Iwona Wlodarska

Summary

Affiliation: Katholieke Universiteit Leuven
Country: Belgium

Publications

  1. pmc Telomeric IGH losses detectable by fluorescence in situ hybridization in chronic lymphocytic leukemia reflect somatic VH recombination events
    Iwona Wlodarska
    Center for Human Genetics, Catholoc University Leuven, Leuven, Belgium
    J Mol Diagn 9:47-54. 2007
  2. ncbi request reprint Translocations targeting CCND2, CCND3, and MYCN do occur in t(11;14)-negative mantle cell lymphomas
    Iwona Wlodarska
    Center for Human Genetics, Katholieke Universiteit Leuven, Leuven, Belgium
    Blood 111:5683-90. 2008
  3. ncbi request reprint Variant t(2;11)(p11;q13) associated with the IgK-CCND1 rearrangement is a recurrent translocation in leukemic small-cell B-non-Hodgkin lymphoma
    I Wlodarska
    Center for Human Genetics, Katholieke Universiteit Leuven, Belgium
    Leukemia 18:1705-10. 2004
  4. ncbi request reprint FOXP1, a gene highly expressed in a subset of diffuse large B-cell lymphoma, is recurrently targeted by genomic aberrations
    I Wlodarska
    Center for Human Genetics, Katholieke Universiteit Leuven, Leuven, Belgium
    Leukemia 19:1299-305. 2005
  5. ncbi request reprint Forkhead box protein P1 expression in mucosa-associated lymphoid tissue lymphomas predicts poor prognosis and transformation to diffuse large B-cell lymphoma
    Xavier Sagaert
    Department of Morphology and Molecular Pathology, and the Centre for Human Genetics, Catholic University Leuven, Leuven, Belgium
    J Clin Oncol 24:2490-7. 2006
  6. pmc ALK-positive large B-cell lymphomas with cryptic SEC31A-ALK and NPM1-ALK fusions
    Katrien Van Roosbroeck
    Center for Human Genetics, K U Leuven, Gasthuisberg, Herestraat 49, Box 602, B 3000 Leuven, Belgium
    Haematologica 95:509-13. 2010
  7. ncbi request reprint Fusion of EML1 to ABL1 in T-cell acute lymphoblastic leukemia with cryptic t(9;14)(q34;q32)
    Kim De Keersmaecker
    Department of Human Genetics, Flanders Interuniversity Institute for Biotechnology, Leuven, Belgium
    Blood 105:4849-52. 2005
  8. pmc Deletion of the protein tyrosine phosphatase gene PTPN2 in T-cell acute lymphoblastic leukemia
    Maria Kleppe
    Department of Molecular and Developmental Genetics, VIB, Leuven, Belgium
    Nat Genet 42:530-5. 2010
  9. ncbi request reprint Frequent occurrence of BCL6 rearrangements in nodular lymphocyte predominance Hodgkin lymphoma but not in classical Hodgkin lymphoma
    Iwona Wlodarska
    Center for Human Genetics, Catholic University of Leuven, Belgium
    Blood 101:706-10. 2003
  10. ncbi request reprint Heterogeneity of BCL6 rearrangements in nodular lymphocyte predominant Hodgkin's lymphoma
    Iwona Wlodarska
    Center for Human Genetics, Katholieke Universiteit Leuven, Belgium
    Haematologica 89:965-72. 2004

Detail Information

Publications50

  1. pmc Telomeric IGH losses detectable by fluorescence in situ hybridization in chronic lymphocytic leukemia reflect somatic VH recombination events
    Iwona Wlodarska
    Center for Human Genetics, Catholoc University Leuven, Leuven, Belgium
    J Mol Diagn 9:47-54. 2007
    ....
  2. ncbi request reprint Translocations targeting CCND2, CCND3, and MYCN do occur in t(11;14)-negative mantle cell lymphomas
    Iwona Wlodarska
    Center for Human Genetics, Katholieke Universiteit Leuven, Leuven, Belgium
    Blood 111:5683-90. 2008
    ..Clinically important is a predisposition of t(11;14)-negative MCLs to the central nervous system involvement...
