Przemyslaw Blyszczuk

Summary

Publications

  1. pmc Profibrotic potential of prominin-1+ epithelial progenitor cells in pulmonary fibrosis
    Przemyslaw Blyszczuk
    Cardiovascular Research and Zürich Center for Integrative Human Physiology, Institute of Physiology, University of Zurich, Winterthurerstr, 190, CH 8057 Zurich, Switzerland
    Respir Res 12:126. 2011
  2. doi request reprint Innate signaling promotes formation of regulatory nitric oxide-producing dendritic cells limiting T-cell expansion in experimental autoimmune myocarditis
    Gabriela Kania
    Cardioimmunology, Cardiovascular Research, Institute of Physiology, University of Zurich, Winterthurerstrasse 190, CH 8057 Zurich, Switzerland
    Circulation 127:2285-94. 2013
  3. doi request reprint Nitric oxide synthase 2 is required for conversion of pro-fibrogenic inflammatory CD133(+) progenitors into F4/80(+) macrophages in experimental autoimmune myocarditis
    Przemyslaw Blyszczuk
    Cardioimmunology, Cardiovascular Research and Zürich Center for Integrative Human Physiology, Institute of Physiology, University of Zurich, Winterthurerstr 190, Zurich CH 8057, Switzerland
    Cardiovasc Res 97:219-29. 2013
  4. doi request reprint Heart-infiltrating prominin-1+/CD133+ progenitor cells represent the cellular source of transforming growth factor beta-mediated cardiac fibrosis in experimental autoimmune myocarditis
    Gabriela Kania
    Division of Cardioimmunology, Institute of Physiology, University of Zurich, Winterthurerstrasse 190, CH 8057 Zurich, Switzerland
    Circ Res 105:462-70. 2009
  5. doi request reprint GM-CSF promotes inflammatory dendritic cell formation but does not contribute to disease progression in experimental autoimmune myocarditis
    Przemyslaw Blyszczuk
    University of Zurich, CH 8057 Zurich, Switzerland
    Biochim Biophys Acta 1833:934-44. 2013
  6. doi request reprint Mechanisms of cardiac fibrosis in inflammatory heart disease
    Gabriela Kania
    Cardioimmunology, Cardiovascular Research Department, Institute of Physiology, University of Zurich, Winterthurerstrasse 190, CH 8057 Zurich, Switzerland
    Trends Cardiovasc Med 19:247-52. 2009
  7. ncbi request reprint Novel therapeutic options in inflammatory cardiomyopathy
    Gabriela Kania
    Cardioimmunology, Cardiovascular Research, Institute of Physiology, ZÃ rich, Switzerland
    Swiss Med Wkly 143:w13841. 2013
  8. doi request reprint Abnormal high-density lipoprotein induces endothelial dysfunction via activation of Toll-like receptor-2
    Thimoteus Speer
    Cardiovascular Center, Cardiology, University Hospital Zurich and Cardiovascular Research, Institute of Physiology, University of Zurich, Zurich, Switzerland
    Immunity 38:754-68. 2013

Collaborators

Detail Information

Publications8

  1. pmc Profibrotic potential of prominin-1+ epithelial progenitor cells in pulmonary fibrosis
    Przemyslaw Blyszczuk
    Cardiovascular Research and Zürich Center for Integrative Human Physiology, Institute of Physiology, University of Zurich, Winterthurerstr, 190, CH 8057 Zurich, Switzerland
    Respir Res 12:126. 2011
    ..Herein, we addressed the contribution of lung microenvironment and prominin-1(+) bone marrow-derived epithelial progenitor cells in the mouse model of bleomycin-induced experimental pulmonary fibrosis...
  2. doi request reprint Innate signaling promotes formation of regulatory nitric oxide-producing dendritic cells limiting T-cell expansion in experimental autoimmune myocarditis
    Gabriela Kania
    Cardioimmunology, Cardiovascular Research, Institute of Physiology, University of Zurich, Winterthurerstrasse 190, CH 8057 Zurich, Switzerland
    Circulation 127:2285-94. 2013
    ..Mechanisms that counterregulate exaggerated heart-specific autoimmunity are poorly understood...
  3. doi request reprint Nitric oxide synthase 2 is required for conversion of pro-fibrogenic inflammatory CD133(+) progenitors into F4/80(+) macrophages in experimental autoimmune myocarditis
    Przemyslaw Blyszczuk
    Cardioimmunology, Cardiovascular Research and Zürich Center for Integrative Human Physiology, Institute of Physiology, University of Zurich, Winterthurerstr 190, Zurich CH 8057, Switzerland
    Cardiovasc Res 97:219-29. 2013
    ....
  4. doi request reprint Heart-infiltrating prominin-1+/CD133+ progenitor cells represent the cellular source of transforming growth factor beta-mediated cardiac fibrosis in experimental autoimmune myocarditis
    Gabriela Kania
    Division of Cardioimmunology, Institute of Physiology, University of Zurich, Winterthurerstrasse 190, CH 8057 Zurich, Switzerland
    Circ Res 105:462-70. 2009
    ..Myocardial fibrosis is a hallmark of inflammation-triggered end-stage heart disease, a common cause of heart failure in young patients...
  5. doi request reprint GM-CSF promotes inflammatory dendritic cell formation but does not contribute to disease progression in experimental autoimmune myocarditis
    Przemyslaw Blyszczuk
    University of Zurich, CH 8057 Zurich, Switzerland
    Biochim Biophys Acta 1833:934-44. 2013
    ..Its specific role in the progression of myocarditis into end stage heart failure is not known...
  6. doi request reprint Mechanisms of cardiac fibrosis in inflammatory heart disease
    Gabriela Kania
    Cardioimmunology, Cardiovascular Research Department, Institute of Physiology, University of Zurich, Winterthurerstrasse 190, CH 8057 Zurich, Switzerland
    Trends Cardiovasc Med 19:247-52. 2009
    ..In this minireview, we provide an update on these mechanisms and discuss their significance in pathologic remodeling and heart failure progression after myocarditis...
  7. ncbi request reprint Novel therapeutic options in inflammatory cardiomyopathy
    Gabriela Kania
    Cardioimmunology, Cardiovascular Research, Institute of Physiology, ZÃ rich, Switzerland
    Swiss Med Wkly 143:w13841. 2013
    ..In this review we update our knowledge on disease mechanisms, summarise current clinical approaches for iDCM patients, and discuss future therapeutic options. ..
  8. doi request reprint Abnormal high-density lipoprotein induces endothelial dysfunction via activation of Toll-like receptor-2
    Thimoteus Speer
    Cardiovascular Center, Cardiology, University Hospital Zurich and Cardiovascular Research, Institute of Physiology, University of Zurich, Zurich, Switzerland
    Immunity 38:754-68. 2013
    ....