D Peus

Summary

Affiliation: University of Munich
Country: Germany

Publications

  1. ncbi H2O2 is required for UVB-induced EGF receptor and downstream signaling pathway activation
    D Peus
    Department of Dermatology, Mayo Clinic Foundation, Rochester, MN, USA
    Free Radic Biol Med 27:1197-202. 1999
  2. ncbi Anti-psoriatic drug anthralin activates JNK via lipid peroxidation: mononuclear cells are more sensitive than keratinocytes
    D Peus
    Department of Dermatology, Biochemistry and Molecular Biology and Rheumatology, Mayo Clinic Foundation, Rochester, Minnesota, USA
    J Invest Dermatol 114:688-92. 2000
  3. ncbi UVB-induced epidermal growth factor receptor phosphorylation is critical for downstream signaling and keratinocyte survival
    D Peus
    Department of Dermatology, Mayo Clinic Foundation, Rochester, MN, USA
    Photochem Photobiol 72:135-40. 2000
  4. ncbi H(2)O(2) mediates oxidative stress-induced epidermal growth factor receptor phosphorylation
    A Meves
    Department of Dermatology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Toxicol Lett 122:205-14. 2001

Collaborators

  • A Meves
  • A Beyerle
  • M R Pittelkow
  • S N Stock

Detail Information

Publications4

  1. ncbi H2O2 is required for UVB-induced EGF receptor and downstream signaling pathway activation
    D Peus
    Department of Dermatology, Mayo Clinic Foundation, Rochester, MN, USA
    Free Radic Biol Med 27:1197-202. 1999
    ..Our results identify UVB-induced H2O2 as a second messenger that is required for EGFR and dependent downstream signaling pathways activation...
  2. ncbi Anti-psoriatic drug anthralin activates JNK via lipid peroxidation: mononuclear cells are more sensitive than keratinocytes
    D Peus
    Department of Dermatology, Biochemistry and Molecular Biology and Rheumatology, Mayo Clinic Foundation, Rochester, Minnesota, USA
    J Invest Dermatol 114:688-92. 2000
    ....
  3. ncbi UVB-induced epidermal growth factor receptor phosphorylation is critical for downstream signaling and keratinocyte survival
    D Peus
    Department of Dermatology, Mayo Clinic Foundation, Rochester, MN, USA
    Photochem Photobiol 72:135-40. 2000
    ..Our data suggest that ligand-independent phosphorylation of EGFR and likely dependent downstream signaling pathways regulate cellular defense mechanisms important for cell survival following oxidative stress...
  4. ncbi H(2)O(2) mediates oxidative stress-induced epidermal growth factor receptor phosphorylation
    A Meves
    Department of Dermatology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Toxicol Lett 122:205-14. 2001
    ..We propose that depletion of GSH and accumulation of H(2)O(2) are upstream events and critical mediators required for ligand-independent phosphorylation of growth factor receptors in response to oxidative stress...