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Genomes and Genes | D M JuriloffSummaryAffiliation: University of British Columbia Country: Canada Publications
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Publications
Investigations of the genomic region that contains the clf1 mutation, a causal gene in multifactorial cleft lip and palate in miceDiana M Juriloff
Department of Medical Genetics, University of British Columbia, Vancouver, British Columbia, Canada
Birth Defects Res A Clin Mol Teratol 73:103-13. 2005..A potentially homologous gene, clf1 on distal chromosome 11, is part of the digenic cause of the 10-30% CL(P) in the A/WySn mouse strain. Here we report our progress toward identifying the clf1 mutation...
Mouse models for neural tube closure defectsD M Juriloff
Department of Medical Genetics, University of British Columbia, 6174 University Boulevard, Vancouver, British Columbia, Canada
Hum Mol Genet 9:993-1000. 2000....
The open-eyelid mutation, lidgap-Gates, is an eight-exon deletion in the mouse Map3k1 geneDiana M Juriloff
Department of Medical Genetics, University of British Columbia, 6174 University Boulevard, Vancouver, BC, Canada V6T 1Z3
Genomics 85:139-42. 2005..The lg(Ga) mutation is consistent with a pattern suggesting that the waves of mutation in BALB/cGa and its descendants tend to be large deletions or ETn insertions, whose elevated rate of occurrence is due to an unknown mechanism...
A digenic cause of cleft lip in A-strain mice and definition of candidate genes for the two lociDiana M Juriloff
Department of Medical Genetics, University of British Columbia, Vancouver, British ColumbiaV6T 1Z3, Canada
Birth Defects Res A Clin Mol Teratol 70:509-18. 2004..Our previous studies mapped two loci: clf1 on Chr11 and clf2 on Chr13--with a strong genetic maternal effect on the level of risk. Here we test the hypothesis that CL(P) is digenic and identify candidate genes for clf1 and clf2...
Multifactorial genetics of exencephaly in SELH/Bc miceD M Juriloff
Department of Medical Genetics, University of British Columbia, Vancouver, BC, V6T 1Z3, Canada
Teratology 64:189-200. 2001..The SELH/Bc mouse strain has 10-30% exencephaly and is an animal model for human neural tube closure defects. This study examined the number of causative genes, their dominance relationships, and linkage map positions...
Unravelling the complex genetics of cleft lip in the mouse modelD M Juriloff
Department of Medical Genetics, University of British Columbia, 6174 University Boulevard, Vancouver, B C, V6T 1Z3, Canada
Mamm Genome 12:426-35. 2001..Human homologs of clf1 and clf2 are expected to be on 17q and 5q/9q...
Gaping lids, gp, a mutation on centromeric chromosome 11 that causes defective eyelid development in miceD M Juriloff
Department of Medical Genetics, University of British Columbia, 6174 University Boulevard, Vancouver, British Columbia V6T 1Z3, Canada
Mamm Genome 11:440-7. 2000..The gaping lids mutation identifies a previously unknown locus on proximal Chromosome (Chr) 11 that has a strong role in fetal eyelid growth...
Wnt9b is the mutated gene involved in multifactorial nonsyndromic cleft lip with or without cleft palate in A/WySn mice, as confirmed by a genetic complementation testDiana M Juriloff
Department of Medical Genetics, University of British Columbia, Vancouver, British Columbia, Canada
Birth Defects Res A Clin Mol Teratol 76:574-9. 2006..Independently a recessive knockout mutation of Wnt9b (Wnt9b-) was reported to cause a lethal syndrome that includes some CLP...
Mouse genetic models of cleft lip with or without cleft palateDiana M Juriloff
Department of Medical Genetics, University of British Columbia, Vancouver, British Columbia, Canada
Birth Defects Res A Clin Mol Teratol 82:63-77. 2008....
Nonallelic noncomplementation models in mice: the first arch and lidgap-Gates mutationsM J Harris
Department of Medical Genetics, University of British Columbia, Vancouver, Canada
Genome 41:789-96. 1998..Based on our data on open eyelids at birth, we suggest that false allelism may be common in mammalian birth defects that result from failure to meet developmental thresholds, even when the "causal" mutations are Mendelian...
The major locus for multifactorial nonsyndromic cleft lip maps to mouse chromosome 11D M Juriloff
Department of Medical Genetics, University of British Columbia, Vancouver, Canada
Mamm Genome 6:63-9. 1995..This locus, here named clf1 (cleft lip) maps to Chromosome (Chr) 11 to a region having linkage homology with human 17q21-24, supporting reports of association of human CL(P) with the retinoic acid receptor alpha (RARA) locus...
The first arch (Far) mutation in mice recombines with Hoxd and MdkR T Dreger
Department of Medical Genetics, University of British Columbia, Vancouver, Canada
Mamm Genome 6:662-3. 1995
Three spontaneous mutations at the albino locus in SELH/Bc miceD M Juriloff
Department of Medical Genetics, University of British Columbia, Vancouver, Canada
Genome 37:190-7. 1994..SELH/Bc mice may provide an animal model for the study of mechanisms underlying genetic instability...
