Research Topics
Species | Ingo KennerknechtSummaryCountry: Germany Publications
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Publications
First report of prevalence of non-syndromic hereditary prosopagnosia (HPA)Ingo Kennerknecht
Institute of Human Genetics, Westfalische Wilhelms Universitat, Munster, Germany
Am J Med Genet A 140:1617-22. 2006..The segregation pattern of this hereditary prosopagnosia (HPA) is fully compatible with autosomal dominant inheritance...
Hereditary prosopagnosia (HPA): the first report outside the Caucasian populationIngo Kennerknecht
Institut fur Humangenetik, Westfalische Wilhelms Universitat, Munster, Germany
J Hum Genet 52:230-6. 2007..Several other members of her large family reported the same impairment of face recognition. The segregation pattern of PA in this family is also compatible with autosomal-dominant inheritance...
Hereditary prosopagnosia: the first case seriesMartina Grueter
Institute of Human Genetics, Westfalische Wilhelms University, Munster, Germany
Cortex 43:734-49. 2007..The results provide compelling evidence for significant genetic contribution to face recognition skills and contribute to the promise offered by the emerging field of cognitive neurogenetics...
Congenital prosopagnosia--a common hereditary cognitive dysfunction in humansIngo Kennerknecht
Institut fuer Humangenetik, Westfaelische Wilhelms Universitaet, Muenster, Germany
Front Biosci 13:3150-8. 2008..Still fitting the concept of autosomal dominant inheritance, we have evidence for a slightly reduced penetrance (4 normal transmitters from distinct families) and one or two de novo mutations...
Prevalence of hereditary prosopagnosia (HPA) in Hong Kong Chinese populationIngo Kennerknecht
Institute of Human Genetics, Westfalische Wilhelms Universitat, Munster, Germany
Am J Med Genet A 146:2863-70. 2008..Each had other first-degree relatives with the same visual cognitive dysfunction. Thus, as in the Caucasians, regular autosomal dominant inheritance might best explain the segregation pattern...
Novel der(1)t(1;19) in two patients with myeloid neoplasiasJoelle Tchinda
Institut fur Humangenetik, Westfalische Wilhelms Universitat, Vesaliusweg 12 14, Munster, Germany
Cancer Genet Cytogenet 133:61-5. 2002..Translocation (1;19)(p13;p13.1) may play a role in the leukemogenesis of myeloid diseases...
A novel TACSTD2 gene mutation in a Turkish family with a gelatinous drop-like corneal dystrophyArseni Markoff
Institute of Medical Biochemistry, Zentrum für Molekularbiologie der Entzündung ZMBE, Munster, Germany
Mol Vis 12:1473-6. 2006..To identify the molecular defect causing gelatinous drop-like corneal dystrophy in a Turkish family and assign affected and carriership status...
Mild phenotype in two unrelated patients with a partial deletion of 21q22.2-q22.3 defined by FISH and molecular studiesDaniela Ehling
Praenadia GmbH, Muenster, Germany
Am J Med Genet A 131:265-72. 2004..9 Mb of 21q, may result in mild phenotypes comprising facial anomalies, thin marfanoid build, and mild MR, with or without signs of holoprosencephaly...
Multicolor karyotyping in acute myeloid leukemiaJoelle Tchinda
Institut fur Humangenetik, Universitatsklinikum Munster, Vesaliusweg 12 14, 48129 Munster, Germany
Leuk Lymphoma 44:1843-53. 2003..Since, more than 600 cases of AML and MDS have been analyzed. Herein, we attempt to summarize the data published and discuss what has been achieved towards realization of these goals...
Gaze behaviour in hereditary prosopagnosiaGudrun Schwarzer
University of Giessen, Otto Behaghel Strasse 10F, 35394 Giessen, Germany
Psychol Res 71:583-90. 2007..They had a more dispersed gaze and also fixated external facial features. Thus, the face recognition impairment of the hereditary prosopagnosics is reflected in their gaze behaviour...
Situs ambiguus in a female fetus with balanced (X;21) translocation--evidence for functional nullisomy of the ZIC3 gene?Barbara Fritz
Institut für Klinische Genetik der Philipps Universität Marburg, Germany
Eur J Hum Genet 13:34-40. 2005..A positional effect caused by the balanced (X;21) translocation may be responsible for functional nullisomy of ZIC3 and thus explain the situs and cardiac abnormalities in the fetus...
