Bernhard Kadenbach

Summary

Affiliation: Philipps University
Country: Germany

Publications

  1. doi request reprint Degenerative diseases, oxidative stress and cytochrome c oxidase function
    Bernhard Kadenbach
    Fachbereich Chemie, Philipps University, D 35032 Marburg, Germany
    Trends Mol Med 15:139-47. 2009
  2. doi request reprint High efficiency versus maximal performance--the cause of oxidative stress in eukaryotes: a hypothesis
    Bernhard Kadenbach
    Department of Chemistry, Philipps University, D 35032 Marburg, Germany
    Mitochondrion 13:1-6. 2013
  3. doi request reprint Introduction to mitochondrial oxidative phosphorylation
    Bernhard Kadenbach
    Philipps University Marburg, Marburg, Germany
    Adv Exp Med Biol 748:1-11. 2012
  4. doi request reprint The role of mitochondrial membrane potential in ischemic heart failure
    Bernhard Kadenbach
    Fachbereich Chemie, Philipps Universitaet, D 35032 Marburg, Germany
    Mitochondrion 11:700-6. 2011
  5. doi request reprint New extension of the Mitchell Theory for oxidative phosphorylation in mitochondria of living organisms
    Bernhard Kadenbach
    Fachbereich Chemie, Cardiovascular Laboratory, Philipps University, D 35032 Marburg, Germany
    Biochim Biophys Acta 1800:205-12. 2010
  6. ncbi request reprint Intrinsic and extrinsic uncoupling of oxidative phosphorylation
    Bernhard Kadenbach
    Fachbereich Chemie, Philipps Universitat, Hans Meerwein Strasse, D 35032 Marburg, Germany
    Biochim Biophys Acta 1604:77-94. 2003
  7. doi request reprint Individual biochemical behaviour versus biological robustness: spotlight on the regulation of cytochrome c oxidase
    Rabia Ramzan
    Philipps University, Marburg, Germany
    Adv Exp Med Biol 748:265-81. 2012
  8. ncbi request reprint The possible role of cytochrome c oxidase in stress-induced apoptosis and degenerative diseases
    Bernhard Kadenbach
    Fachbereich Chemie, Philipps Universitat, Hans Meerwein Strasse, D 35032 Marburg, Germany
    Biochim Biophys Acta 1655:400-8. 2004
  9. doi request reprint Mitochondrial respiration and membrane potential are regulated by the allosteric ATP-inhibition of cytochrome c oxidase
    Rabia Ramzan
    Biomedical Research Center, Cardiovascular Laboratory, Philipps University, D 35032 Marburg, Germany
    Biochim Biophys Acta 1797:1672-80. 2010
  10. ncbi request reprint Control of mitochondrial membrane potential and ROS formation by reversible phosphorylation of cytochrome c oxidase
    Icksoo Lee
    Fachereich Chemie, Philipps University, Marburg, Germany
    Mol Cell Biochem 234:63-70. 2002

Collaborators

Detail Information

Publications10

  1. doi request reprint Degenerative diseases, oxidative stress and cytochrome c oxidase function
    Bernhard Kadenbach
    Fachbereich Chemie, Philipps University, D 35032 Marburg, Germany
    Trends Mol Med 15:139-47. 2009
    ..This mechanism is proposed to represent a missing molecular link between stress and degenerative diseases...
  2. doi request reprint High efficiency versus maximal performance--the cause of oxidative stress in eukaryotes: a hypothesis
    Bernhard Kadenbach
    Department of Chemistry, Philipps University, D 35032 Marburg, Germany
    Mitochondrion 13:1-6. 2013
    ..We conclude that 'oxidative stress' occurs when the control of COX activity by the [ATP]/[ADP] ratio is switched off via neural signals...
  3. doi request reprint Introduction to mitochondrial oxidative phosphorylation
    Bernhard Kadenbach
    Philipps University Marburg, Marburg, Germany
    Adv Exp Med Biol 748:1-11. 2012
    ....
  4. doi request reprint The role of mitochondrial membrane potential in ischemic heart failure
    Bernhard Kadenbach
    Fachbereich Chemie, Philipps Universitaet, D 35032 Marburg, Germany
    Mitochondrion 11:700-6. 2011
    ..Ischemic preconditioning is suggested to cause additional phosphorylation of CcO, protecting the enzyme from immediate dephosphorylation via ROS signaling...
  5. doi request reprint New extension of the Mitchell Theory for oxidative phosphorylation in mitochondria of living organisms
    Bernhard Kadenbach
    Fachbereich Chemie, Cardiovascular Laboratory, Philipps University, D 35032 Marburg, Germany
    Biochim Biophys Acta 1800:205-12. 2010
    ....
  6. ncbi request reprint Intrinsic and extrinsic uncoupling of oxidative phosphorylation
    Bernhard Kadenbach
    Fachbereich Chemie, Philipps Universitat, Hans Meerwein Strasse, D 35032 Marburg, Germany
    Biochim Biophys Acta 1604:77-94. 2003
    ..Finally, the regulation of DeltaPsi and the production of reactive oxygen species (ROS) in mitochondria at high DeltaPsi values (150-200 mV) are discussed...
  7. doi request reprint Individual biochemical behaviour versus biological robustness: spotlight on the regulation of cytochrome c oxidase
    Rabia Ramzan
    Philipps University, Marburg, Germany
    Adv Exp Med Biol 748:265-81. 2012
    ....
  8. ncbi request reprint The possible role of cytochrome c oxidase in stress-induced apoptosis and degenerative diseases
    Bernhard Kadenbach
    Fachbereich Chemie, Philipps Universitat, Hans Meerwein Strasse, D 35032 Marburg, Germany
    Biochim Biophys Acta 1655:400-8. 2004
    ..Consequently, energy metabolism changes to an excited state. We propose that this change causes an increase in DeltaPsi(m), a condition for the formation of ROS and induction of apoptosis...
  9. doi request reprint Mitochondrial respiration and membrane potential are regulated by the allosteric ATP-inhibition of cytochrome c oxidase
    Rabia Ramzan
    Biomedical Research Center, Cardiovascular Laboratory, Philipps University, D 35032 Marburg, Germany
    Biochim Biophys Acta 1797:1672-80. 2010
    ..quot; Only when the allosteric ATP-inhibition is switched off under stress, respiration is regulated by "respiratory control," based on DeltaPsi(m) according to the Mitchell Theory...
  10. ncbi request reprint Control of mitochondrial membrane potential and ROS formation by reversible phosphorylation of cytochrome c oxidase
    Icksoo Lee
    Fachereich Chemie, Philipps University, Marburg, Germany
    Mol Cell Biochem 234:63-70. 2002
    ....