Research Topics
| Richard EgelSummaryAffiliation: University of Copenhagen Country: Denmark Publications
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Detail Information
Publications
DNA replication: stalling a fork for imprinting and switchingRichard Egel
Department of Genetics, University of Copenhagen, Øster Farimagsgade 2A, DK 1353 Copenhagen K, Denmark
Curr Biol 14:R915-7. 2004..This imprint has now been firmly characterized as a protected site-specific and strand-specific nick. New work also links the widely conserved Swi1-Swi3 complex to the protection of stalled replication forks in general...
Fission yeast mating-type switching: programmed damage and repairRichard Egel
Department of Genetics, University of Copenhagen, Øster Farimagsgade 2A, DK 1353 Copenhagen K, Denmark
DNA Repair (Amst) 4:525-36. 2005..The versatility of this experimental system has increased further yet by the recent development of a conditional setup, where imprinting and switching can be repressed or derepressed in response to external stimuli...
RecA-DNA filament topology: the overlooked alternative of an unconventional syn-syn duplex intermediateRichard Egel
Institute of Molecular Biology and Physiology, University of Copenhagen, Ole Maaløe Vej 5, DK 2100 Copenhagen Ø, Denmark
DNA Repair (Amst) 6:669-75. 2007....
The fission yeast heterochromatin protein Rik1 is required for telomere clustering during meiosisCreighton T Tuzon
Telomere Biology Laboratory, Cancer Research UK, London, England, UK
J Cell Biol 165:759-65. 2004..Thus, a Swi6-independent heterochromatin function distinct from that at centromeres and the mating-type locus operates at telomeres during sexual differentiation...
A novel type of silencing factor, Clr2, is necessary for transcriptional silencing at various chromosomal locations in the fission yeast Schizosaccharomyces pombePernilla Bjerling
Department of Biosciences, Karolinska Institutet, Sodertorn University College, 141 89 Huddinge, Sweden
Nucleic Acids Res 32:4421-8. 2004..Using chromatin immunoprecipitation, we show that Clr2 is necessary for histone hypoacetylation in the mating-type region, suggesting that Clr2 acts upstream of histone deacetylases to promote transcriptional silencing...