  3. ncbi request reprint Variant t(2;11)(p11;q13) associated with the IgK-CCND1 rearrangement is a recurrent translocation in leukemic small-cell B-non-Hodgkin lymphoma
    I Wlodarska
    Center for Human Genetics, Katholieke Universiteit Leuven, Belgium
    Leukemia 18:1705-10. 2004
    ..Our findings indicate that variant t(2;11)(p11;q13) does not typify a classical MCL but possibly a more indolent leukemic lymphoma originating from an antigen experienced (mutated) B cell...
  4. ncbi request reprint FOXP1, a gene highly expressed in a subset of diffuse large B-cell lymphoma, is recurrently targeted by genomic aberrations
    I Wlodarska
    Center for Human Genetics, Katholieke Universiteit Leuven, Leuven, Belgium
    Leukemia 19:1299-305. 2005
    ..However, over-representation of the FOXP1 locus found in one additional case of DLBCL may represent another potential mechanism underlying an increased expression of this gene...
  5. ncbi request reprint Forkhead box protein P1 expression in mucosa-associated lymphoid tissue lymphomas predicts poor prognosis and transformation to diffuse large B-cell lymphoma
    Xavier Sagaert
    Department of Morphology and Molecular Pathology, and the Centre for Human Genetics, Catholic University Leuven, Leuven, Belgium
    J Clin Oncol 24:2490-7. 2006
    ..In this study, we investigated FOXP1 expression in its relationship to morphology, genetic features, and prognosis in a series of 70 MALT lymphomas...
  6. pmc ALK-positive large B-cell lymphomas with cryptic SEC31A-ALK and NPM1-ALK fusions
    Katrien Van Roosbroeck
    Center for Human Genetics, K U Leuven, Gasthuisberg, Herestraat 49, Box 602, B 3000 Leuven, Belgium
    Haematologica 95:509-13. 2010
    ....
  7. ncbi request reprint Fusion of EML1 to ABL1 in T-cell acute lymphoblastic leukemia with cryptic t(9;14)(q34;q32)
    Kim De Keersmaecker
    Department of Human Genetics, Flanders Interuniversity Institute for Biotechnology, Leuven, Belgium
    Blood 105:4849-52. 2005
    ..These data further demonstrate the involvement of ABL1 fusions in the pathogenesis of T-ALL and identify EML1-ABL1 as a novel therapeutic target of imatinib...
  8. pmc Deletion of the protein tyrosine phosphatase gene PTPN2 in T-cell acute lymphoblastic leukemia
    Maria Kleppe
    Department of Molecular and Developmental Genetics, VIB, Leuven, Belgium
    Nat Genet 42:530-5. 2010
    ..Our study provides genetic and functional evidence for a tumor suppressor role of PTPN2 and suggests that expression of PTPN2 may modulate response to treatment...
  9. ncbi request reprint Frequent occurrence of BCL6 rearrangements in nodular lymphocyte predominance Hodgkin lymphoma but not in classical Hodgkin lymphoma
    Iwona Wlodarska
    Center for Human Genetics, Catholic University of Leuven, Belgium
    Blood 101:706-10. 2003
    ....
  10. ncbi request reprint Heterogeneity of BCL6 rearrangements in nodular lymphocyte predominant Hodgkin's lymphoma
    Iwona Wlodarska
    Center for Human Genetics, Katholieke Universiteit Leuven, Belgium
    Haematologica 89:965-72. 2004
    ..We present our results of metaphase-FISH analyses aimed at identifying and characterizing BCL6-related chromosomal translocations in NLPHL...
  11. doi request reprint JAK2 rearrangements, including the novel SEC31A-JAK2 fusion, are recurrent in classical Hodgkin lymphoma
    Katrien Van Roosbroeck
    Center for Human Genetics, Katholieke Universiteit Leuven, Leuven, Belgium
    Blood 117:4056-64. 2011
    ..The finding that SEC31A-JAK2 responds to JAK inhibitors indicates that patients with cHL and JAK2 rearrangements may benefit from targeted therapies...
  12. doi request reprint Recurrent breakpoints in 14q32.13/TCL1A region in mature B-cell neoplasms with villous lymphocytes
    Helena Urbankova
    Center for Human Genetics, KU Leuven, Leuven, Belgium
    Leuk Lymphoma 53:2449-55. 2012
    ..13/TCL1A-TCL6 region are recurrent in mature B-cell neoplasms with villous lymphocytes. Despite extensive qRT-PCR studies, molecular consequences of these novel aberrations remain elusive...
