H Y Lan

Summary

Affiliation: University of Hong Kong
Country: China

Publications

  1. ncbi Expression of macrophage migration inhibitory factor in human glomerulonephritis
    H Y Lan
    Department of Nephrology, Monash Medical Center, Clayton, Victoria, Australia
    Kidney Int 57:499-509. 2000
  2. ncbi Macrophage migration inhibitory factor expression in human renal allograft rejection
    H Y Lan
    Department of Nephrology, Monash Medical Centre, Clayton, Victoria, Australia
    Transplantation 66:1465-71. 1998
  3. ncbi De Novo renal expression of macrophage migration inhibitory factor during the development of rat crescentic glomerulonephritis
    H Y Lan
    Department of Nephrology, Monash Medical Centre, Clayton, Australia
    Am J Pathol 149:1119-27. 1996
  4. ncbi IL-1 up-regulates osteopontin expression in experimental crescentic glomerulonephritis in the rat
    X Q Yu
    Department of Nephrology, Monash Medical Centre, Monash University, Clayton, Victoria, Australia
    Am J Pathol 154:833-41. 1999
  5. ncbi Tubules are the major site of M-CSF production in experimental kidney disease: correlation with local macrophage proliferation
    N M Isbel
    Department of Nephrology, Monash Medical Centre, Clayton, Victoria, Australia
    Kidney Int 60:614-25. 2001
  6. ncbi Macrophage apoptosis in rat crescentic glomerulonephritis
    H Y Lan
    Department of Nephrology, Monash Medical Centre, Clayton, Victoria, Australia
    Am J Pathol 151:531-8. 1997
  7. ncbi Up-regulation of macrophage colony-stimulating factor (M-CSF) and migration inhibitory factor (MIF) expression and monocyte recruitment during lipid-induced glomerular injury in the exogenous hypercholesterolaemic (ExHC) rat
    K Miyazaki
    Department of Nephrology, Monash Medical Centre, Clayton, Victoria, Australia
    Clin Exp Immunol 108:318-23. 1997
  8. ncbi Up-regulation of macrophage migration inhibitory factor in acute renal allograft rejection in the rat
    F G Brown
    Department of Nephrology, Monash Medical Centre and Department of Medicine, Monash University, Clayton, Victoria, Australia
    Clin Exp Immunol 118:329-36. 1999
  9. ncbi Macrophage and myofibroblast proliferation in remnant kidney: role of angiotensin II
    L L Wu
    Department of Medicine, Austin and Repatriation Medical Center, Victoria, Australia
    Kidney Int Suppl 63:S221-5. 1997
  10. ncbi CD44-mediated neutrophil apoptosis in the rat
    K Takazoe
    Department of Nephrology, Monash Medical Center, Clayton, Victoria, Australia
    Kidney Int 58:1920-30. 2000

Collaborators

Detail Information

Publications11

  1. ncbi Expression of macrophage migration inhibitory factor in human glomerulonephritis
    H Y Lan
    Department of Nephrology, Monash Medical Center, Clayton, Victoria, Australia
    Kidney Int 57:499-509. 2000
    ..We have recently demonstrated that macrophage migration inhibitory factor (MIF) plays a pathogenic role in experimental glomerulonephritis (GN). The aim of the current study was to investigate MIF expression in human GN...
  2. ncbi Macrophage migration inhibitory factor expression in human renal allograft rejection
    H Y Lan
    Department of Nephrology, Monash Medical Centre, Clayton, Victoria, Australia
    Transplantation 66:1465-71. 1998
    ..Despite the long-standing association of MIF with the delayed-type hypersensitivity response, the potential role of MIF in allograft rejection is unknown...
  3. ncbi De Novo renal expression of macrophage migration inhibitory factor during the development of rat crescentic glomerulonephritis
    H Y Lan
    Department of Nephrology, Monash Medical Centre, Clayton, Australia
    Am J Pathol 149:1119-27. 1996
    ..Furthermore, the up-regulation of MIF expression may play an important role in macrophage accumulation and progressive renal injury in rat crescentic glomerulonephritis...
  4. ncbi IL-1 up-regulates osteopontin expression in experimental crescentic glomerulonephritis in the rat
    X Q Yu
    Department of Nephrology, Monash Medical Centre, Monash University, Clayton, Victoria, Australia
    Am J Pathol 154:833-41. 1999
    ....
  5. ncbi Tubules are the major site of M-CSF production in experimental kidney disease: correlation with local macrophage proliferation
    N M Isbel
    Department of Nephrology, Monash Medical Centre, Clayton, Victoria, Australia
    Kidney Int 60:614-25. 2001
    ..These results suggest that M-CSF production within the kidney, particularly by tubular epithelial cells, plays an important role in regulating local macrophage proliferation in experimental kidney disease...
  6. ncbi Macrophage apoptosis in rat crescentic glomerulonephritis
    H Y Lan
    Department of Nephrology, Monash Medical Centre, Clayton, Victoria, Australia
    Am J Pathol 151:531-8. 1997
    ..Indeed, apoptosis may be a central regulator of the progression and resolution of macrophage-mediated tissue injury...
  7. ncbi Up-regulation of macrophage colony-stimulating factor (M-CSF) and migration inhibitory factor (MIF) expression and monocyte recruitment during lipid-induced glomerular injury in the exogenous hypercholesterolaemic (ExHC) rat
    K Miyazaki
    Department of Nephrology, Monash Medical Centre, Clayton, Victoria, Australia
    Clin Exp Immunol 108:318-23. 1997
    ..This is the first study to implicate local synthesis of MIF in the pathogenesis of lipid-induced lesions...
  8. ncbi Up-regulation of macrophage migration inhibitory factor in acute renal allograft rejection in the rat
    F G Brown
    Department of Nephrology, Monash Medical Centre and Department of Medicine, Monash University, Clayton, Victoria, Australia
    Clin Exp Immunol 118:329-36. 1999
    ..These results suggest that MIF may play an important role in the cellular immune response mediating acute allograft rejection...
  9. ncbi Macrophage and myofibroblast proliferation in remnant kidney: role of angiotensin II
    L L Wu
    Department of Medicine, Austin and Repatriation Medical Center, Victoria, Australia
    Kidney Int Suppl 63:S221-5. 1997
    ..In conclusion, inhibition of local macrophage and myofibroblast proliferation may be an important mechanism by which Ang II blockade attenuated renal injury following renal ablation...
  10. ncbi CD44-mediated neutrophil apoptosis in the rat
    K Takazoe
    Department of Nephrology, Monash Medical Center, Clayton, Victoria, Australia
    Kidney Int 58:1920-30. 2000
    ..Apoptosis is an important mechanism by which neutrophils are removed from sites of inflammation, including the kidney. This study investigated whether ligation of the cell-surface adhesion molecule, CD44, can trigger neutrophil apoptosis...
  11. ncbi Immunoregulatory activity in adult rat testicular interstitial fluid: relationship with intratesticular CD8+ lymphocytes following treatment with ethane dimethane sulfonate and testosterone implants
    M P Hedger
    Institute of Reproduction and Development, Monash University, Clayton, Victoria, Australia
    Biol Reprod 58:935-42. 1998
    ....