Bernd Hohenstein

Summary

Country: Germany

Publications

  1. ncbi Immunoadsorption for connective tissue disease
    B Hohenstein
    Department of Internal Medicine III, University Hospital Carl Gustav Carus, Fetscherstr 74, 01307 Dresden, Germany Electronic address
    Atheroscler Suppl 14:185-9. 2013
  2. ncbi Enhanced progenitor cell recruitment and endothelial repair after selective endothelial injury of the mouse kidney
    Bernd Hohenstein
    Division of Nephrology, Department of Internal Medicine III, University of Technology Dresden, Germany
    Am J Physiol Renal Physiol 298:F1504-14. 2010
  3. ncbi Autocrine VEGF-VEGF-R loop on podocytes during glomerulonephritis in humans
    Bernd Hohenstein
    Division of Nephrology, Medical Clinic III, University of Dresden, Dresden, Germany
    Nephrol Dial Transplant 25:3170-80. 2010
  4. ncbi Vasodilator-stimulated phosphoprotein-deficient mice demonstrate increased platelet activation but improved renal endothelial preservation and regeneration in passive nephrotoxic nephritis
    Bernd Hohenstein
    Division of Nephrology, University Erlangen Nuremberg, Loschgestrasse 8, Erlangen 91054, Germany
    J Am Soc Nephrol 16:986-96. 2005
  5. ncbi Platelets are not critical effector cells for the time course of murine passive crescentic glomerulonephritis
    Bernd Hohenstein
    Division of Nephrology, Department of Internal Medicine III, University of Technology, Dresden, Germany
    Platelets 24:267-74. 2013

Detail Information

Publications5

  1. ncbi Immunoadsorption for connective tissue disease
    B Hohenstein
    Department of Internal Medicine III, University Hospital Carl Gustav Carus, Fetscherstr 74, 01307 Dresden, Germany Electronic address
    Atheroscler Suppl 14:185-9. 2013
    ..Despite the presence of a large number of case series and few controlled trials using IAS, there is a need for further prospective randomized trials to clearly define the role of IAS in these CTD's...
  2. ncbi Enhanced progenitor cell recruitment and endothelial repair after selective endothelial injury of the mouse kidney
    Bernd Hohenstein
    Division of Nephrology, Department of Internal Medicine III, University of Technology Dresden, Germany
    Am J Physiol Renal Physiol 298:F1504-14. 2010
    ..Injection of a high dose of erythropoietin 2 h after disease induction markedly attenuated vascular injury through nonhemodynamic mechanisms, possibly involving vascular endothelial growth factor release...
  3. ncbi Autocrine VEGF-VEGF-R loop on podocytes during glomerulonephritis in humans
    Bernd Hohenstein
    Division of Nephrology, Medical Clinic III, University of Dresden, Dresden, Germany
    Nephrol Dial Transplant 25:3170-80. 2010
    ..Its activity is critical for capillary/glomerular preservation and repair, and recent studies have also demonstrated its relevance for the preservation of podocytes...
  4. ncbi Vasodilator-stimulated phosphoprotein-deficient mice demonstrate increased platelet activation but improved renal endothelial preservation and regeneration in passive nephrotoxic nephritis
    Bernd Hohenstein
    Division of Nephrology, University Erlangen Nuremberg, Loschgestrasse 8, Erlangen 91054, Germany
    J Am Soc Nephrol 16:986-96. 2005
    ..Despite an enhanced injury response early on, VASP -/- mice are protected from long-term progression of nephrotoxic nephritis, which is associated with improved renal endothelial cell preservation and regeneration...
  5. ncbi Platelets are not critical effector cells for the time course of murine passive crescentic glomerulonephritis
    Bernd Hohenstein
    Division of Nephrology, Department of Internal Medicine III, University of Technology, Dresden, Germany
    Platelets 24:267-74. 2013
    ..These data indicate that platelets play a minor role during the time course of this disease model in the mouse...