution, irradiated site and time from start of treatmentAli Sak
Department of Radiotherapy, University Hospital Essen, Essen, Germany
Int J Radiat Biol 83:639-52. 2007..To evaluate the relationship between an estimated integral total body radiation dose delivered and phosphorylated histone H2AX protein (gamma-H2AX) foci formation in peripheral blood lymphocytes of cancer patients...
Targeting of AKT1 enhances radiation toxicity of human tumor cells by inhibiting DNA-PKcs-dependent DNA double-strand break repairMahmoud Toulany
Division of Radiobiology and Molecular Environmental Research, Department of Radiation Oncology, University of Tuebingen, Eberhard Karls University Tuebingen, Roentgenweg 11, 72076 Tuebingen, Germany
Mol Cancer Ther 7:1772-81. 2008..Thus, targeting of AKT enhances radiation sensitivity of lung cancer cell lines A549 and H460 most likely through specific inhibition of DNA-PKcs-dependent DNA-dsb repair but not through enhancement of radiation-induced apoptosis...
