Verena Pawlak

Summary

Affiliation: Max Planck Institute for Biological Cybernetics
Country: Germany

Publications

  1. doi request reprint Dopamine receptor activation is required for corticostriatal spike-timing-dependent plasticity
    Verena Pawlak
    Department of Cell Physiology, Max Planck Institute for Medical Research, 69120 Heidelberg, Germany
    J Neurosci 28:2435-46. 2008
  2. ncbi request reprint Frequency-dependent impairment of hippocampal LTP from NMDA receptors with reduced calcium permeability
    Verena Pawlak
    Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Heidelberg, Germany
    Eur J Neurosci 22:476-84. 2005
  3. ncbi request reprint Excitotoxicity in vitro by NR2A- and NR2B-containing NMDA receptors
    Jakob von Engelhardt
    IZN and Department of Clinical Neurobiology, University Hospital of Neurology, INF 364, 69120 Heidelberg, Germany
    Neuropharmacology 53:10-7. 2007
  4. ncbi request reprint Lack of NMDA receptor subtype selectivity for hippocampal long-term potentiation
    Sven Berberich
    Department of Molecular Neurobiology, Max Planck Institute for Medical Research, D 69120 Heidelberg, Germany
    J Neurosci 25:6907-10. 2005
  5. pmc Impaired synaptic scaling in mouse hippocampal neurones expressing NMDA receptors with reduced calcium permeability
    Verena Pawlak
    Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Jahnstrasse 29, D 69120 Heidelberg, Germany
    J Physiol 562:771-83. 2005
  6. ncbi request reprint Actin/alpha-actinin-dependent transport of AMPA receptors in dendritic spines: role of the PDZ-LIM protein RIL
    Torsten W Schulz
    Max Planck Institute for Medical Research, Department of Molecular Neurobiology, 69120 Heidelberg, Germany
    J Neurosci 24:8584-94. 2004

Collaborators

Detail Information

Publications6

  1. doi request reprint Dopamine receptor activation is required for corticostriatal spike-timing-dependent plasticity
    Verena Pawlak
    Department of Cell Physiology, Max Planck Institute for Medical Research, 69120 Heidelberg, Germany
    J Neurosci 28:2435-46. 2008
    ..Thus, the timing of APs relative to cortical inputs alone is not enough to induce corticostriatal plasticity, implying that ongoing activity does not affect synaptic strength unless dopamine receptors are activated...
  2. ncbi request reprint Frequency-dependent impairment of hippocampal LTP from NMDA receptors with reduced calcium permeability
    Verena Pawlak
    Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Heidelberg, Germany
    Eur J Neurosci 22:476-84. 2005
    ..Moreover, LTP induced by the first HFS stimulus train required lower Ca2+ levels than the additional LTP obtained by repeated trains...
  3. ncbi request reprint Excitotoxicity in vitro by NR2A- and NR2B-containing NMDA receptors
    Jakob von Engelhardt
    IZN and Department of Clinical Neurobiology, University Hospital of Neurology, INF 364, 69120 Heidelberg, Germany
    Neuropharmacology 53:10-7. 2007
    ..Our findings predict that successful therapeutic intervention in stroke based on currently available NMDA receptor subtype-selective blockers is unlikely...
  4. ncbi request reprint Lack of NMDA receptor subtype selectivity for hippocampal long-term potentiation
    Sven Berberich
    Department of Molecular Neurobiology, Max Planck Institute for Medical Research, D 69120 Heidelberg, Germany
    J Neurosci 25:6907-10. 2005
    ....
  5. pmc Impaired synaptic scaling in mouse hippocampal neurones expressing NMDA receptors with reduced calcium permeability
    Verena Pawlak
    Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Jahnstrasse 29, D 69120 Heidelberg, Germany
    J Physiol 562:771-83. 2005
    ..Our results indicate that NMDAR-mediated Ca(2+) signalling is important for synaptic scaling of AMPA currents in the hippocampus in vivo...
  6. ncbi request reprint Actin/alpha-actinin-dependent transport of AMPA receptors in dendritic spines: role of the PDZ-LIM protein RIL
    Torsten W Schulz
    Max Planck Institute for Medical Research, Department of Molecular Neurobiology, 69120 Heidelberg, Germany
    J Neurosci 24:8584-94. 2004
    ....