  13. ncbi request reprint Alterations of loci encoding PU.1, BOB1, and OCT2 transcription regulators do not correlate with their suppressed expression in Hodgkin lymphoma
    Francesco Cavazzini
    Center for Human Genetics, Katholieke Universiteit Leuven, Leuven, Belgium
    Cancer Genet Cytogenet 158:167-71. 2005
    ..These findings indicate that genomic imbalances or rearrangements are not a cause of PU.1, BOB1, and OCT2 deficiency in cHL and argue for another mechanism underlying this phenomenon...
  14. ncbi request reprint Fusion of ETV6 to GOT1 in a case with myelodysplastic syndrome and t(10;12)(q24;p13)
    Hilde Janssen
    Department of Human Genetics, Flanders Interuniversity Institute for Biotechnology, University of Leuven, Leuven, Belgium
    Haematologica 91:949-51. 2006
    ..This work identifies GOT1 as a novel fusion partner of ETV6 in myelodysplastic syndrome...
  15. doi request reprint Non-IG aberrations of FOXP1 in B-cell malignancies lead to an aberrant expression of N-truncated isoforms of FOXP1
    Leila Rouhigharabaei
    Center for Human Genetics, KU Leuven, Leuven, Belgium
    PLoS ONE 9:e85851. 2014
    ....
  16. doi request reprint Activity of imatinib in systemic mastocytosis with chronic basophilic leukemia and a PRKG2-PDGFRB fusion
    Idoya Lahortiga
    Human Genome Laboratory, Department of Molecular and Developmental Genetics, VIB, Leuven, Belgium
    Haematologica 93:49-56. 2008
    ..We report an unusual case of a patient presenting with peripheral basophilia and systemic mastocytosis in whom cytogenetic analysis revealed a t(4;5)(q21.1;q31.3)...
  17. ncbi request reprint Duplication of the MYB oncogene in T cell acute lymphoblastic leukemia
    Idoya Lahortiga
    Human Genome Laboratory, Department of Molecular and Developmental Genetics, Vlaams Instituut voor Biotechnologie VIB, Leuven, Belgium
    Nat Genet 39:593-5. 2007
    ..Our results identify duplication of MYB as an oncogenic event and suggest that MYB could be a therapeutic target in human T-ALL...
  18. doi request reprint BMI1, the polycomb-group gene, is recurrently targeted by genomic rearrangements in progressive B-cell leukemia/lymphoma
    Leila Rouhigharabaei
    Center for Human Genetics, Katholieke Universiteit Leuven, Leuven, Belgium
    Genes Chromosomes Cancer 52:928-44. 2013
    ..In summary, our study provides new insights into genetic molecular mechanisms underlying aberrant expression of BMI1 in lymphoma and documents its contribution in the pathogenesis of Richter syndrome and MCL...
  19. pmc Mutation analysis of the tyrosine phosphatase PTPN2 in Hodgkin's lymphoma and T-cell non-Hodgkin's lymphoma
    Maria Kleppe
    Department of Molecular and Developmental Genetics, VIB, Leuven, Belgium
    Haematologica 96:1723-7. 2011
    ..These results, together with our own data on T-cell acute lymphoblastic leukemia, demonstrate that PTPN2 is a tumor suppressor gene in T-cell malignancies...
  20. pmc MOHITO, a novel mouse cytokine-dependent T-cell line, enables studies of oncogenic signaling in the T-cell context
    Maria Kleppe
    Department of Molecular and Developmental Genetics, VIB, Leuven, Belgium
    Haematologica 96:779-83. 2011
    ....
  21. doi request reprint Interphase fluorescence in situ hybridization on selected plasma cells is superior in the detection of cytogenetic aberrations in plasma cell dyscrasia
    Natalie Put
    Centrum voor Menselijke Erfelijkheid, Katholieke Universiteit Leuven, Leuven, Belgium
    Genes Chromosomes Cancer 49:991-7. 2010
    ..001) and identified higher percentages of abnormal nuclei (P < 0.001). This study indicates that FISH on PCs is the preferred technique for routine cytogenetic investigation of MM...
