G A Enders

Summary

Affiliation: University of Munich
Country: Germany

Publications

  1. pmc Cyclooxygenase-2 overexpression abrogates the antiproliferative effects of TGF-beta
    G A Enders
    Institute for Surgical Research, LMU Munchen, Marchioninistr 27, München D 81366, Germany
    Br J Cancer 97:1388-92. 2007
  2. ncbi request reprint Surgical research and Helicobacter pylori infection -- a contradiction?
    G A Enders
    Institute for Surgical Research, LMU Munich, Klinikum Grosshadern, Munich, Germany
    Eur Surg Res 34:18-21. 2002
  3. ncbi request reprint Role of virulence factors, cell components and adhesion in Helicobacter pylori-mediated iNOS induction in murine macrophages
    I A Assmann
    Department of Surgery, Klinikum Grosshadern, Ludwig-Maximilians-University Munich, Germany
    FEMS Immunol Med Microbiol 30:133-8. 2001

Collaborators

  • R Hatz
  • I A Assmann
  • G Rieder
  • R Haas

Detail Information

Publications3

  1. pmc Cyclooxygenase-2 overexpression abrogates the antiproliferative effects of TGF-beta
    G A Enders
    Institute for Surgical Research, LMU Munchen, Marchioninistr 27, München D 81366, Germany
    Br J Cancer 97:1388-92. 2007
    ..Therefore, inhibiting both COX-2 as well as the PPAR and TGF/EGF pathway could be effective in the inhibition of adenoma or even carcinoma development in the intestine...
  2. ncbi request reprint Surgical research and Helicobacter pylori infection -- a contradiction?
    G A Enders
    Institute for Surgical Research, LMU Munich, Klinikum Grosshadern, Munich, Germany
    Eur Surg Res 34:18-21. 2002
  3. ncbi request reprint Role of virulence factors, cell components and adhesion in Helicobacter pylori-mediated iNOS induction in murine macrophages
    I A Assmann
    Department of Surgery, Klinikum Grosshadern, Ludwig-Maximilians-University Munich, Germany
    FEMS Immunol Med Microbiol 30:133-8. 2001
    ..In contrast, iNOS was not induced in THP-1. This indicates that H. pylori-mediated iNOS induction in J774A.1 is independent of important virulence factors whereas cell contact is crucial which suggests a role of adhesion or phagocytosis...