Research Topics
Genomes and Genes | Carlos Fernandez-PatronSummaryAffiliation: University of Alberta Country: Canada Publications
| Collaborators
|
Detail Information
Publications
Tumor necrosis factor-alpha-converting enzyme is a key regulator of agonist-induced cardiac hypertrophy and fibrosisXiang Wang
Department of Biochemistry, University of Alberta, Edmonton, Alberta, Canada
Hypertension 54:575-82. 2009..Targeting TACE has potential therapeutic importance for modulating agonist-induced cardiac remodeling...
MMP-2 mediates angiotensin II-induced hypertension under the transcriptional control of MMP-7 and TACEJeffrey Odenbach
Department of Biochemistry, School of Molecular and Systems Medicine, University of Alberta, Edmonton, Alberta, Canada
Hypertension 57:123-30. 2011..This suggests a functional specialization of MMP-2 in agonist-induced cardiovascular disease development that has potential implications for the design of metalloproteinase-based therapeutic strategies...
Agonist-induced activation of matrix metalloproteinase-7 promotes vasoconstriction through the epidermal growth factor-receptor pathwayLi Hao
Department of Biochemistry, University of Alberta, Edmonton, Alberta, Canada
Circ Res 94:68-76. 2004..Because this mechanism is triggered by agonists typically overexpressed in hypertension, its blockade may have therapeutic potential for simultaneously inhibiting pathological vasoconstriction and growth in hypertensive disorders...
Maintenance of adrenergic vascular tone by MMP transactivation of the EGFR requires PI3K and mitochondrial ATP synthesisPrabhakara Reddy Nagareddy
Department of Biochemistry, Faculty of Medicine and Dentistry, University of Alberta, Alberta, Canada T6G 2H7
Cardiovasc Res 84:368-77. 2009..In the present study, we examined whether MMP modulation of vascular tone involves phosphoinositide 3-kinase (PI3K) and mitochondrial ATP synthesis...
Matrix metalloproteinase-7 and ADAM-12 (a disintegrin and metalloproteinase-12) define a signaling axis in agonist-induced hypertension and cardiac hypertrophyXiang Wang
Department of Biochemistry, University of Alberta, Edmonton, Alberta, Canada
Circulation 119:2480-9. 2009..Here, we report that matrix metalloproteinase-7 (MMP-7) and a disintegrin and metalloproteinase-12 (ADAM-12) form a novel signaling axis in these processes...
Mitochondrial dysfunction in the hypertensive rat brain: respiratory complexes exhibit assembly defects in hypertensionAna Lopez-Campistrous
Department of Biochemistry, Institute for Biomolecular Design, University of Alberta, Edmonton, Alberta, Canada
Hypertension 51:412-9. 2008..This mitochondrial dysfunction localizes to the brain stem and is, therefore, likely to contribute to the development, as well as to pathophysiological complications, of hypertension...
Therapeutic potential of the epidermal growth factor receptor transactivation in hypertension: a convergent signaling pathway of vascular tone, oxidative stress, and hypertrophic growth downstream of vasoactive G-protein-coupled receptors?Carlos Fernandez-Patron
Department of Biochemistry, University of Alberta, Edmonton, AB T6G 2H7, Canada
Can J Physiol Pharmacol 85:97-104. 2007..If this hypothesis is proven generally correct, then transactivation blockers have general therapeutic potential in hypertension regardless of the causative agonist...
Vascular responses to alpha1-adrenergic receptors in small rat mesenteric arteries depend on mitochondrial reactive oxygen speciesLi Hao
Department of Biochemistry, University of Alberta, Edmonton, Alberta T6G 2H7, Canada
Arterioscler Thromb Vasc Biol 26:819-25. 2006....
Insights into the activity, differential expression, mutual regulation, and functions of matrix metalloproteinases and a disintegrin and metalloproteinases in hypertension and cardiac diseaseEvan Berry
Department of Biochemistry, School of Translational Medicine, University of Alberta, Edmonton, Alta, Canada
J Vasc Res 50:52-68. 2013..The emerging links between metalloproteinases and lipids are intriguing and suggest new therapeutic targets in hypertensive cardiac disease...
