Olayinka Raheem

Summary

Affiliation: University of Tampere
Country: Finland

Publications

  1. pmc New immunohistochemical method for improved myotonia and chloride channel mutation diagnostics
    Olayinka Raheem
    Neuromuscular Research Unit, University of Tampere and Tampere University Hospital, Tampere, Finland
    Neurology 79:2194-200. 2012
  2. doi request reprint Novel myosin heavy chain immunohistochemical double staining developed for the routine diagnostic separation of I, IIA and IIX fibers
    Olayinka Raheem
    Department of Neurology, Neuromuscular Molecular Pathology, University of Tampere, Biokatu 10, Finn Medi 3, 33520 Tampere, Finland
    Acta Neuropathol 119:495-500. 2010
  3. pmc Mutant (CCTG)n expansion causes abnormal expression of zinc finger protein 9 (ZNF9) in myotonic dystrophy type 2
    Olayinka Raheem
    Neuromuscular Research Unit, University of Tampere and Tampere University Hospital, Tampere, Finland
    Am J Pathol 177:3025-36. 2010
  4. doi request reprint Distinct distal myopathy phenotype caused by VCP gene mutation in a Finnish family
    Johanna Palmio
    Neuromuscular Research Unit, Department of Neurology, University Hospital and University of Tampere, Finland
    Neuromuscul Disord 21:551-5. 2011
  5. doi request reprint The enigma of 7q36 linked autosomal dominant limb girdle muscular dystrophy
    Satu Sandell
    Department of Neurology, Seinajoki Central Hospital, Seinajoki, Finland
    J Neurol Neurosurg Psychiatry 81:834-9. 2010

Collaborators

Detail Information

Publications5

  1. pmc New immunohistochemical method for improved myotonia and chloride channel mutation diagnostics
    Olayinka Raheem
    Neuromuscular Research Unit, University of Tampere and Tampere University Hospital, Tampere, Finland
    Neurology 79:2194-200. 2012
    ..The objective of this study was to validate the immunohistochemical assay for the diagnosis of nondystrophic myotonia and to provide full clarification of clinical disease to patients in whom basic genetic testing has failed to do so...
  2. doi request reprint Novel myosin heavy chain immunohistochemical double staining developed for the routine diagnostic separation of I, IIA and IIX fibers
    Olayinka Raheem
    Department of Neurology, Neuromuscular Molecular Pathology, University of Tampere, Biokatu 10, Finn Medi 3, 33520 Tampere, Finland
    Acta Neuropathol 119:495-500. 2010
    ..The method provides even more detailed information of fast fiber subtypes and highly atrophic fibers on one single slide...
  3. pmc Mutant (CCTG)n expansion causes abnormal expression of zinc finger protein 9 (ZNF9) in myotonic dystrophy type 2
    Olayinka Raheem
    Neuromuscular Research Unit, University of Tampere and Tampere University Hospital, Tampere, Finland
    Am J Pathol 177:3025-36. 2010
    ..Although toxic RNA effects likely explain overlapping phenotypic manifestations between DM1 and DM2, abnormal ZNF9 levels in DM2 may account for the differences in DM1...
  4. doi request reprint Distinct distal myopathy phenotype caused by VCP gene mutation in a Finnish family
    Johanna Palmio
    Neuromuscular Research Unit, Department of Neurology, University Hospital and University of Tampere, Finland
    Neuromuscul Disord 21:551-5. 2011
    ..VCP needs to be considered in the differential diagnostic work-up in patients with distal myopathy phenotype...
  5. doi request reprint The enigma of 7q36 linked autosomal dominant limb girdle muscular dystrophy
    Satu Sandell
    Department of Neurology, Seinajoki Central Hospital, Seinajoki, Finland
    J Neurol Neurosurg Psychiatry 81:834-9. 2010
    ..The locus has been termed both LGMD1D and 1E, but because of lack of additional families to narrow down the linked region of interest, this disease has remained elusive...