  22. ncbi request reprint Clinicopathologic Comparison of Plasmablastic Lymphoma in HIV-positive, Immunocompetent, and Posttransplant Patients: Single-center Series of 25 Cases and Meta-analysis of 277 Reported Cases
    Julie Morscio
    Translational Cell and Tissue Research Lab, Department for Imaging and Pathology Center of Human Genetics, KU Leuven Departments of Hematology Pathology, University Hospitals, UZ Leuven, Leuven, Belgium
    Am J Surg Pathol 38:875-86. 2014
    ..The results of this retrospective study suggest different pathogenic mechanisms of PBL in different immunologic settings and a potentially important impact of EBV and CD45 on prognosis. ..
  23. pmc Refinement of 1p36 alterations not involving PRDM16 in myeloid and lymphoid malignancies
    Francois P Duhoux
    Center for Human Genetics, Cliniques Universitaires Saint Luc, Universite Catholique de Louvain, Brussels, Belgium
    PLoS ONE 6:e26311. 2011
    ..It is unclear how the latter rearrangements occurred and whether they represent oncogenic events or result from chromosomal instability during oncogenesis...
  24. pmc t(X;14)(p11.4;q32.33) is recurrent in marginal zone lymphoma and up-regulates GPR34
    Mathijs Baens
    Center for Human Genetics, KU Leuven, Leuven, Belgium
    Haematologica 97:184-8. 2012
    ..Although functional consequences of t(X;14) have not been identified, our studies suggest that up-regulated GPR34 activate neither nuclear factor-κB nor ELK-related tyrosine kinase...
  25. ncbi request reprint MALT1 and BCL10 aberrations in MALT lymphomas and their effect on the expression of BCL10 in the tumour cells
    Xavier Sagaert
    Division of Morphology and Molecular Pathology, Katholieke Universiteit Leuven, Leuven, Belgium
    Mod Pathol 19:225-32. 2006
    ....
  26. ncbi request reprint Comparative expressed sequence hybridization studies of hairy cell leukemia show uniform expression profile and imprint of spleen signature
    Vera Vanhentenrijk
    Division of Morphology and Molecualr Pathology and Center for Human Genetics, Katholieke Universiteit Leuven, Belgium
    Blood 104:250-5. 2004
    ..3-q21 were the most significantly overexpressed. These regions possibly harbor genes related to the biology and the pathogenesis of HCL. Their identification warrants further molecular investigations...
  27. pmc Comprehensive analysis of transcriptome variation uncovers known and novel driver events in T-cell acute lymphoblastic leukemia
    Zeynep Kalender Atak
    Laboratory of Computational Biology, Center for Human Genetics, KU Leuven, Leuven, Belgium
    PLoS Genet 9:e1003997. 2013
    ....
  28. ncbi request reprint The der(12)t(12;16) breakpoint in an acute leukaemia case targets a Sec7 domain containing protein
    Anniek Corveleyn
    Human Genome Laboratory, Department of Human Genetics, Flanders Interuniversity Institute for Biotechnology VIB, Katholieke Universiteit Leuven, Herestraat 49, B 3000 Leuven, Belgium
    Int J Oncol 26:1111-20. 2005
    ..However, the putative SLAG-MYH11 fusions showed only weak transforming properties in NIH3T3 cells in a focus formation assay...
  29. ncbi request reprint Methylation analysis of the imprinted DLK1-GTL2 domain supports the random parental origin of the IGH-involving del(14q) in B-cell malignancies
    Beata Katrincsakova
    Center for Human Genetics, Catholic University Leuven, Leuven, Belgium
    Epigenetics 4:469-75. 2009
    ..These findings argue against the concept that a TSG/anti-oncomir located in the imprinted region is systematically inactivated by a targeted deletion of its functional allele...
  30. ncbi request reprint Identification of novel fusion partners of ALK, the anaplastic lymphoma kinase, in anaplastic large-cell lymphoma and inflammatory myofibroblastic tumor
    Jan Cools
    Center for Human Genetics, Flanders Interuniversity Institute for Biotechnology VIB, University of Leuven, Campus Gasthuisberg O and N 06, Herestraat 49, B 3000 Leuven, Belgium
    Genes Chromosomes Cancer 34:354-62. 2002
    ..These results confirm the recurrent involvement of ALK in IMT and further demonstrate the diversity of ALK fusion partners, with the ability to homodimerize as a common characteristic...
  31. pmc The accuracy of positron emission tomography in the detection of posttransplant lymphoproliferative disorder
    Daan Dierickx
    Department of Hematology, University Hospitals Leuven, Leuven, Belgium
    Haematologica 98:771-5. 2013
    ....
