Genomes and Genes
Affiliation: Max Planck Institute for Molecular Genetics
- Disruption and pseudoautosomal localization of the major histocompatibility complex in monotremesJuliane C Dohm
Max Planck Institute for Molecular Genetics, Ihnestr, 63 73, 14195 Berlin, Germany
Genome Biol 8:R175. 2007..To understand the evolution of the mammalian major histocompatibility complex (MHC), the analysis of the monotreme genome is vital...
- Interspersed repetitive sequence (IRS)-PCR for typing of whole genome radiation hybrid panelsH Himmelbauer
Max Planck Institute of Molecular Genetics, Ihnestrasse 73, D 14195 Berlin Dahlem, Germany
Nucleic Acids Res 28:e7. 2000..To test the technique, we have mapped 48 BAC clones derived from mouse chromosome 12 which we mostly identified using complex probes. As mammalian genomes are repeat-rich, the technology can easily be adapted to species other than mouse...
- Expression of the von Hippel-Lindau-binding protein-1 (Vbp1) in fetal and adult mouse tissuesM Hemberger
Max Planck Institut fur Molekulare Genetik, Ihnestrasse 73, D 14195 Berlin Dahlem, Germany, Fakultät für Biologie III, Universitat Freiburg, Freiburg, Germany
Hum Mol Genet 8:229-36. 1999..Mapping of the murine Vbp1 gene revealed conserved chromosomal localization between mouse and human in a region homologous to human Xq28...
- Advanced integrated mouse YAC map including BAC frameworkL C Schalkwyk
Max Planck Institute of Molecular Genetics, Ihnestrasse 73, D 14195 Berlin, Germany
Genome Res 11:2142-50. 2001..and Nusbaum et al. There is a total of 20,205 markers on the final map, 12,033 from our own data, and a total of 56,093 YACs, of which 44,401 are positive for more than one marker...
- Characterization of the mouse Src homology 3 domain gene Sh3d2c on Chr 7 demonstrates coexpression with huntingtin in the brain and identifies the processed pseudogene Sh3d2c-ps1 on Chr 2U Zechner
Max Planck Institute for Molecular Genetics, Ihnestrasse 73, Berlin Dahlem, D14195, Germany
Genomics 54:505-10. 1998..Because huntingtin and Sh3d2c are coexpressed in most regions of the brain, it can be speculated that there is a link between the association of huntingtin/Sh3d2c and the pathogenesis of Huntington disease...