Romeo Ricci

Summary

Country: Canada

Publications

  1. pmc c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity
    Ewa Stepniak
    Research Institute of Molecular Pathology IMP, A 1030 Vienna, Austria
    Genes Dev 20:2306-14. 2006
  2. ncbi Requirement of JNK2 for scavenger receptor A-mediated foam cell formation in atherogenesis
    Romeo Ricci
    Cardiovascular Research, Institute of Physiology, and Division of Cardiology, University Hospital Zurich, CH 8057 Zurich, Switzerland
    Science 306:1558-61. 2004
  3. ncbi Molecular imaging of atherosclerotic plaques using a human antibody against the extra-domain B of fibronectin
    Christian M Matter
    Cardiovascular Research, Institute of Physiology, University of Zurich and Cardiovascular Center, Zurich University Hospital, Switzerland
    Circ Res 95:1225-33. 2004
  4. ncbi Stage-specific control of neural crest stem cell proliferation by the small rho GTPases Cdc42 and Rac1
    Sebastian Fuchs
    Institute of Anatomy, University of Zurich, CH 8057 Zurich, Switzerland
    Cell Stem Cell 4:236-47. 2009
  5. ncbi Impaired heart contractility in Apelin gene-deficient mice associated with aging and pressure overload
    Keiji Kuba
    Institute of Molecular Biotechnology, Austrian Academy of Sciences, Vienna, Austria
    Circ Res 101:e32-42. 2007
  6. ncbi Regression of abdominal aortic aneurysm by inhibition of c-Jun N-terminal kinase
    Koichi Yoshimura
    Department of Molecular Cardiovascular Biology, Yamaguchi University School of Medicine, 1 1 1 Minami kogushi, Ube, Yamaguchi 755 8505, Japan
    Nat Med 11:1330-8. 2005
  7. pmc JNK1 is not essential for TNF-mediated joint disease
    Marcus Koller
    Department of Internal Medicine III, Division of Rheumatology, Medical University of Vienna, Austria
    Arthritis Res Ther 7:R166-73. 2005
  8. pmc Loss of the anaphase-promoting complex in quiescent cells causes unscheduled hepatocyte proliferation
    Karin G Wirth
    Research Institute of Molecular Pathology IMP, A 1030 Vienna, Austria
    Genes Dev 18:88-98. 2004
  9. pmc Distinct functions of junD in cardiac hypertrophy and heart failure
    Romeo Ricci
    Institute of Molecular Pathology, A 1030 Vienna, Austria
    Genes Dev 19:208-13. 2005
  10. ncbi Dendritic cell-induced autoimmune heart failure requires cooperation between adaptive and innate immunity
    Urs Eriksson
    Institute of Molecular Biotechnology of the Austrian Academy of Sciences, Dr Bohrgasse 7, A 1030 Vienna, Austria
    Nat Med 9:1484-90. 2003