  32. doi request reprint Chronic lymphocytic leukemia and prolymphocytic leukemia with MYC translocations: a subgroup with an aggressive disease course
    Natalie Put
    Center for Human Genetics, Catholic University of Leuven, Belgium
    Ann Hematol 91:863-73. 2012
    ..In conclusion, CLL/PLL with MYC translocations is a rare entity, which seems to be associated with adverse prognostic features and unfavorable outcome...
  33. doi request reprint Accurate determination of copy number variations (CNVs): application to the alpha- and beta-defensin CNVs
    Hilde Nuytten
    Department of Human Genetics, Katholieke Universiteit Leuven, Leuven, Belgium
    J Immunol Methods 344:35-44. 2009
    ..This strategy can be easily transferred to any CNV. With this method we structurally analyzed the alpha- and beta-defensin region in 334 Belgian individuals...
  34. ncbi request reprint Fluorescence in situ hybridization study of chromosome 7 aberrations in hepatosplenic T-cell lymphoma: isochromosome 7q as a common abnormality accumulating in forms with features of cytologic progression
    Iwona Wlodarska
    Center for Human Genetics, University of Leuven, Leuven, Belgium
    Genes Chromosomes Cancer 33:243-51. 2002
    ..An increased number of 7q signals was found in three cases with cytologic features of progression, indicating a tendency of HSTCL to multiply the i(7)(q10) chromosome during evolution...
  35. doi request reprint Exome sequencing identifies mutation in CNOT3 and ribosomal genes RPL5 and RPL10 in T-cell acute lymphoblastic leukemia
    Kim De Keersmaecker
    Center for Human Genetics, KU Leuven, Leuven, Belgium
    Nat Genet 45:186-90. 2013
    ..Our data provide insights into the mutational landscape of pediatric versus adult T-ALL and identify the ribosome as a potential oncogenic factor...
  36. ncbi request reprint Leukemic recombinations involving heterochromatin in myeloproliferative disorders with t(1;9)
    Constantina Sambani
    Laboratory of Health Physics and Environmental Hygiene, NCSR Demokritos, 15310 Aghia Paraskevi, Athens, Greece
    Cancer Genet Cytogenet 162:45-9. 2005
    ..The molecular event underlying juxtaposition of satellite II of chromosome 1 and the satellite III of chromosome 9 remains to be elucidated...
  37. ncbi request reprint PVRL2 is translocated to the TRA@ locus in t(14;19)(q11;q13)-positive peripheral T-cell lymphomas
    Carole Almire
    Groupe d Etude des Proliférations Lymphoides GPL, European Institute for Peptides Research IFRMP23, Centre Henri Becquerel, Rouen, France
    Genes Chromosomes Cancer 46:1011-8. 2007
    ..These results suggest that both BCL3 and PVRL2 may participate in the pathogenesis of these PTCLs, but further studies should be undertaken to investigate the precise role of these genes...
  38. ncbi request reprint PAX5/IGH rearrangement is a recurrent finding in a subset of aggressive B-NHL with complex chromosomal rearrangements
    Bruce Poppe
    Center for Medical Genetics, Ghent University Hospital, Ghent, Belgium
    Genes Chromosomes Cancer 44:218-23. 2005
    ..In addition, the recurrent incidence of this rearrangement in both HRTR-BCL (4 cases) and PTLD-DLBCL (2 cases) was previously unrecognized and is intriguing...
  39. ncbi request reprint Validation of an interphase fluorescence in situ hybridization approach for the detection of MLL gene rearrangements and of the MLL/AF9 fusion in acute myeloid leukemia
    Francesco Cavazzini
    Dipartimento di Scienze Biomediche e Terapie Avanzate, Sezione di Ematologia, University of Ferrara, Italy
    Haematologica 91:381-5. 2006
    ..This is a reliable method for the identification of MLL/AF9 fusion in interphase cells, allowing for a reclassification of cases with suboptimal chromosome morphology. The frequency of deletion surrounding MLL and AF9 breakpoint is low...
  40. ncbi request reprint Two new translocations involving the 11q23 region map outside the MLL locus in myeloid leukemias
    Emilia Giugliano
    Medicina Interna II, Dipartimento di Scienze Cliniche e Biologiche, Osp S Luigi Gonzaga, Orbassano, Italy
    Haematologica 87:1014-20. 2002
    ..In these two patients, involvement of the MLL gene was analyzed by molecular cytogenetic techniques which also allowed a more precise mapping of the breakpoints...
