Heta Yrjänäinen

Summary

Affiliation: University of Turku
Country: Finland

Publications

  1. ncbi Persistent joint swelling and Borrelia-specific antibodies in Borrelia garinii-infected mice after eradication of vegetative spirochetes with antibiotic treatment
    Heta Yrjänäinen
    Department of Medical Microbiology, University of Turku, Kiinamyllynkatu 13, 20520, Turku, Finland
    Microbes Infect 8:2044-51. 2006
  2. ncbi Anti-tumor necrosis factor-alpha treatment activates Borrelia burgdorferi spirochetes 4 weeks after ceftriaxone treatment in C3H/He mice
    Heta Yrjänäinen
    Department of Medical Microbiology, University of Turku, Turku, 20520, Finland
    J Infect Dis 195:1489-96. 2007
  3. ncbi Persistence of borrelial DNA in the joints of Borrelia burgdorferi-infected mice after ceftriaxone treatment
    Heta Yrjänäinen
    Department of Medical Microbiology and Immunology, University of Turku, Finland
    APMIS 118:665-73. 2010
  4. ncbi Disordered lymphoid purine metabolism contributes to the pathogenesis of persistent Borrelia garinii infection in mice
    Gennady G Yegutkin
    MediCity Research Laboratory, University of Turku, Tykistokatu 6A, 20520 Turku, Finland
    J Immunol 184:5112-20. 2010
  5. ncbi TLR2 utilization of Borrelia does not induce p38- and IFN-beta autocrine loop-dependent expression of CD38, resulting in poor migration and weak IL-12 secretion of dendritic cells
    Pauliina Hartiala
    Department of Medical Microbiology and Immunology, University of Turku, Finland
    J Immunol 184:5732-42. 2010

Detail Information

Publications5

  1. ncbi Persistent joint swelling and Borrelia-specific antibodies in Borrelia garinii-infected mice after eradication of vegetative spirochetes with antibiotic treatment
    Heta Yrjänäinen
    Department of Medical Microbiology, University of Turku, Kiinamyllynkatu 13, 20520, Turku, Finland
    Microbes Infect 8:2044-51. 2006
    ..The results suggest that the presence of vegetative spirochetes is no prerequisite for persisting joint symptoms and elevated anti-borrelia IgG levels in these B. garinii-infected mice...
  2. ncbi Anti-tumor necrosis factor-alpha treatment activates Borrelia burgdorferi spirochetes 4 weeks after ceftriaxone treatment in C3H/He mice
    Heta Yrjänäinen
    Department of Medical Microbiology, University of Turku, Turku, 20520, Finland
    J Infect Dis 195:1489-96. 2007
    ..The effect of anti-tumor necrosis factor (TNF)-alpha treatment in Borrelia burgdorferi-infected and ceftriaxone-treated C3H/He mice was evaluated...
  3. ncbi Persistence of borrelial DNA in the joints of Borrelia burgdorferi-infected mice after ceftriaxone treatment
    Heta Yrjänäinen
    Department of Medical Microbiology and Immunology, University of Turku, Finland
    APMIS 118:665-73. 2010
    ..In conclusion, these results combined with earlier results suggest that the joint or a tissue adjacent to the joint is the niche of persisting B. burgdorferi in ceftriaxone-treated mice...
  4. ncbi Disordered lymphoid purine metabolism contributes to the pathogenesis of persistent Borrelia garinii infection in mice
    Gennady G Yegutkin
    MediCity Research Laboratory, University of Turku, Tykistokatu 6A, 20520 Turku, Finland
    J Immunol 184:5112-20. 2010
    ....
  5. ncbi TLR2 utilization of Borrelia does not induce p38- and IFN-beta autocrine loop-dependent expression of CD38, resulting in poor migration and weak IL-12 secretion of dendritic cells
    Pauliina Hartiala
    Department of Medical Microbiology and Immunology, University of Turku, Finland
    J Immunol 184:5732-42. 2010
    ..Our results provide evidence for the first time that different TLR use results in different CD38 expression, which correlates with the migratory potential of DCs...