Research Topics
Genomes and GenesSpecies | Magdalena HuberSummaryAffiliation: Philipps University Country: Germany Publications
| Collaborators
|
Detail Information
Publications
c-Rel is crucial for the induction of Foxp3(+) regulatory CD4(+) T cells but not T(H)17 cellsAlexander Visekruna
Institute for Medical Microbiology and Hygiene, University of Marburg, Marburg, Germany
Eur J Immunol 40:671-6. 2010..The addition of exogenous IL-2 was sufficient to rescue the development of c-Rel-deficient iTreg. Thus, c-Rel is essential for the development of Foxp3(+) Treg but not for T(H)17 cells via regulating the production of IL-2...
IL-27 inhibits the development of regulatory T cells via STAT3Magdalena Huber
Institut fur Medizinische Mikrobiologie und Hygiene, Universitat Marburg, Hans Meerwein Strasse 2, 35043 Marburg, Germany
Int Immunol 20:223-34. 2008..Thus, IL-27 and TGFbeta mutually control their effects on CD4+ T-cell differentiation, whereby IL-27 favours inflammatory conditions through a STAT3-dependent inhibition of Treg generation...
IRF4 is essential for IL-21-mediated induction, amplification, and stabilization of the Th17 phenotypeMagdalena Huber
Institut fur Medizinische Mikrobiologie und Hygiene, Universitat Marburg, 35043 Marburg, Germany
Proc Natl Acad Sci U S A 105:20846-51. 2008..These data highlight IRF4 as a decisive factor during the IL-21-mediated steps of Th17 development by influencing the balance of Foxp3, RORalpha, and RORgammat...
A Th17-like developmental process leads to CD8(+) Tc17 cells with reduced cytotoxic activityMagdalena Huber
Institute for Medical Microbiology and Hygiene, University of Marburg, Germany
Eur J Immunol 39:1716-25. 2009..Furthermore, Tc17 cells producing IL-17 but not granzyme B are also detectable in EAE, a mouse model for multiple sclerosis. Our data point to the existence of mutually exclusive CTL and Tc17 developmental pathways in vitro and in vivo...
The development of inflammatory T(H)-17 cells requires interferon-regulatory factor 4Anne Brüstle
Institut für Medizinische Mikrobiologie und Krankenhaushygiene, 35043 Marburg, Germany
Nat Immunol 8:958-66. 2007..Our data position IRF4 at the center of T helper cell development, influencing not only T helper type 2 but also T(H)-17 differentiation...
Transcription factor IRF4 determines germinal center formation through follicular T-helper cell differentiationNadine Bollig
Institut für Medizinische Mikrobiologie und Krankenhaushygiene, Universitat Marburg, 35043 Marburg, Germany
Proc Natl Acad Sci U S A 109:8664-9. 2012..Together with its known B lineage-specific roles during plasma cell maturation and class switch, our study places IRF4 in the center of antibody production toward T-cell-dependent antigens...
IL-17A secretion by CD8+ T cells supports Th17-mediated autoimmune encephalomyelitisMagdalena Huber
Institute for Medical Microbiology and Hygiene, University of Marburg, Marburg, Germany
J Clin Invest 123:247-60. 2013..Our results reveal that Tc17 cells contribute to the initiation of CNS autoimmunity in mice and humans by supporting Th17 cell pathogenicity...
c-Rel promotes type 1 and type 17 immune responses during Leishmania major infectionKatharina Reinhard
Institute for Medical Microbiology and Hygiene, University of Marburg, Marburg, Germany
Eur J Immunol 41:1388-98. 2011..In conclusion, our data suggest that the expression of c-Rel in myeloid cells is essential for clearance of L. major and that this c-Rel-mediated effect is dominant over the lack of Tregs...
The role of NF-κB activation during protection against Leishmania infectionKatharina Reinhard
Institute for Medical Microbiology and Hygiene, University of Marburg, Hans Meerwein Straße 2, 35032 Marburg, Germany
Int J Med Microbiol 302:230-5. 2012....
Tc9 cells, a new subset of CD8(+) T cells, support Th2-mediated airway inflammationAlexander Visekruna
Institute for Medical Microbiology and Hygiene, University of Marburg, Marburg, Germany
Eur J Immunol 43:606-18. 2013..Thus, our data point to the existence of Tc9 cells and to their supportive function in Th2-dependent airway inflammation, suggesting that these cells might be a therapeutic target in allergic disorders...
Multidrug-resistance-associated protein 1 (Mrp1) is probably not required for murine Th cell activationPeter Kleemann
Institut fuer Medizinische Mikrobiologie und Krankenhaushygiene, University of Marburg, BMFZ, Hans Meerweinstrasse, 35043 Marburg, Germany
Int Immunol 18:1603-6. 2006....
