Melita Salkovic-Petrisic

Summary

Affiliation: University of Zagreb
Country: Croatia

Publications

  1. ncbi request reprint Alzheimer-like changes in protein kinase B and glycogen synthase kinase-3 in rat frontal cortex and hippocampus after damage to the insulin signalling pathway
    Melita Salkovic-Petrisic
    Department of Pharmacology and Croatian Institute for Brain Research, School of Medicine, University of Zagreb, Zagreb, Croatia
    J Neurochem 96:1005-15. 2006
  2. doi request reprint What have we learned from the streptozotocin-induced animal model of sporadic Alzheimer's disease, about the therapeutic strategies in Alzheimer's research
    Melita Salkovic-Petrisic
    Department of Pharmacology and Croatian Institute for Brain Research, School of Medicine, University of Zagreb, Salata 11, HR 10000, Zagreb, Croatia
    J Neural Transm 120:233-52. 2013
  3. doi request reprint Cerebral amyloid angiopathy in streptozotocin rat model of sporadic Alzheimer's disease: a long-term follow up study
    Melita Salkovic-Petrisic
    Department of Pharmacology, School of Medicine, University of Zagreb, Zagreb, Croatia
    J Neural Transm 118:765-72. 2011
  4. doi request reprint Modeling sporadic Alzheimer's disease: the insulin resistant brain state generates multiple long-term morphobiological abnormalities including hyperphosphorylated tau protein and amyloid-beta
    Melita Salkovic-Petrisic
    Department of Pharmacology and Croatian Institute for Brain Research, School of Medicine, University of Zagreb, HR 10 000 Zagreb, Croatia
    J Alzheimers Dis 18:729-50. 2009
  5. ncbi request reprint Usage of the Internet pharmacology resources among European pharmacologists: a preliminary investigation
    M Salkovic-Petrisic
    Department of Pharmacology, School of Medicine, University of Zagreb, Salata 11, HR 10000 Zagreb, POB 916, Croatia
    Fundam Clin Pharmacol 15:55-60. 2001
  6. ncbi request reprint Central administration of alloxan impairs glucose tolerance in rats
    M Salkovic-Petrisic
    Department of Pharmacology, School of Medicine and Croatian Institute for Brain Research, University of Zagreb, Croatia
    J Neural Transm 112:1391-5. 2005
  7. ncbi request reprint Intracerebroventricular administration of betacytotoxics alters expression of brain monoamine transporter genes
    M Salkovic-Petrisic
    Laboratory of Molecular Neuropharmacology, Department of Pharmacology, Medical School, University of Zagreb, Zagreb, Croatia
    J Neural Transm 110:15-29. 2003
  8. doi request reprint Chronic exogenous corticosterone administration generates an insulin-resistant brain state in rats
    Jelena Osmanovic
    Department of Pharmacology and Croatian Institute for Brain Research, School of Medicine, University of Zagreb, Zagreb, Croatia
    Stress 13:123-31. 2010
  9. doi request reprint Botulinum toxin type A reduces pain supersensitivity in experimental diabetic neuropathy: bilateral effect after unilateral injection
    Lidija Bach-Rojecky
    Department of Pharmacology, University of Zagreb School of Pharmacy and Biochemistry, Zagreb, Croatia
    Eur J Pharmacol 633:10-4. 2010
  10. ncbi request reprint Brain insulin system dysfunction in streptozotocin intracerebroventricularly treated rats generates hyperphosphorylated tau protein
    Edna Grunblatt
    Clinical Neurochemistry and National Parkinson Foundation Centre of Excellence Laboratory, Clinic for Psychiatry and Psychotherapy, Bayrische Julius Maximilian University of Würzburg, Wurzburg, Germany
    J Neurochem 101:757-70. 2007

