Juan Francisco Santibanez

Summary

Affiliation: Universidad de Chile
Country: Chile

Publications

  1. ncbi TGF-beta(1) and Smad4 overexpression induce a less invasive phenotype in highly invasive spindle carcinoma cells
    Juan Francisco Santibanez
    Laboratorio de Biologia Celular, INTA, Universidad de Chile, Casilla 138, Santiago 11, Chile
    FEBS Lett 520:171-6. 2002
  2. ncbi Transforming growth factor-beta1 modulates matrix metalloproteinase-9 production through the Ras/MAPK signaling pathway in transformed keratinocytes
    Juan Francisco Santibanez
    Laboratorio de Biologia Celular, INTA, Universidad de Chile Casilla 138, Santiago, Chile
    Biochem Biophys Res Commun 296:267-73. 2002
  3. ncbi Cyclic AMP inhibits TGFbeta1-induced cell-scattering and invasiveness in murine-transformed keratinocytes
    Juan F Santibanez
    Laboratorio de Biología Celular INTA, Universidad de Chile, Santiago, Chile
    Int J Cancer 107:715-20. 2003
  4. ncbi EGF receptor transactivation by urokinase receptor stimulus through a mechanism involving Src and matrix metalloproteinases
    Javier Guerrero
    Laboratorio de Biologia Celular, INTA, Universidad de Chile, Santiago 11, Chile
    Exp Cell Res 292:201-8. 2004

Collaborators

Detail Information

Publications4

  1. ncbi TGF-beta(1) and Smad4 overexpression induce a less invasive phenotype in highly invasive spindle carcinoma cells
    Juan Francisco Santibanez
    Laboratorio de Biologia Celular, INTA, Universidad de Chile, Casilla 138, Santiago 11, Chile
    FEBS Lett 520:171-6. 2002
    ..This activation was abolished with a dominant-negative Smad4 construct. These results lead us to conclude that both TGF-beta(1) and Smad4 overexpression reduce the invasive potential of Car C cells, probably via the Smad pathway...
  2. ncbi Transforming growth factor-beta1 modulates matrix metalloproteinase-9 production through the Ras/MAPK signaling pathway in transformed keratinocytes
    Juan Francisco Santibanez
    Laboratorio de Biologia Celular, INTA, Universidad de Chile Casilla 138, Santiago, Chile
    Biochem Biophys Res Commun 296:267-73. 2002
    ....
  3. ncbi Cyclic AMP inhibits TGFbeta1-induced cell-scattering and invasiveness in murine-transformed keratinocytes
    Juan F Santibanez
    Laboratorio de Biología Celular INTA, Universidad de Chile, Santiago, Chile
    Int J Cancer 107:715-20. 2003
    ..The latter responses seem to depend on the activity of protein kinase A, as demonstrated by the activation of the Ras-ERK pathway by specific protein kinase A inhibitors...
  4. ncbi EGF receptor transactivation by urokinase receptor stimulus through a mechanism involving Src and matrix metalloproteinases
    Javier Guerrero
    Laboratorio de Biologia Celular, INTA, Universidad de Chile, Santiago 11, Chile
    Exp Cell Res 292:201-8. 2004
    ....