Diego F Calvisi

Summary

Affiliation: University of Greifswald
Country: Germany

Publications

  1. ncbi Increased lipogenesis, induced by AKT-mTORC1-RPS6 signaling, promotes development of human hepatocellular carcinoma
    Diego F Calvisi
    Institut fur Pathologie, Ernst Moritz Arndt Universitat, Greifswald, Germany
    Gastroenterology 140:1071-83. 2011
  2. ncbi [De novo lipogenesis: role in hepatocellular carcinoma]
    D F Calvisi
    Institut fur Pathologie, Ernst Moritz Arndt Universitätsmedizin, Friedrich Löffler Str 23e, 17489, Greifswald, Deutschland
    Pathologe 32:174-80. 2011
  3. ncbi V-AKT murine thymoma viral oncogene homolog/mammalian target of rapamycin activation induces a module of metabolic changes contributing to growth in insulin-induced hepatocarcinogenesis
    Matthias Evert
    Institut fur Pathologie, Ernst Moritz Arndt Universitat, Greifswald, Germany
    Hepatology 55:1473-84. 2012
  4. ncbi Pathogenetic and Prognostic Significance of Inactivation of RASSF Proteins in Human Hepatocellular Carcinoma
    Diego F Calvisi
    Institut fur Pathologie, Universitätsmedizin Greifswald, 17489 Greifswald, Germany
    Mol Biol Int 2012:849874. 2012
  5. ncbi NORE1A tumor suppressor candidate modulates p21CIP1 via p53
    Diego F Calvisi
    Institut fur Pathologie, Ernst Moritz Arndt Universitat, Greifswald, Germany
    Cancer Res 69:4629-37. 2009

Collaborators

  • Frank Dombrowski
  • Andrea Thiele
  • Xin Chen
  • Matthias Evert
  • Sara Ladu
  • Gioia Gasparetti
  • Valentina De Murtas
  • Antje Zimmermann
  • Sara Thiel
  • Salvatore Delogu
  • Sandra Mattu
  • Giulia Destefanis
  • Katja Evert
  • Silvia Ribback

Detail Information

Publications5

  1. ncbi Increased lipogenesis, induced by AKT-mTORC1-RPS6 signaling, promotes development of human hepatocellular carcinoma
    Diego F Calvisi
    Institut fur Pathologie, Ernst Moritz Arndt Universitat, Greifswald, Germany
    Gastroenterology 140:1071-83. 2011
    ..De novo lipogenesis is believed to be involved in oncogenesis. We investigated the role of aberrant lipid biosynthesis in the pathogenesis of human hepatocellular carcinoma (HCC)...
  2. ncbi [De novo lipogenesis: role in hepatocellular carcinoma]
    D F Calvisi
    Institut fur Pathologie, Ernst Moritz Arndt Universitätsmedizin, Friedrich Löffler Str 23e, 17489, Greifswald, Deutschland
    Pathologe 32:174-80. 2011
    ....
  3. ncbi V-AKT murine thymoma viral oncogene homolog/mammalian target of rapamycin activation induces a module of metabolic changes contributing to growth in insulin-induced hepatocarcinogenesis
    Matthias Evert
    Institut fur Pathologie, Ernst Moritz Arndt Universitat, Greifswald, Germany
    Hepatology 55:1473-84. 2012
    ..Thus, inhibition of AKT/mTOR and related metabolic changes might represent a novel preventive and therapeutic approach to effectively inhibit insulin-induced hepatocarcinogenesis...
  4. ncbi Pathogenetic and Prognostic Significance of Inactivation of RASSF Proteins in Human Hepatocellular Carcinoma
    Diego F Calvisi
    Institut fur Pathologie, Universitätsmedizin Greifswald, 17489 Greifswald, Germany
    Mol Biol Int 2012:849874. 2012
    ..Also, the present findings speak in favour of therapeutic strategies aimed at reexpressing RASSF1A, RASSF2, and RASSF5 genes and/or inactivating the RASSF cellular inhibitors for the treatment of human liver cancer...
  5. ncbi NORE1A tumor suppressor candidate modulates p21CIP1 via p53
    Diego F Calvisi
    Institut fur Pathologie, Ernst Moritz Arndt Universitat, Greifswald, Germany
    Cancer Res 69:4629-37. 2009
    ..Thus, we define the molecular basis of NORE1A-mediated growth inhibition and implicate NORE1A as a potential component of the ill-defined connection between Ras and p53...