The lidgap-Gates (lgGa) mutation for open eyelids at birth maps to mouse chromosome 13D M Juriloff
Department of Medical Genetics, University of British Columbia, Canada
Mamm Genome 7:403-7. 1996..Evidence is also presented for a common unlinked recessive suppressor of the open eyelids trait caused by lgGa...
Current status of genetic linkage studies of a major gene that causes CL(P) in mice: exclusion mapD M Juriloff
Department of Medical Genetics, University of British Columbia, Vancouver, Canada
J Craniofac Genet Dev Biol 13:223-9. 1993..More than 40% of the mouse genome has been swept. Some candidate regions include mouse chromosomes 12, 18, and 19, and candidate loci on other chromosomes should include Rara, Tgfa, Bmp2b1, and Igf-2...
Disruption of pattern formation in palatal rugae in fetal mice heterozygous for First arch (Far)M J Harris
Department of Medical Genetics, University of British Columbia, Vancouver, Canada
J Craniofac Genet Dev Biol 10:363-71. 1990..The First arch mutation, which in heterozygotes disrupts pattern formation in both palatal rugae and maxillary vibrissae, can be used to study genetic control of pattern formation in mammalian embryos...
Genetic analysis of the construction of the AEJ.A congenic strain indicates that nonsyndromic CL(P) in the mouse is caused by two loci with epistatic interactionD M Juriloff
Department of Medical Genetics, University of British Columbia, Vancouver, Canada
J Craniofac Genet Dev Biol 15:1-12. 1995....
Genetic landmarks for defects in mouse neural tube closureM J Harris
Department of Medical Genetics, University of British Columbia, Vancouver, Canada
Teratology 56:177-87. 1997....
Mapping the mouse craniofacial mutation first arch (Far) to chromosome 2D M Juriloff
Department of Medical Genetics, University of British Columbia, Vancouver, B C Canada
J Hered 82:402-5. 1991..It may therefore be significant that, in mapping near Ulnaless, Far also maps in the vicinity of the Hox-4 gene cluster...
Studies of a spontaneous lethal mutation at the albino locus in SELH/Bc miceD M Juriloff
Department of Medical Genetics, University of British Columbia, Vancouver, Canada
Genome 35:342-6. 1992..This lethal albino mutation that appears to be a postmeiotic deletion should be useful in the search for the mechanism of mutagenesis in SELH/Bc mice. It may also be useful in mapping essential genes in the c-locus region...
Mouse mutants with neural tube closure defects and their role in understanding human neural tube defectsMuriel J Harris
Department of Medical Genetics, University of British Columbia, Vancouver, British Columbia, Canada
Birth Defects Res A Clin Mol Teratol 79:187-210. 2007..The number of mouse mutants and strains with neural tube closure defects (NTDs) now exceeds 190, including 155 involving known genes, 33 with unidentified genes, and eight "multifactorial" strains...
Maternal diet alters exencephaly frequency in SELH/Bc strain mouse embryosMuriel J Harris
Department of Medical Genetics, University of British Columbia, Vancouver, Canada
Birth Defects Res A Clin Mol Teratol 73:532-40. 2005..The SELH/Bc mouse inbred strain, with a high frequency of nonsyndromic, genetically-multifactorial exencephaly, is a model for human cranial neural tube defects (NTDs). Maternal diet affects risk of human NTDs...
An update to the list of mouse mutants with neural tube closure defects and advances toward a complete genetic perspective of neural tube closureMuriel J Harris
Department of Medical Genetics, University of British Columbia, Vancouver, British Columbia, Canada
Birth Defects Res A Clin Mol Teratol 88:653-69. 2010....
Whiskers amiss, a new vibrissae and hair mutation near the Krt1 cluster on mouse chromosome 11L A Taylor
Department of Medical Genetics, University of British Columbia, Vancouver, Canada
Mamm Genome 11:255-9. 2000..The SELH/Bc strain background appears to have an unusually high rate of spontaneous mutation; wam is the sixth mutation to be described...
Mini-review: toward understanding mechanisms of genetic neural tube defects in miceM J Harris
Department of Medical Genetics, University of British Columbia, Vancouver, British Columbia, Canada V6T 1Z3
Teratology 60:292-305. 1999....
Is Far a Hox mutation?D M Juriloff
Department of Medical Genetics, University of British Columbia, Vancouver, Canada
J Craniofac Genet Dev Biol 12:119-29. 1992..abstract truncated at 400 words)..
Structure and expression of mobile ETnII retroelements and their coding-competent MusD relatives in the mouseCorinna Baust
Terry Fox Laboratory, B C Cancer Agency, Department of Medical Genetics, University of British Columbia, Vancouver, British Columbia, Canada
J Virol 77:11448-58. 2003..We also show that ETnII is expressed up to 30-fold more than MusD, which could explain why only ETnII, but not MusD, elements have been positively identified as new insertions...
Accelerated embryonic development associated with increased risk of neural tube defects induced by maternal diet in offspring of SELH/Bc miceKatharine L Stoate
Department of Medical Genetics, University of British Columbia, Vancouver, British Columbia, Canada
Birth Defects Res A Clin Mol Teratol 82:720-7. 2008..Maternal diets affect SELH/Bc exencephaly rates: 25-35% on Purina Diet 5015 versus 5-10% on Purina Diet 5001. We hypothesized that in SELH/Bc, the diets affect maternal blood glucose and embryonic developmental rate...
