Marcelo O Dietrich

Summary

Publications

  1. ncbi request reprint Megalin mediates the transport of leptin across the blood-CSF barrier
    Marcelo O Dietrich
    Department of Biochemistry, ICBS, University Federal do Rio Grande de Sul, Porto Alegre, RS, Brazil
    Neurobiol Aging 29:902-12. 2008
  2. ncbi request reprint Western style diet impairs entrance of blood-borne insulin-like growth factor-1 into the brain
    Marcelo O Dietrich
    Department of Biochemistry, Universidade Federal do Rio Grande do Sul, Porto Alegre, Brazil
    Neuromolecular Med 9:324-30. 2007
  3. ncbi request reprint Disturbed cross talk between insulin-like growth factor I and AMP-activated protein kinase as a possible cause of vascular dysfunction in the amyloid precursor protein/presenilin 2 mouse model of Alzheimer's disease
    Cristina Lopez-Lopez
    Laboratory of Neuroendocrinology, Cajal Institute, Consejo Superior de Investigaciones Cientificas, 28002 Madrid, Spain
    J Neurosci 27:824-31. 2007

Collaborators

Detail Information

Publications3

  1. ncbi request reprint Megalin mediates the transport of leptin across the blood-CSF barrier
    Marcelo O Dietrich
    Department of Biochemistry, ICBS, University Federal do Rio Grande de Sul, Porto Alegre, RS, Brazil
    Neurobiol Aging 29:902-12. 2008
    ..The present data indicate that leptin represents a novel megalin ligand of importance in the levels and therapeutic actions of leptin into the brain...
  2. ncbi request reprint Western style diet impairs entrance of blood-borne insulin-like growth factor-1 into the brain
    Marcelo O Dietrich
    Department of Biochemistry, Universidade Federal do Rio Grande do Sul, Porto Alegre, Brazil
    Neuromolecular Med 9:324-30. 2007
    ..Reduced brain entrance of circulating IGF-1 elicited by a western-style diet suggests that the higher incidence of brain diseases related to inadequate diets is due in part to diminished neurotrophic support...
  3. ncbi request reprint Disturbed cross talk between insulin-like growth factor I and AMP-activated protein kinase as a possible cause of vascular dysfunction in the amyloid precursor protein/presenilin 2 mouse model of Alzheimer's disease
    Cristina Lopez-Lopez
    Laboratory of Neuroendocrinology, Cajal Institute, Consejo Superior de Investigaciones Cientificas, 28002 Madrid, Spain
    J Neurosci 27:824-31. 2007
    ..Therefore, high IGF-I receptor and phosphoAMPK levels in APP/PS2 brains may reflect imbalanced IGF-I and AMPK angiogenic cross talk that could underlie vascular dysfunction in this model of AD...