George D Dalton

Summary

Publications

  1. doi request reprint CB₁ cannabinoid receptors promote maximal FAK catalytic activity by stimulating cooperative signaling between receptor tyrosine kinases and integrins in neuronal cells
    George D Dalton
    Department of Physiology and Pharmacology, Wake Forest University School of Medicine, Winston Salem, NC 27157, USA
    Cell Signal 25:1665-77. 2013
  2. pmc Cannabinoid CB1 receptors transactivate multiple receptor tyrosine kinases and regulate serine/threonine kinases to activate ERK in neuronal cells
    George D Dalton
    Department of Physiology and Pharmacology, Wake Forest University School of Medicine, Winston Salem, NC 27157, USA
    Br J Pharmacol 165:2497-511. 2012
  3. pmc Striatal CB1 and D2 receptors regulate expression of each other, CRIP1A and δ opioid systems
    Lawrence C Blume
    Department of Physiology and Pharmacology, Wake Forest University Health Sciences, Winston Salem, NC 27157, USA
    J Neurochem 124:808-20. 2013

Collaborators

  • Lawrence C Blume
  • David C S Roberts
  • Caroline E Bass
  • Ruoyu Xiao
  • Jasmine M Richardson
  • Dana E Selley
  • Allyn C Howlett
  • Steven R Childers

Detail Information

Publications3

  1. doi request reprint CB₁ cannabinoid receptors promote maximal FAK catalytic activity by stimulating cooperative signaling between receptor tyrosine kinases and integrins in neuronal cells
    George D Dalton
    Department of Physiology and Pharmacology, Wake Forest University School of Medicine, Winston Salem, NC 27157, USA
    Cell Signal 25:1665-77. 2013
    ..Taken together, these studies have identified a complex integrative pathway utilized by CB₁ to stimulate maximal FAK 576/577 Tyr-P in neuronal cells...
  2. pmc Cannabinoid CB1 receptors transactivate multiple receptor tyrosine kinases and regulate serine/threonine kinases to activate ERK in neuronal cells
    George D Dalton
    Department of Physiology and Pharmacology, Wake Forest University School of Medicine, Winston Salem, NC 27157, USA
    Br J Pharmacol 165:2497-511. 2012
    ..We investigated the cellular mechanisms involved in CB(1) receptor-stimulated ERK phosphorylation in a neuronal cell model...
  3. pmc Striatal CB1 and D2 receptors regulate expression of each other, CRIP1A and δ opioid systems
    Lawrence C Blume
    Department of Physiology and Pharmacology, Wake Forest University Health Sciences, Winston Salem, NC 27157, USA
    J Neurochem 124:808-20. 2013
    ....