Collaborators

Detail Information

Publications11

  1. ncbi request reprint Alzheimer-like changes in protein kinase B and glycogen synthase kinase-3 in rat frontal cortex and hippocampus after damage to the insulin signalling pathway
    Melita Salkovic-Petrisic
    Department of Pharmacology and Croatian Institute for Brain Research, School of Medicine, University of Zagreb, Zagreb, Croatia
    J Neurochem 96:1005-15. 2006
    ..The IR signalling cascade alteration and its consequences in rats treated with STZ are similar to those found in humans with sporadic Alzheimer's disease, and our results suggest a role for GLUT2 in Alzheimer's pathophysiology...
  2. doi request reprint What have we learned from the streptozotocin-induced animal model of sporadic Alzheimer's disease, about the therapeutic strategies in Alzheimer's research
    Melita Salkovic-Petrisic
    Department of Pharmacology and Croatian Institute for Brain Research, School of Medicine, University of Zagreb, Salata 11, HR 10000, Zagreb, Croatia
    J Neural Transm 120:233-52. 2013
    ....
  3. doi request reprint Cerebral amyloid angiopathy in streptozotocin rat model of sporadic Alzheimer's disease: a long-term follow up study
    Melita Salkovic-Petrisic
    Department of Pharmacology, School of Medicine, University of Zagreb, Zagreb, Croatia
    J Neural Transm 118:765-72. 2011
    ....
  4. doi request reprint Modeling sporadic Alzheimer's disease: the insulin resistant brain state generates multiple long-term morphobiological abnormalities including hyperphosphorylated tau protein and amyloid-beta
    Melita Salkovic-Petrisic
    Department of Pharmacology and Croatian Institute for Brain Research, School of Medicine, University of Zagreb, HR 10 000 Zagreb, Croatia
    J Alzheimers Dis 18:729-50. 2009
    ..A self-propagating process is thus generated, including the increased formation of hyperphosphorylated tau-protein and Abeta as abnormal terminal events in sAD rather than causing the disorder, as elaborated in the review...
  5. ncbi request reprint Usage of the Internet pharmacology resources among European pharmacologists: a preliminary investigation
    M Salkovic-Petrisic
    Department of Pharmacology, School of Medicine, University of Zagreb, Salata 11, HR 10000 Zagreb, POB 916, Croatia
    Fundam Clin Pharmacol 15:55-60. 2001
    ..Also, 35% of participants felt the need for improvement of the Internet pharmacological information evaluation, which should be the goal of pharmacology professional...
  6. ncbi request reprint Central administration of alloxan impairs glucose tolerance in rats
    M Salkovic-Petrisic
    Department of Pharmacology, School of Medicine and Croatian Institute for Brain Research, University of Zagreb, Croatia
    J Neural Transm 112:1391-5. 2005
    ..Centrally administered alloxan seems to have brain related effect on the regulation of peripheral glucose tolerance and insulin secretion...
  7. ncbi request reprint Intracerebroventricular administration of betacytotoxics alters expression of brain monoamine transporter genes
    M Salkovic-Petrisic
    Laboratory of Molecular Neuropharmacology, Department of Pharmacology, Medical School, University of Zagreb, Zagreb, Croatia
    J Neural Transm 110:15-29. 2003
    ..Observed changes may suggest altered response to antidepressants in streptozotocin-icv treated rats, used as an animal model of sporadic Alzheimer's disease...
  8. doi request reprint Chronic exogenous corticosterone administration generates an insulin-resistant brain state in rats
    Jelena Osmanovic
    Department of Pharmacology and Croatian Institute for Brain Research, School of Medicine, University of Zagreb, Zagreb, Croatia
    Stress 13:123-31. 2010
    ..In summary, chronic CST administration affects gene expression in the brain IR signaling cascade and increases tau gene expression, which is associated with reductions in cognition capacity in rats...
  9. doi request reprint Botulinum toxin type A reduces pain supersensitivity in experimental diabetic neuropathy: bilateral effect after unilateral injection
    Lidija Bach-Rojecky
    Department of Pharmacology, University of Zagreb School of Pharmacy and Biochemistry, Zagreb, Croatia
    Eur J Pharmacol 633:10-4. 2010
    ....
  10. ncbi request reprint Brain insulin system dysfunction in streptozotocin intracerebroventricularly treated rats generates hyperphosphorylated tau protein
    Edna Grunblatt
    Clinical Neurochemistry and National Parkinson Foundation Centre of Excellence Laboratory, Clinic for Psychiatry and Psychotherapy, Bayrische Julius Maximilian University of Würzburg, Wurzburg, Germany
    J Neurochem 101:757-70. 2007
    ..Brain insulin system dysfunction is discussed as possible pathological core in the generation of hyperphosphorylated tau protein as a morphological marker of sporadic Alzheimer's disease...
  11. ncbi request reprint Reduced brain antioxidant capacity in rat models of betacytotoxic-induced experimental sporadic Alzheimer's disease and diabetes mellitus
    Ismet Tahirovic
    Department of Chemistry, Faculty of Science, University of Sarajevo, Zmaja od Bosne 35, 71 000, Sarajevo, Bosnia and Herzegovina
    Neurochem Res 32:1709-17. 2007
    ..Only those diabetic animals which demonstrated decreased withdrawal threshold values in comparison with the control non-diabetic animals (C) were considered to have developed the PDN...