Teymuras V Kurzchalia

Summary

Affiliation: Max Planck Institute of Molecular Cell Biology and Genetics
Country: Germany

Publications

  1. ncbi request reprint Why do worms need cholesterol?
    Teymuras V Kurzchalia
    Max Planck Institute for Molecular Cell Biology and Genetics, Dresden, D 01307, Germany
    Nat Cell Biol 5:684-8. 2003
  2. doi request reprint Methylation of the sterol nucleus by STRM-1 regulates dauer larva formation in Caenorhabditis elegans
    J Thomas Hannich
    Max Planck Institute for Molecular Cell Biology and Genetics, Dresden, Germany
    Dev Cell 16:833-43. 2009
  3. doi request reprint Two cytochrome P450s in Caenorhabditis elegans are essential for the organization of eggshell, correct execution of meiosis and the polarization of embryo
    Gaspare Benenati
    Max Planck Institute for Molecular Cell Biology and Genetics, Dresden, Germany
    Mech Dev 126:382-93. 2009
  4. pmc Sterol-derived hormone(s) controls entry into diapause in Caenorhabditis elegans by consecutive activation of DAF-12 and DAF-16
    Vitali Matyash
    Max Planck Institute for Molecular Cell Biology and Genetics, Dresden, Germany
    PLoS Biol 2:e280. 2004
  5. ncbi request reprint Requirement of sterols in the life cycle of the nematode Caenorhabditis elegans
    Eugeni V Entchev
    MPI for Molecular Cell Biology and Genetics, Pfotenhauerstrasse 108, 01307 Dresden, Germany
    Semin Cell Dev Biol 16:175-82. 2005
  6. doi request reprint LET-767 is required for the production of branched chain and long chain fatty acids in Caenorhabditis elegans
    Eugeni V Entchev
    Max Planck Institute of Molecular Cell Biology and Genetics, Pfotenhauerstrasse 108, 01307 Dresden, Germany
    J Biol Chem 283:17550-60. 2008
  7. pmc Lipid extraction by methyl-tert-butyl ether for high-throughput lipidomics
    Vitali Matyash
    Max Planck Institute of Molecular Cell Biology and Genetics, 01307 Dresden, Germany
    J Lipid Res 49:1137-46. 2008
  8. ncbi request reprint Cholesterol-dependent lipid assemblies regulate the activity of the ecto-nucleotidase CD39
    Agathi Papanikolaou
    Laboratory of Biological Chemistry, Medical School, University of Ioannina, 45110 Ioannina, Greece
    J Biol Chem 280:26406-14. 2005
  9. pmc Ultrastructural identification of uncoated caveolin-independent early endocytic vehicles
    Matthew Kirkham
    Institute for Molecular Bioscience, University of Queensland, Queensland 4072, Australia
    J Cell Biol 168:465-76. 2005
  10. ncbi request reprint Caveolin interacts with the angiotensin II type 1 receptor during exocytic transport but not at the plasma membrane
    Bruce D Wyse
    Department of Molecular and Cellular Pathology, University of Queensland, Brisbane, Queensland, Australia
    J Biol Chem 278:23738-46. 2003

Collaborators

  • Christoph Thiele
  • Vyacheslav Zagoriy
  • Bianca Habermann
  • Robert G Parton
  • Eugeni V Entchev
  • Vitali Matyash
  • Gaspare Benenati
  • J Thomas Hannich
  • Andrej Shevchenko
  • Dominik Schwudke
  • Hans Joachim Knölker
  • Fanny Mende
  • Agathi Papanikolaou
  • Matthew Kirkham
  • John F Hancock
  • Bruce D Wyse
  • Sider Penkov
  • Gabriele Theumer
  • Rene Martin
  • Hristio Boytchev
  • Thomas Müller-Reichert
  • Isabelle Riezman
  • Howard Riezman
  • Aliona Bogdanova
  • Gerhard Liebisch
  • Lin Zhu
  • Rahul Chadda
  • Orestes Tsolas
  • Akikazu Fujita
  • Michael Kasper
  • Alexandra Papafotika
  • Richard E Pagano
  • Marek Drab
  • Satyajit Mayor
  • Susan J Nixon
  • Deepak K Sharma
  • Thomais Papamarcaki
  • Carol Murphy
  • Savvas Christoforidis
  • Samuel Ward
  • Arndt W Schmidt
  • Michaela Wilsch-Bräuninger
  • Hongwei Qian
  • Ian A Prior
  • Isabel C Morrow
  • Susan Nixon
  • Walter G Thomas
  • Cornelia Muncke