  41. ncbi request reprint Evidence for position effects as a variant ETV6-mediated leukemogenic mechanism in myeloid leukemias with a t(4;12)(q11-q12;p13) or t(5;12)(q31;p13)
    Jan Cools
    Human Genome Laboratory, Center for Human Genetics Flanders Interuniversity Institute for Biotechnology VIB, University of Leuven, Belgium
    Blood 99:1776-84. 2002
    ....
  42. pmc Comparative genomic hybridization pattern distinguishes T-cell/histiocyte-rich B-cell lymphoma from nodular lymphocyte predominance Hodgkin's lymphoma
    Sabine Franke
    Department of Human Genetics, Catholic University of Leuven, Belgium
    Am J Pathol 161:1861-7. 2002
    ..Altogether, our CGH findings of shared as well as distinctive cytogenetic features in both diseases suggest that T/HRBCL constitutes a separate lymphoma entity, possibly originating from the same precursor cell as LPHL...
  43. ncbi request reprint ALK activation by the CLTC-ALK fusion is a recurrent event in large B-cell lymphoma
    Pascale De Paepe
    Department of Pathology, Center of Medical Genetics and Pediatric Oncology, Ghent University Hospital, Belgium
    Blood 102:2638-41. 2003
    ..Although a similar CLTC-ALK aberration was previously identified in ALK-positive T-/null cell ALCL and inflammatory myofibroblastic tumor, its association with ALK-positive LBCL seems to be specific and intriguing...
  44. ncbi request reprint A tyrosine kinase created by fusion of the PDGFRA and FIP1L1 genes as a therapeutic target of imatinib in idiopathic hypereosinophilic syndrome
    Jan Cools
    Brigham and Women s Hospital and Harvard Medical School, Boston, USA
    N Engl J Med 348:1201-14. 2003
    ..Recent reports of responses to imatinib in patients with the syndrome suggested that an activated kinase such as ABL, platelet-derived growth factor receptor (PDGFR), or KIT, all of which are inhibited by imatinib, might be the cause...
  45. ncbi request reprint Activating mutation in the tyrosine kinase JAK2 in polycythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosis
    Ross L Levine
    Division of Hematology, Department of Medicine, Brigham and Women s Hospital, Harvard Medical School, Boston, Massachusetts 02115, USA
    Cancer Cell 7:387-97. 2005
    ..In vitro analysis demonstrated that JAK2V617F is a constitutively active tyrosine kinase...
  46. ncbi request reprint Large cleaved and immunoblastic lymphoma may represent two distinct clinicopathologic entities within the group of diffuse large B-cell lymphomas
    Pascale De Paepe
    Department of Pathology, Ghent University Hospital, Ghent, Belgium
    J Clin Oncol 23:7060-8. 2005
    ..Furthermore, we investigated whether both subgroups could comprise clinicopathologic entities recognized by their morphology and characterized by a distinct phenotype, specific genetic abnormalities, and clinical characteristics...
  47. doi request reprint NOTCH2 mutations in marginal zone lymphoma
    Gunhild Trøen
    Haematologica 93:1107-9. 2008
  48. ncbi request reprint Prognostic significance of nuclear and cytoplasmic expression of metallothioneins as related to proliferative activity in squamous cell carcinomas of oral cavity
    Jolanta Szelachowska
    Department of Oncology, Wroclaw Medical University, Wroclaw, Poland
    Histol Histopathol 23:843-51. 2008
    ..Conclusion: This is relevant evidence that MT overexpression could be related to worse prognosis in patients with oral cancer. We have found no relationship between MT expression and proliferative activity...
  49. ncbi request reprint Is there a difference in childhood T-cell acute lymphoblastic leukaemia and T-cell lymphoblastic lymphoma?
    Anne Uyttebroeck
    Department of Paediatric Haemato Oncology, University Hospital Leuven, Belgium
    Leuk Lymphoma 48:1745-54. 2007
    ....
  50. ncbi request reprint Chromosomal rearrangements involving the BCL3 locus are recurrent in classical Hodgkin and peripheral T-cell lymphoma
    Jose I Martin-Subero
    Blood 108:401-2; author reply 402-3. 2006