Laurent Reber

Summary

Country: France

Publications

  1. ncbi Stem cell factor expression, mast cells and inflammation in asthma
    Carla A Da Silva
    EA3771, Inflammation and Environment in Asthma, , BP 24, 67401 Illkirch Cedex, France
    Fundam Clin Pharmacol 20:21-39. 2006
  2. ncbi Stem cell factor and its receptor c-Kit as targets for inflammatory diseases
    Laurent Reber
    EA 3771 Inflammation and Environment in Asthma, Université Louis Pasteur Strasbourg I, Faculte de Pharmacie, Illkirch, France
    Eur J Pharmacol 533:327-40. 2006
  3. ncbi Ser276 phosphorylation of NF-kB p65 by MSK1 controls SCF expression in inflammation
    Laurent Reber
    EA3771, Inflammation and Environment in Asthma, Université Louis Pasteur Strasbourg I, Faculte de Pharmacie, Illkirch, France
    PLoS ONE 4:e4393. 2009

Detail Information

Publications3

  1. ncbi Stem cell factor expression, mast cells and inflammation in asthma
    Carla A Da Silva
    EA3771, Inflammation and Environment in Asthma, , BP 24, 67401 Illkirch Cedex, France
    Fundam Clin Pharmacol 20:21-39. 2006
    ..We here report the main pathways of SCF synthesis and signalling, and its potential role on airway function and asthma...
  2. ncbi Stem cell factor and its receptor c-Kit as targets for inflammatory diseases
    Laurent Reber
    EA 3771 Inflammation and Environment in Asthma, Université Louis Pasteur Strasbourg I, Faculte de Pharmacie, Illkirch, France
    Eur J Pharmacol 533:327-40. 2006
    ..Taken together, these data suggest that SCF/c-Kit may be a potential therapeutic target for the control of mast cell and eosinophil number and activation in inflammatory diseases...
  3. ncbi Ser276 phosphorylation of NF-kB p65 by MSK1 controls SCF expression in inflammation
    Laurent Reber
    EA3771, Inflammation and Environment in Asthma, Université Louis Pasteur Strasbourg I, Faculte de Pharmacie, Illkirch, France
    PLoS ONE 4:e4393. 2009
    ..Our study provides therefore a direct link between MSK1-mediated phosphorylation of Ser276 p65 of NF-kappaB, allowing its binding to the SCF intronic enhancer, and pathophysiological SCF expression in inflammation...