Detail Information

Publications10

  1. ncbi request reprint Why do worms need cholesterol?
    Teymuras V Kurzchalia
    Max Planck Institute for Molecular Cell Biology and Genetics, Dresden, D 01307, Germany
    Nat Cell Biol 5:684-8. 2003
    ..Instead, its most probable signalling functions are to control molting and induce a specialized non-feeding larval stage, although no cholesterol-derived signalling molecule has yet been identified for these or any other functions...
  2. doi request reprint Methylation of the sterol nucleus by STRM-1 regulates dauer larva formation in Caenorhabditis elegans
    J Thomas Hannich
    Max Planck Institute for Molecular Cell Biology and Genetics, Dresden, Germany
    Dev Cell 16:833-43. 2009
    ..Moreover, the C-4 sterol nucleus methylation catalyzed by STRM-1 is unique to nematodes and thus could be a target for therapeutic strategies against parasitic nematode infections...
  3. doi request reprint Two cytochrome P450s in Caenorhabditis elegans are essential for the organization of eggshell, correct execution of meiosis and the polarization of embryo
    Gaspare Benenati
    Max Planck Institute for Molecular Cell Biology and Genetics, Dresden, Germany
    Mech Dev 126:382-93. 2009
    ..Using a complementation with lipid extracts, we show that POD-7/POD-8 function together with a NADPH cytochrome P450 reductase, coded by emb-8, and are involved in the production of lipid(s) required for eggshell formation...
  4. pmc Sterol-derived hormone(s) controls entry into diapause in Caenorhabditis elegans by consecutive activation of DAF-12 and DAF-16
    Vitali Matyash
    Max Planck Institute for Molecular Cell Biology and Genetics, Dresden, Germany
    PLoS Biol 2:e280. 2004
    ..This hormonal control of DAF-16 activation is, however, independent of insulin signalling and has no influence on life span...
  5. ncbi request reprint Requirement of sterols in the life cycle of the nematode Caenorhabditis elegans
    Eugeni V Entchev
    MPI for Molecular Cell Biology and Genetics, Pfotenhauerstrasse 108, 01307 Dresden, Germany
    Semin Cell Dev Biol 16:175-82. 2005
    ..Investigations on C. elegans could provide information on the etiology of human diseases that display defects in the transport or metabolism of sterols...
  6. doi request reprint LET-767 is required for the production of branched chain and long chain fatty acids in Caenorhabditis elegans
    Eugeni V Entchev
    Max Planck Institute of Molecular Cell Biology and Genetics, Pfotenhauerstrasse 108, 01307 Dresden, Germany
    J Biol Chem 283:17550-60. 2008
    ..elegans required for the bulk production of monomethyl branched and long chain fatty acids, and the developmental arrest in let-767 (RNAi) worms is caused by the deficiency of the former...
  7. pmc Lipid extraction by methyl-tert-butyl ether for high-throughput lipidomics
    Vitali Matyash
    Max Planck Institute of Molecular Cell Biology and Genetics, 01307 Dresden, Germany
    J Lipid Res 49:1137-46. 2008
    ..Rigorous testing demonstrated that the MTBE protocol delivers similar or better recoveries of species of most all major lipid classes compared with the "gold-standard" Folch or Bligh and Dyer recipes...
  8. ncbi request reprint Cholesterol-dependent lipid assemblies regulate the activity of the ecto-nucleotidase CD39
    Agathi Papanikolaou
    Laboratory of Biological Chemistry, Medical School, University of Ioannina, 45110 Ioannina, Greece
    J Biol Chem 280:26406-14. 2005
    ..These data suggest a functional link between the localization of CD39 in cholesterol-rich domains of the membrane and its role in thromboregulation...
  9. pmc Ultrastructural identification of uncoated caveolin-independent early endocytic vehicles
    Matthew Kirkham
    Institute for Molecular Bioscience, University of Queensland, Queensland 4072, Australia
    J Cell Biol 168:465-76. 2005
    ..These carriers contained GPI-anchored proteins and fluid phase markers and represented the major vehicles mediating CTB uptake in both WT and caveolae-null cells...
  10. ncbi request reprint Caveolin interacts with the angiotensin II type 1 receptor during exocytic transport but not at the plasma membrane
    Bruce D Wyse
    Department of Molecular and Cellular Pathology, University of Queensland, Brisbane, Queensland, Australia
    J Biol Chem 278:23738-46. 2003
    ..Caveolin therefore acts as a molecular chaperone rather than a plasma membrane scaffold for AT1-R...