Collaborators

Detail Information

Publications13

  1. pmc c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity
    Ewa Stepniak
    Research Institute of Molecular Pathology IMP, A 1030 Vienna, Austria
    Genes Dev 20:2306-14. 2006
    ..These data demonstrate that c-Jun/AP-1 regulates liver regeneration through a novel molecular pathway that involves p53, p21, and the stress kinase p38alpha...
  2. ncbi Requirement of JNK2 for scavenger receptor A-mediated foam cell formation in atherogenesis
    Romeo Ricci
    Cardiovascular Research, Institute of Physiology, and Division of Cardiology, University Hospital Zurich, CH 8057 Zurich, Switzerland
    Science 306:1558-61. 2004
    ..Thus, JNK2-dependent phosphorylation of SR-A promotes uptake of lipids in macrophages, thereby regulating foam cell formation, a critical step in atherogenesis...
  3. ncbi Molecular imaging of atherosclerotic plaques using a human antibody against the extra-domain B of fibronectin
    Christian M Matter
    Cardiovascular Research, Institute of Physiology, University of Zurich and Cardiovascular Center, Zurich University Hospital, Switzerland
    Circ Res 95:1225-33. 2004
    ..Thus, our findings may set the stage for antibody-based molecular imaging of atherosclerotic plaques in the intact organism...
  4. ncbi Stage-specific control of neural crest stem cell proliferation by the small rho GTPases Cdc42 and Rac1
    Sebastian Fuchs
    Institute of Anatomy, University of Zurich, CH 8057 Zurich, Switzerland
    Cell Stem Cell 4:236-47. 2009
    ..Thus, our data reveal distinct mechanisms for growth control of NCSCs from different developmental stages...
  5. ncbi Impaired heart contractility in Apelin gene-deficient mice associated with aging and pressure overload
    Keiji Kuba
    Institute of Molecular Biotechnology, Austrian Academy of Sciences, Vienna, Austria
    Circ Res 101:e32-42. 2007
    ..These genetic data show that the endogenous peptide Apelin is crucial to maintain cardiac contractility in pressure overload and aging...
  6. ncbi Regression of abdominal aortic aneurysm by inhibition of c-Jun N-terminal kinase
    Koichi Yoshimura
    Department of Molecular Cardiovascular Biology, Yamaguchi University School of Medicine, 1 1 1 Minami kogushi, Ube, Yamaguchi 755 8505, Japan
    Nat Med 11:1330-8. 2005
    ..Thus, JNK promotes abnormal extracellular matrix metabolism in the tissue of AAA and may represent a therapeutic target...
  7. pmc JNK1 is not essential for TNF-mediated joint disease
    Marcus Koller
    Department of Internal Medicine III, Division of Rheumatology, Medical University of Vienna, Austria
    Arthritis Res Ther 7:R166-73. 2005
    ..Thus, JNK1 activation does not seem to be essential for TNF-mediated arthritis...
  8. pmc Loss of the anaphase-promoting complex in quiescent cells causes unscheduled hepatocyte proliferation
    Karin G Wirth
    Research Institute of Molecular Pathology IMP, A 1030 Vienna, Austria
    Genes Dev 18:88-98. 2004
    ..Re-entry into the cell cycle either occurred without any proliferative stimulus or could be easily induced. We demonstrate that the APC has an additional function to prevent hepatocytes from unscheduled re-entry into the cell cycle...
  9. pmc Distinct functions of junD in cardiac hypertrophy and heart failure
    Romeo Ricci
    Institute of Molecular Pathology, A 1030 Vienna, Austria
    Genes Dev 19:208-13. 2005
    ..These data suggest that junD promotes both adaptive-protective and maladaptive hypertrophy in heart, depending on its expression levels...
  10. ncbi Dendritic cell-induced autoimmune heart failure requires cooperation between adaptive and innate immunity
    Urs Eriksson
    Institute of Molecular Biotechnology of the Austrian Academy of Sciences, Dr Bohrgasse 7, A 1030 Vienna, Austria
    Nat Med 9:1484-90. 2003
    ..DC-induced myocarditis provides a unifying theory as to how tissue damage and activation of TLRs during infection can induce autoimmunity, relapses and cardiomyopathy...
  11. pmc Separase: a universal trigger for sister chromatid disjunction but not chromosome cycle progression
    Karin G Wirth
    Research Institute of Molecular Pathology, A 1030 Vienna, Austria
    J Cell Biol 172:847-60. 2006
    ..Separase depletion in bone marrow causes aplasia and the presumed death of hematopoietic cells other than erythrocytes. Destruction of sister chromatid cohesion by Separase may be a universal feature of mitosis in eukaryotic cells...
  12. pmc Osteoclasts are essential for TNF-alpha-mediated joint destruction
    Kurt Redlich
    Department of Internal Medicine III, Division of Rheumatology, University of Vienna, Vienna, Austria
    J Clin Invest 110:1419-27. 2002
    ..Therefore, in addition to the use of anti-inflammatory therapies, osteoclast inhibition could be beneficial for the treatment of rheumatoid arthritis...
  13. ncbi JunB can substitute for Jun in mouse development and cell proliferation
    Emmanuelle Passegue
    Research Institute of Molecular Pathology, Dr Bohr Gasse 7, A 1030 Vienna, Austria
    Nat Genet 30:158-66. 2002
    ....